EDITORIAL
Published on 05 Jan 2016
Editorial: Neuronal Self-Defense: Compensatory Mechanisms in Neurodegenerative Disorders
doi 10.3389/fncel.2015.00499
- 2,306 views
- 3 citations
- Frontiers in Cellular Neuroscience
- Cellular Neurophysiology
- Research Topics
- Neuronal Self-Defense: Compensatory Mechanisms in Neurodegenerative Disorders
Neuronal Self-Defense: Compensatory Mechanisms in Neurodegenerative Disorders
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Articles
ORIGINAL RESEARCH
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SINEUPs are modular antisense long non-coding RNAs that increase synthesis of target proteins in cells
doi 10.3389/fncel.2015.00174
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ORIGINAL RESEARCH
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Expression analysis of the long non-coding RNA antisense to Uchl1 (AS Uchl1) during dopaminergic cells' differentiation in vitro and in neurochemical models of Parkinson's disease
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MINI REVIEW
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Compensatory mechanisms in genetic models of neurodegeneration: are the mice better than humans?
doi 10.3389/fncel.2015.00056
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REVIEW
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Legal but lethal: functional protein aggregation at the verge of toxicity
doi 10.3389/fncel.2015.00045
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ORIGINAL RESEARCH
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Involvement of ELAV RNA-binding proteins in the post-transcriptional regulation of HO-1
doi 10.3389/fncel.2014.00459
- 3,253 views
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REVIEW
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RNA-mediated pathogenic mechanisms in polyglutamine diseases and amyotrophic lateral sclerosis
doi 10.3389/fncel.2014.00431
- 7,674 views
- 26 citations
MINI REVIEW
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ER stress and unfolded protein response in amyotrophic lateral sclerosis—a controversial role of protein disulphide isomerase
doi 10.3389/fncel.2014.00402
- 9,726 views
- 63 citations
ORIGINAL RESEARCH
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Elevated mRNA-levels of distinct mitochondrial and plasma membrane Ca2+ transporters in individual hypoglossal motor neurons of endstage SOD1 transgenic mice
doi 10.3389/fncel.2014.00353
- 4,697 views
- 21 citations
ORIGINAL RESEARCH
Published on 13 Nov 2014
De-regulation of gene expression and alternative splicing affects distinct cellular pathways in the aging hippocampus
doi 10.3389/fncel.2014.00373
- 17,638 views
- 108 citations
HYPOTHESIS AND THEORY
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Prognostic role of “prion-like propagation” in SOD1-linked familial ALS: an alternative view
doi 10.3389/fncel.2014.00359
- 4,766 views
- 12 citations
ORIGINAL RESEARCH
Published on 22 Oct 2014
Compensatory Motor Neuron Response to Chromatolysis in the Murine hSOD1G93A Model of Amyotrophic Lateral Sclerosis
doi 10.3389/fncel.2014.00346
- 6,123 views
- 30 citations
