- 1Department of Social and Behavioural Sciences, City University of Hong Kong, Hong Kong, China
- 2Department of Educational Psychology, Faculty of Education, The Chinese University of Hong Kong, Shatin, Hong Kong
Objectives: Posttraumatic stress disorder (PTSD) symptoms following past trauma could lead to psychological distress. Little is known, however, about the roles of defense mechanisms and alexithymia may play in the process. The current study aimed to examine the potential impact of alexithymia and defense mechanisms on the relationship between past trauma and distress among Chinese university students.
Method: 455 university students completed a set of questionnaires: PTSD Checklists for DSM-5, Toronto Alexithymia Scale (TAS-20), Defense Style Questionnaire, and General Health Questionnaire-28.
Results: PTSD following past trauma was associated with increased psychological distress. Alexithymia and defenses (especially immature defense) mediated the path between PTSD and psychological co-morbidities.
Conclusion: Following past trauma, people developed PTSD and other psychological symptoms. The severity of these distress symptoms was influenced by the way they defended themselves psychologically, and their ability to identify, express, and process distressing emotions.
Introduction
Exposure to a traumatic event is a statistically normative experience in the general population, as exemplified by large scale epidemiology studies (Kilpatrick et al., 2013; Benjet et al., 2016), many of which (approximately 56–74%) have reported exposure to at least one potentially traumatic event (e.g., assault or accident) during their lives (Norris, 1992; Kessler et al., 1995; Breslau, 2002; Atwoli et al., 2015). Increasing evidence from research and clinical practice shows that a history of trauma is a potent risk factor for psychological disorders (Davidson et al., 1991; Halligan and Yehuda, 2000; Keane et al., 2006) of which posttraumatic stress disorder (PTSD) is the most common (APA, 2013).
Notwithstanding this, following a trauma, not all victims will go on to develop PTSD; some may experience disruptions in functioning but gradually return to normal, others maintain clinically significant psychological co-morbidities (e.g., Smyth et al., 2008) such as depression (Hill, 2003; Briere, 2004) and anxiety (Heim and Nemeroff, 2001) which require prolonged intervention (Kessler et al., 2005; Espié et al., 2009; Bonanno and Mancini, 2012). Individual differences suggest that the impact of traumatic life events on distress outcomes is not a direct one (Verhaeghe and Vanheule, 2005) but through a variety of factors. The current research endeavored to understand the mediating mechanisms that link PTSD from past trauma with psychological distress by focusing on two potential psychodynamic constructs: ego defense mechanisms and alexithymia.
Psychological defenses following traumas aim to hide or alleviate conflicts or stressors that give rise to anxiety (Beresford, 2012). It is an automatic and unconscious psychological process that aims to protect the individual against psychological distress and prevent awareness of internal or external danger and stress (Vaillant, 1971, 1992). Although it has been removed from the Diagnostic and Statistical Manual of Mental Disorders (5th ed; APA, 2013), its clinical utility and predictive value for positive adaptation is profound (Vaillant, 2011). Defenses may be ordered hierarchically and divided into four categories according to their psychosocial maturity and level of adaptiveness (Vaillant, 1992): (1) psychotic defenses (e.g., psychotic denial and distortion) which aim to avoid or circumscribe conflicts encountered in the relationship with the external world; (2) immature defenses (e.g., acting out, passive aggression, and projection) which aim to lessen distress and anxiety caused by threatening individuals or uncomfortable reality; (3) neurotic defenses (e.g., displacement, isolation, and repression) which aim to keep potentially threatening feelings, ideas, memories, wishes, or fears out of awareness (Bond, 1986); and (4) mature defenses (e.g., sublimation, humor, and altruism) which may maximize gratification and allow more conscious awareness of feelings, ideas, and their consequences (Vaillant, 2011).
