Cardiovascular diseases are a leading cause of mortality worldwide, being affected by various factors and pathophysiological pathways. While allergic diseases are an atopic phenomenon, they share an essential pathology with cardiovascular diseases; increased inflammation and increased immune system activity.
Previous studies have shown that immunoglobulin E increases after the incidence of myocardial infarction. The increased levels of proinflammatory cytokines, mast cells and basophils in allergic reactions potentially contribute to endothelium damage, plaque disruption and acute coronary syndromes. Meanwhile the pharmacotherapy being used in allergic disease like corticosteroids are an important cardiovascular risk factors in these patients.
While it has been hypothesized that both allergic reactions and cardiovascular disorders affect one another, the aim of this research topic is to increase the evidence addressing the controversies and pathophysiology between cardiovascular and allergic diseases.
All manuscript types accepted by Frontiers in Allergy are welcome. All submissions must address the link between cardiovascular and allergic diseases.
The scope of papers can be included but not limited to:
• Pathologic effects of hypersensitivity reactions on the cardiovascular system
• Adverse effects of allergic disease pharmacotherapeutics on the cardiovascular system
• The role of Immunoglobulin E and acute phase reactants in cardiovascular diseases
• Allergies and cardiovascular risk factors
• Kounis syndrome (allergic angina and allergic myocardial infarction)
• Management of allergies in cardiovascular patients
Keywords:
Cardiovascular Disorders, Allergy, Allergic reactions, Kounis syndrome, acute coronary syndromes, hypersensitivity reactions
Important Note:
All contributions to this Research Topic must be within the scope of the section and journal to which they are submitted, as defined in their mission statements. Frontiers reserves the right to guide an out-of-scope manuscript to a more suitable section or journal at any stage of peer review.
Cardiovascular diseases are a leading cause of mortality worldwide, being affected by various factors and pathophysiological pathways. While allergic diseases are an atopic phenomenon, they share an essential pathology with cardiovascular diseases; increased inflammation and increased immune system activity.
Previous studies have shown that immunoglobulin E increases after the incidence of myocardial infarction. The increased levels of proinflammatory cytokines, mast cells and basophils in allergic reactions potentially contribute to endothelium damage, plaque disruption and acute coronary syndromes. Meanwhile the pharmacotherapy being used in allergic disease like corticosteroids are an important cardiovascular risk factors in these patients.
While it has been hypothesized that both allergic reactions and cardiovascular disorders affect one another, the aim of this research topic is to increase the evidence addressing the controversies and pathophysiology between cardiovascular and allergic diseases.
All manuscript types accepted by Frontiers in Allergy are welcome. All submissions must address the link between cardiovascular and allergic diseases.
The scope of papers can be included but not limited to:
• Pathologic effects of hypersensitivity reactions on the cardiovascular system
• Adverse effects of allergic disease pharmacotherapeutics on the cardiovascular system
• The role of Immunoglobulin E and acute phase reactants in cardiovascular diseases
• Allergies and cardiovascular risk factors
• Kounis syndrome (allergic angina and allergic myocardial infarction)
• Management of allergies in cardiovascular patients
Keywords:
Cardiovascular Disorders, Allergy, Allergic reactions, Kounis syndrome, acute coronary syndromes, hypersensitivity reactions
Important Note:
All contributions to this Research Topic must be within the scope of the section and journal to which they are submitted, as defined in their mission statements. Frontiers reserves the right to guide an out-of-scope manuscript to a more suitable section or journal at any stage of peer review.