ORIGINAL RESEARCH
Accepted on 07 Mar 2025
Midline-1 inhibited high glucose-induced epithelial-mesenchymal transition, fibrosis and inflammation through WNT/β-catenin signaling in benign prostatic hyperplasia
doi 10.3389/fendo.2025.1543295
- Frontiers in Endocrinology
- Cellular Endocrinology
- Research Topics
- Epithelial-Mesenchymal Transition (EMT) in Endocrine Disorders: Mechanisms, Hormonal Regulation, and Therapeutic Potential
Epithelial-Mesenchymal Transition (EMT) in Endocrine Disorders: Mechanisms, Hormonal Regulation, and Therapeutic Potential
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