Defense style may be an important factor in explaining the aforementioned individual differences in trauma reactions (Jun et al., 2015). Some defenses are more associated with psychiatric symptoms than others (Jun et al., 2015). For example, psychotic and immature defenses have been found in PTSD victims of different types including combat veterans (Silverstein, 1996; Vaillant, 2011). Abundant studies have also reported that immature defense styles were associated with distress and psychological symptoms (Nickel and Egle, 2006; Diehl et al., 2014; Martino et al., 2020) such as anxiety and depressive disorders (Bond, 2004) among people with a history of childhood trauma and major illness. Other findings also demonstrated that immature defense mediated the relationship between childhood trauma and psychopathology in adulthood (Finzi-Dottan and Karu, 2006; Nickel and Egle, 2006). As such, it is reasonable to speculate that the development of psychological problems after trauma may be mediated by a specific type of defense mechanism. However, to the best of our knowledge, no study has investigated whether defenses might mediate the impact of PTSD from past trauma onto psychological distress among non-clinical samples (Jun et al., 2015).
The stress response syndrome (Horowitz, 1976) emphasizes the need for trauma victims to make the internal model of psychic trauma coherent with existing mental schemas (the completion tendency). However, the trauma-related information may be so incompatible with the existing schema of the world and themselves that it creates overwhelming emotional distress. To prevent emotional exhaustion, an inhibitory mechanism may be activated to inhibit intensive and negative emotions (Helmes et al., 2008; Meganck et al., 2013) and avoid frightening or intolerable feelings (Busch, 2014). This response aims to keep not only distressing emotions at bay but also the individual from integrating the traumatic experience with long-term meaning representations (Horowitz, 1986).
The emergence of alexithymia can be seen as one of these unconscious inhibitory responses which is in itself a defense mechanism (Kooiman et al., 1998). Coined by Nemiah and Sifneos (1970), “alexithymia” is defined as having difficulty identifying and describing feelings, relying instead on external oriented thinking (EOT; Taylor et al., 1997). It has been postulated that victims subconsciously adopt alexithymia to cut off overwhelming emotional distress by preventing themselves from accessing internal feelings (Ahrens and Deffner, 1986; Helmes et al., 2008; Meganck et al., 2013; Chung et al., 2016) but focusing on external facts (e.g., Gross, 1998; Kupchik et al., 2007; Declercq et al., 2010). However, unresolved and unprocessed feelings accumulated in the body can cause later disturbance in the physiological and neurological system, giving rise to health difficulties and a range of psychological distress symptoms (e.g., Pennebaker and Beall, 1986; Karukivi et al., 2010; Marchini et al., 2018; Martino et al., 2019a). It is therefore unsurprising that alexithymia often negatively correlates with physical and psychological well-being, but positively associates with physical illness, somatization (Taylor et al., 1991; Sayar et al., 2003), anxiety (Karukivi et al., 2010), and depression (Honkalampi et al., 2000). As such, alexithymia may act as another mediator in the relationship between PTSD from past trauma and psychological distress. This has received support from several studies (Helmes et al., 2008; Chung et al., 2013; Chung and Hunt, 2014; Chen and Chung, 2016).
As was mentioned, since alexithymia can function as a defense, it is not surprising that it has been correlated with increased use of defenses in several studies (e.g., Besharat and Shahidi, 2011; Evren et al., 2012). Specifically, some studies found that alexithymia was positively associated with immature defenses among samples from different countries (Wise et al., 1991; Parker et al., 1998). Similarly, alexithymia showed moderate positive correlations with neurotic defense among both clinical and non-clinical samples (e.g., Parker et al., 1998); this result is not always consistent however (e.g., Bogutyn et al., 1999). Although research has demonstrated that alexithymia was negatively correlated with mature defenses among psychiatric outpatients (Kooiman et al., 1998), a recent study showed that alexithymia and emotional suppression (as a mature defense) “independently” mediated the path between PTSD from past trauma to distress outcomes (Chung and Hunt, 2014). On the contrary, another finding showed that suppression as a defense did not mediate outcomes in conjunction with alexithymia among asthma sufferers (Chung et al., 2012). In short, evidence on the interlocking relationship between alexithymia and defenses is inconsistent.
