EBV Strategies to Counteract the Immune Response

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ORIGINAL RESEARCH

Published on 29 Nov 2024

Entry into the lytic cycle exposes EBV-infected cells to NK cell killing via upregulation of the MICB ligand for NKG2D and activation of the CD56bright and NKG2A+KIR+CD56dim subsets

Maria Giovanna Desimio
Daniela Angela Covino
Caterina Cancrini
Margherita Doria

doi 10.3389/fimmu.2024.1467304

390 views

REVIEW

Published on 09 Jul 2024

Hypocortisolemic ASIA: a vaccine- and chronic infection-induced syndrome behind the origin of long COVID and myalgic encephalomyelitis

Manuel Ruiz-Pablos
Bruno Paiva
Aintzane Zabaleta

doi 10.3389/fimmu.2024.1422940

14,002 views
1 citation

REVIEW

Published on 07 Feb 2024

Epstein-Barr virus: the mastermind of immune chaos

Jean de Melo Silva
Carlos Eduardo de Castro Alves
Gemilson Soares Pontes

doi 10.3389/fimmu.2024.1297994

9,423 views
11 citations

REVIEW

Published on 21 Dec 2023

Mechanisms of T cell evasion by Epstein-Barr virus and implications for tumor survival

D. G. Sausen
M. C. Poirier
L. M. Spiers
E. N. Smith

doi 10.3389/fimmu.2023.1289313

2,293 views
1 citation

ORIGINAL RESEARCH

Published on 22 Jun 2023

Inhibitory NKG2A+ and absent activating NKG2C+ NK cell responses are associated with the development of EBV+ lymphomas

Hannes Vietzen
Philipp B. Staber
Sarah M. Berger
Philippe L. Furlano
Laura M. Kühner
Simone Lubowitzki
Alexander Pichler
Robert Strassl
Jan J. Cornelissen
Elisabeth Puchhammer-Stöckl

doi 10.3389/fimmu.2023.1183788

2,244 views
5 citations

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Frontiers in Immunology

Viral Immunology

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Articles

Entry into the lytic cycle exposes EBV-infected cells to NK cell killing via upregulation of the MICB ligand for NKG2D and activation of the CD56bright and NKG2A+KIR+CD56dim subsets

Hypocortisolemic ASIA: a vaccine- and chronic infection-induced syndrome behind the origin of long COVID and myalgic encephalomyelitis

Epstein-Barr virus: the mastermind of immune chaos

Mechanisms of T cell evasion by Epstein-Barr virus and implications for tumor survival

Inhibitory NKG2A+ and absent activating NKG2C+ NK cell responses are associated with the development of EBV+ lymphomas

Frontiers in Immunology

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