The current study aimed to examine a theoretical framework demonstrating the potential mechanisms of defense styles and alexithymia mediating the impact of PTSD from past trauma onto psychological distress. This framework is shown in Figure 1. Our hypotheses were as follows: (1) PTSD following past trauma would be positively associated with psychological distress; (2) alexithymia and defense style would mediate the links between PTSD and distress outcomes respectively; and (3) alexithymia would be significantly positively associated with defense style.
Figure 1. Theoretical model. PTSD = posttraumatic stress disorder symptoms, Distress = psychological distress.
Materials and Methods
Participants and Procedure
Approval was obtained from the ethics committee at the affiliated university (approval number: EDU2016-0218). Four hundred and fifty-five university students from Mainland China and Hong Kong participated in this study. Participants were recruited via advertisements posted in student resident halls and email via the university mass mailing system. The inclusion criteria were: (1) university students with an active student registration status, (2) over 18, (3) no history of major physical illness, and (4) no psychiatric history. A briefing page stated that the study aimed to examine the association among traumatic life events, emotion, psychological defense, and mental health. Students were informed that the study was entirely voluntary and anonymous. Data would be kept confidential. Participants were not financially compensated and entitled to exit from the study at any point.
Measures
Demographic Information
Participants provided information on age, gender, marital status, study mode, and ethnicity.
Alexithymia
Toronto Alexithymia Scales (TAS-20, Bagby et al., 1994) is a 20-item self-report questionnaire on a five-point Likert scale from “1 = Never” to “5 = Very often.” It measures the degree of alexithymia with three dimensions: (1) difficulties in identifying feelings (DIF); (2) difficulties in describing feelings (DDF); and (3) externally oriented thinking (EOT). The reliability of subscales was found to be satisfactory (DIF: 0.86; DDF: 0.71; and EOT: 0.61) and a 3-week test–retest reliability was 0.77 in previous research (Chung and Wall, 2013). Cronbach’s alphas of current study for DIF, DDF, and EOT were 0.84, 0.70, and 0.60.
Defense Styles
Defense Style Questionnaire-40 (DSQ-40, Andrews et al., 1993) aims to measure the hierarchy of defense mechanisms indicating the individual’s typical mode of dealing with conflict (Vaillant, 1977). It is a 40-item questionnaire with a nine-point Likert scale (1 = strongly disagree to 9 = strongly agree) measuring three types of defense styles (immature, neurotic, and mature). Sample items are “I often act impulsively when something is bothering me” (immature style), “If someone mugged me and stole my money, I’d rather he be helped than punished” (neurotic style), and “I work out my anxiety through doing something constructive and creative like painting or wood-work” (mature style). The construct validity has been demonstrated among different clinical populations and test–retest correlations were also reported with an average r of 0.66 using a 4-week time period (Andrews et al., 1993). Cronbach’s alpha ranged from 0.64 to 0.83 (Sinha and Watson, 1999) and were reported as 0.73, 0.70, and 0.88, respectively, for immature, neurotic, and mature defenses, respectively, in this study.
PTSD
Posttraumatic stress disorder Checklist for DSM-5 (PCL-5, Weathers et al., 2013) aims to measure posttraumatic stress reactions following a traumatic event. Part I assesses the trauma exposure in the past and lists 17 categories of traumatic event, such as natural disaster, motor vehicle accident, assault, and sudden death of a loved one. Participants indicated whether they had experienced, witnessed, or never been exposed to them. If they experienced more than one trauma, they proceeded to indicate which trauma was the most traumatic. Part II is a 20-item self-report measure designed to assess the PTSD symptoms (intrusion, avoidance, negative mood and cognition, and hyperarousal) using the rating scale 0 = not at all to 4 = extremely. Cronbach’s alphas were 0.93, 0.90, 0.93, and 0.93, respectively, for the current study.
Psychiatric Co-Morbidity
General Health Questionnaire-28 (Goldberg and Hillier, 1979) is a 28-item scale on a four-point Likert scale (0 = better than usual to 3 = worse than usual) measuring four types of health conditions (somatic symptoms, anxiety and insomnia, psychosocial dysfunction, and severe depression). Numerous studies have shown that the reliability was excellent as α ranging from 0.90 to 0.95 (Sterling, 2011). Cronbach’s alphas were reported as 0.82, 0.90, 0.81, and 0.93 for each dimension for the current study.
Data Analysis
Descriptive statistics were used to describe demographic information and the prevalence rate of PTSD. Correlation coefficients were used to establish whether demographic variables were related to distress outcomes. Multivariate normality was checked and no variables were needed for transformation. No outliers were detected during the exploration of diagnostics (Mahalanobis distance = 3 SD). Assumptions relating to linearity, and homoscedasticity were met. Regression imputation was used to address missing data. With less than 2% of responses missing, as in this case, there is little distortion (Schafer and Graham, 2002).
To test our theoretical model (see Figure 1), the following steps were taken. First, we conducted a confirmatory factor analysis to assess the measurement model (MM). Second, the model was tested with structural equation modeling (SEM) (Arbuckle, 2012) to examine whether alexithymia and each defense style would mediate the relationships between PTSD from past trauma and distress outcomes. Maximum likelihood (ML) estimation was adopted. The evaluation of the overall model fit was based on these indices: chi-square (χ2) statistics, comparative fit index (CFI), Tucker–Lewis index (TLI), and root-mean-square error of approximation (RMSEA), and 90% confidence intervals. For CFI and TLI, values greater than.90 indicate acceptable model fit. For RMSEA, a value less than 0.08 reflects reasonable model fit (Hu and Bentler, 1998). Data were analyzed using SPSS 25.0 version and AMOS 24.0.
Furthermore, we used PROCESS (Hayes, 2016) to verify the mediational effects of the hypothesized model. Bias-corrected bootstrapping was adopted to generate confidence intervals which addressed the problem of power resulting from the asymmetric and non-normal sampling distributions of the indirect effect (MacKinnon et al., 2004). Point estimates and 95% confidence intervals were estimated for the indirect effects. When zero was not contained in the confidence interval, point estimates of indirect effects were then considered to be significant.
Results
Descriptive and Correlation Results
All 455 participants (M = 170, 37%, F = 285, 63%) were full time students, not married, and ranged from 18 to 25 years old (M = 19.75, SD = 1.67). Among them, 65% (n = 295) reported having experienced at least one traumatic life event, of whom 11% experienced only one, 18% two, 10% three, and 26% four or more (LEC total, M = 4.52, SD = 6.12). Using the DSM-5 diagnostic criteria, we found that 23% (n = 105) and 36% (n = 165) met the criteria for full and partial PTSD, respectively. The rest had no PTSD (41%, n = 185). No PTSD referred to participants who did not meet any of the diagnostic criteria for the four clusters of PTSD symptoms. Partial-PTSD referred to those who had met the diagnostic criteria for one or two of the symptom clusters (Chung and Wall, 2013). There were no significant correlations between demographic variables and distress outcomes (gender, r = −0.002, ns; age, r = 0.028, ns). Table 1 shows the results of means, standard deviations (SDs), and Pearson correlations for all observed indicators.
Measurement Model (MM)
All the indicators loaded significantly on their factors, indicating that the variables had a good validity, except EOT. As noted in Section “Measures,” this subscale demonstrated low internal consistency. Similar results were also reported in previous studies (Mazzeo and Espelage, 2002; Mazzeo et al., 2008; Fang and Chung, 2019). Consequently, we dropped the indicator of EOT from the MM. Table 2 shows information on the Goodness of fit of the MM indicating acceptable fit of the data.
Structural Models
For Structural Model 1 (SM1) with immature defense as the index defense, all the coefficients of latent variables reached significance (p < 0.05) ranging from 0.14 to 0.49 (see Figure 2). For Structural Model 2 (SM2) and Structural Model 3 (SM3) with neurotic and mature defenses as index defenses, respectively, alexithymia was significantly correlated with PTSD and distress (see Tables 3, 4). Neurotic defense was also significantly correlated with PTSD. Otherwise, it was not associated with distress or alexithymia. Mature defense was not correlated with any of the other latent variables. Results on model fit indicated acceptable fit (see Table 2).
Figure 2. Structural model and SEM coefficients. **p < 0.01, ***p < 0.001. PTSD = posttraumatic stress disorder symptoms, Distress = psychological distress.
Direct and Indirect Effects of Alexithymia and Defense Mechanism
Based on the SEM results, we further examined the direct and indirect effects of different defense mechanisms and alexithymia on distress outcomes by computing the multiple mediation bootstrap analysis using PROCESS. The indirect effects of PTSD from past trauma on distress outcomes through immature defenses and alexithymia were significant with confidence intervals being statistically different from zero based on a 95% bootstrap. Also, the direct effect of PTSD on distress remained significant (β = 0.29, SE = 0.04, t = 8.07, p < 0.001). In other words, alexithymia and immature defenses partially mediated the association between PTSD from past trauma and distress (see Table 5).
Discussion
This study explored the interrelationships between PTSD from past trauma, alexithymia, defense mechanisms, and psychological distress. Hypotheses were partially supported. First, PTSD from past trauma was significantly associated with psychological distress. Second, alexithymia and immature defense partially mediated the path between PTSD and distress. Third, alexithymia was positively correlated with immature defense. However, no significant correlation was found between alexithymia, mature, and neurotic defenses.
Consistent with literature (e.g., Vrana and Lauterbach, 1994; Bernat et al., 1998), students developed PTSD after traumatic life events. Although past trauma happened one year ago, on average, approximately 23 and 36% of the sample still experienced full and partial PTSD symptoms, respectively. Further analysis revealed that all PTSD subscales were positively correlated with all psychological co-morbid symptoms ranging from 0.22 to 0.50 in r values. This is not surprising given that PTSD is often expressed through other psychological symptoms (Miller et al., 2004).
The prevalence rate for full PTSD was higher than the rates reported in literature ranging from 6 to 17% (e.g., Smyth et al., 2008). This might be due to the different tools used to assess PTSD, as the Impact of Event Scales (IES, Horowitz et al., 1979) were used in Smyth’s study. Furthermore, while intrusive symptoms may be universal, avoidance has been shown to be culturally specific (Marsella et al., 1996; Norris et al., 2001). People with Eastern cultural beliefs may express a higher degree of avoidance and numbness leading to higher PTSD prevalence rates than those with Western cultural characteristics (Foa et al., 1989; Macdonald et al., 2013). However, this interpretation needs to be verified using cross-cultural comparative studies.
The current findings provided further evidence to the relationship between PTSD from past trauma and alexithymia (Bagby and Taylor, 1997; Frewen et al., 2008), between alexithymia and distress (Lumley et al., 2007), and the mediational effect of alexithymia (Helmes et al., 2008; Chung and Hunt, 2014; Chen and Chung, 2016). Students with a high level of trauma severity tended to display a high level of alexithymia which was associated with elevated psychological distress (Zhu et al., 2006). These results have contradicted the claim that alexithymia is an unconscious defense mechanism which acts as a protective strategy by repressing particular stimulations (e.g., Wise et al., 1990; Kooiman et al., 1998). Instead, such inhibitory strategy might be maladaptive in nature and associated with extreme affective impoverishment (Van der Kolk and Ducey, 1989), impacting psychological distress.
In psychoanalytic literature, the inability to put experiences into words and active motivation to forget unacceptable memories leads to repression. Alexithymia may be the presentation of such unconscious repression and acting out (Van der Kolk and Ducey, 1989). As a result, however, traumatized students may continue to experience the effects of the unintegrated experiences and unresolved distressing emotions, until they learn to put into words both the associated facts and the feelings. Accumulated unprocessed distressing feelings are related to disturbances in both physical and psychological health (e.g., Amstadter and Vernon, 2008).
Similarly, traumatized students who used immature defenses tended to report elevated psychological distress. This result echoed previous findings on immature defense mediating the impact of, for example, childhood trauma on psychopathology in adulthood (Finzi-Dottan and Karu, 2006; Nickel and Egle, 2006). It needs to be interpreted with caution, however, because evidence exists to show that immature defenses can be adaptive in certain contexts (Walburg and Chiaramello, 2015). For instance, the adaptive role of denial has been found among individuals with trauma (e.g., Langher et al., 2019) and bereavement experiences especially at the very beginning of their grieving process (Chabrol and Callahan, 2018). Denial can act as a defense against the painful effects of a devastating illness which resembles the process of somatic depersonalization whereby patients detach bodily experiences from the self (Caputo, 2019b). Manic reparation is also thought to be adaptive in facing depressive feelings. The present traumatized students might have relied on it to restore a damaged self by alleviating guilt feelings, enhancing self-efficacy, and preserving hope after trauma (e.g., Caputo et al., 2019). Thus, depending on different contexts in which trauma occurred, students might have benefited from endorsing different immature defenses. But this speculation remains to be investigated.
Immature defense was positively correlated with alexithymia which supported some findings in literature (Wise et al., 1990; Kooiman et al., 1998). Further regression analysis revealed that projection (β = 0.15) and acting out (β = 0.15) were among the strongest predictors to alexithymia. The former is an unconscious desire to displace unwanted or unacceptable feelings onto others while the latter is an attempt to engage in action so that internal feelings are not reflected upon. It is an unconscious expression of an impulse to avoid the awareness of certain affects. In other words, both defenses aim not to confront internal emotions but to deny them. The more students engaged in these specific immature defenses, the more they found difficulty in accessing, articulating and expressing their internal feelings (i.e., alexithymia). This interlocking psychological reaction seemed to have related to the increase of psychological distress for students who had experienced trauma.
This interlocking psychological reaction did not emerge, however, for those who primarily endorsed neurotic and mature defenses. The psychological process whereby alexithymia mediated the impact of past trauma onto distress seems to have occurred independently of the levels of neurotic and mature defenses used. In fact, inconsistent with literature (e.g., Kwon and Lemon, 2000; Kennedy et al., 2001; Watson, 2002; Corruble et al., 2004; Eglinton and Chung, 2011), neurotic and mature defenses did not seem to relate to almost any of the other psychological constructs. In other words, while, according to literature (Walburg and Chiaramello, 2015), it might be beneficial or adaptive to develop greater psychological flexibility of the defense system (i.e., as opposed to relying simply on one defense) in responding to various situations, with defense style correlated with specific psychological distress symptoms or disorder (Akkerman et al., 1992; Bond, 2004; Flett et al., 2005), the present results suggest that some of these defenses might in fact have little impact on distress outcomes. As a result, the notion that all defense styles would influence distress outcomes needs to be revisited.
Nevertheless, these results were mainly based on the relationship between defense mechanisms and distress outcomes. The interactionists (Endler, 1983) would argue that it is an oversight not taking account of environmental or situational factors, personality traits, and cognitive processes (Endler, 1983) which could have produced different results of defense as a coping process for distress. After all, the university students in our samples were mostly “emerging adults,” who, according to Erikson (1979) developmental stages, would have been in a transition stage whereby they have entered into an intellectually stimulating environment at a time when issues pertaining to identity formation are being confronted which would have implications for personality and cognitive developments. These environmental, personality, and cognitive developments could have played a role in influencing the impact of defense on distress outcomes.
Several limitations should be noted before drawing conclusions. First, the use of self-reported measures might have enhanced social desirability and thereby affected the reporting of trauma and well-being measures (e.g., Caputo, 2017). Second, although a mediational relationship was examined in this cross-sectional study, causality between the psychological constructs should be interpreted with caution. We have softened the language by focusing on direct and indirect effects (i.e., structural relationships) rather than causality inference. Nevertheless, our results have set a solid foundation for a future longitudinal study. Third, this study was based on retrospective data, the passage of time and distance from past trauma exposure may have blurred the reporting of PTSD. Fourth, our participants were composed of university students recruited using a convenience sampling method. As a result, this has cast doubt on the generalizability of findings. Other populations will need to be included in future studies. Last but not least, other unobserved variables would have been involved in explaining the mediating effect of current findings. For example, given the low desirability of presenting themselves as traumatized individuals through self-reporting, students might have engaged in a form of self-deceptive process which has been correlated with internal control beliefs and mental health conditions (e.g., Caputo, 2019a). Moreover, the current model mainly focused on “unconscious” factors as defense mechanism and alexithymia, it has been suggested that overwhelming affective stimulation may also lead to a disorganization of thought process. As such, cognitive schemas should be included in the relationship between PTSD from past trauma and distress for a more comprehensive model (Fang and Chung, 2018, 2019). Despite these limitations, the original contribution of this study is the empirical examination of the psychodynamic mechanisms (i.e., defensive organization and alexithymia) underpinning the relationship between PTSD and distress outcomes among university students. Those with PTSD who have alexithymic symptoms and use immature defenses may develop anxiety and thereby affect perceived quality of life (e.g., Martino et al., 2019b).
The present findings have some important clinical implications. First, screening for PTSD symptoms, alexithymia, and defense mechanisms followed by appropriate counseling is paramount for university students. Second, based on our findings, further studies need to be designed to investigate the effectiveness of psychodynamic treatments for PTSD and trauma among university students. These interventions should aim to empower students know more about their defense styles, explore their inner feelings, and get in touch with their internal self (Briere, 1996). The results of these studies will add to existing empirical and clinical evidence suggesting that psychodynamic approaches may increase the usage of mature defenses with concomitant decreased reliance on immature defenses in various trauma victims (e.g., Schottenbauer et al., 2008). Psychodynamic treatments could also be compared with non-invasive brain stimulation techniques for reducing trauma related anxiety. Such comparison might be of interest for scholars from the neuroscience community (Vicario et al., 2019).
Conclusion
To conclude, following exposure to trauma, victims can develop PTSD affecting mental health. The severity of psychological distress was influenced by the degree of immature defenses used and their ability to identify, describe, and process distressing emotions.
Data Availability Statement
The datasets generated for this study are available on request to the corresponding author.
Ethics Statement
The studies involving human participants were reviewed and approved by the Survey and Behavioral Committee of the Chinese University of Hong Kong. The patients/participants provided their written informed consent to participate in this study. The animal study was reviewed and approved by Survey and Behavioral Research Ethics Committee of the Chinese University of Hong Kong.
Author Contributions
The authors made equally important contributions to the manuscript.
Conflict of Interest
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
References
Ahrens, S., and Deffner, G. (1986). Empirical study of alexithymia: methodology and results. Am. J. Psychother. 40, 430–447. doi: 10.1176/appi.psychotherapy.1986.40.3.430
Akkerman, K., Carr, V., and Lewin, T. (1992). Changes in ego defenses with recovery from depression. J. Nerv. Ment. Dis. 180, 634–638. doi: 10.1097/00005053-199210000-00004
Amstadter, A. B., and Vernon, L. L. (2008). A preliminary examination of thought suppression, emotion regulation, and coping in a trauma-exposed sample. J. Aggress. Maltreat. Trauma 17, 279–295. doi: 10.1080/10926770802403236
Andrews, G., Singh, M., and Bond, M. (1993). The defense style questionnaire. J. Nerv. Ment. Dis. 181, 246–256.
APA (2013). Diagnostic and Statistical Manual of Mental Disorders. Arlington: American Psychiatric Publising.
Atwoli, L., Stein, D. J., Koenen, K. C., and McLaughlin, K. A. (2015). Epidemiology of posttraumatic stress disorder: prevalence, correlates and consequences. Curr. Opin. Psychiatry 28:307. doi: 10.1097/YCO.0000000000000167
Bagby, R. M., Parker, J. D. A., and Taylor, G. J. (1994). The twenty- item toronto alexithymia scale—I. Item selection and cross-validation of the factor structure. J. Psychosom. Res. 38, 23–32. doi: 10.1016/0022-3999(94)90005-1
Bagby, R. M., and Taylor, G. J. (1997). “Affect dysregulation and alexithymia,” in Disorders of Affect Regulation: Alexithymia in Medical And Psychiatric Illness, eds G. J. Taylor, R. M. Bagby, and J. D. A. Parker (Cambrige: Cambridge University Press), 26–45.
Benjet, C., Bromet, E., Karam, E., Kessler, R., McLaughlin, K., Ruscio, A., et al. (2016). The epidemiology of traumatic event exposure worldwide: results from the World mental health survey consortium. Psychol. Med. 46, 327–343. doi: 10.1017/S0033291715001981
Beresford, T. P. (2012). Psychological Adaptive Mechanisms: Ego Defense Recognition in Practice and Research. New York, NY: Oxford University Press.
Bernat, J. A., Ronfeldt, H. M., Calhoun, K. S., and Arias, I. (1998). Prevalence of traumatic events and peritraumatic predictors of posttraumatic stress symptoms in a nonclinical sample of college students. J. Traum. Stress 11, 645–664. doi: 10.1023/A:1024485130934
Besharat, M. A., and Shahidi, S. (2011). What is the relationship between alexithymia and ego defense styles? A correlational study with Iranian students. Asian J. Psychiatry 4, 145–149. doi: 10.1016/j.ajp.2011.05.011
Bogutyn, T., Kokoszka, A., Palczynski, J., and Holas, P. (1999). Defense mechanisms in alexithymia. Psychol. Rep. 84, 183–187. doi: 10.2466/pr0.1999.84.1.183
Bonanno, G. A., and Mancini, A. D. (2012). Beyond resilience and PTSD: mapping the heterogeneity of responses to potential trauma. Psychol. Trauma 4, 74–83. doi: 10.1037/a0017829
Bond, M. (1986). “Defense style questionnaire,” in Empirical Studies Of Ego Mechanisms Of Defense, ed. G. E. Vaillant (Washington, DC: American Psychiatric Press).
Bond, M. (2004). Empirical studies of defense style: relationships with psychopathology and change. Harvard Rev. Psychiatry 12, 263–278. doi: 10.1080/10673220490886167
Breslau, N. (2002). Epidemiologic Studies Of Trauma Posttraumatic Stress Disorder And Other Psychiatric Disorders. Los Angeles: Sage Publications.
Briere, J. (1996). “A self-trauma model for treating adult survivors of severe child abuse,” in The APSAC Handbook on Child Maltreatment, 2nd Edn, eds J. Briere, L. Berliner, J. A. Bulkley, C. Jenny, and T. Reid (Newbury Park, CA: Sage Publications), 140–157.
Briere, J. (2004). Psychological Assessment Of Adult Posttraumatic States : Phenomenology Diagnosis And Measurement. Washington, DC: American Psychological Association.
Caputo, A. (2017). Social desirability bias in self-reported well-being measures: evidence from an online survey. Univer. Psychol. 16, 245–255.
Caputo, A. (2019a). Deceptive dynamics in drug addiction and their role in control beliefs and health status reporting: a study on people with substance use disorder in treatment. J. Drug Issues 49, 575–592.
Caputo, A. (2019b). Psychodynamic insights from narratives of people with amyotrophic lateral sclerosis: a qualitative phenomenological study. Mediterran. J. Clin. Psychol. 7:2.
Caputo, A., Fregonese, C., and Langher, V. (2019). Development and validation of the Dynamic Career Scale (DCS): a psychodynamic conceptualization of career adjustment. Intern. J. Educ. Vocat. Guid. 2, 1–30.
Chen, Z. S., and Chung, M. C. (2016). The relationship between gender, posttraumatic stress disorder from past trauma, alexithymia and psychiatric co-morbidity in Chinese adolescents: a moderated mediational analysis. Psychiatr. Q. 87, 1–13. doi: 10.1007/s11126-016-9419-1
Chung, M. C., Allen, R. D., and Dennis, I. (2013). The impact of self-efficacy, alexithymia and multiple traumas on posttraumatic stress disorder and psychiatric co-morbidity following epileptic seizures: a moderated mediation analysis. Psychiatry Res. 210, 1033–1041. doi: 10.1016/j.psychres.2013.07.041
Chung, M. C., Di, X., and Wan, K. H. (2016). Past trauma, alexithymia, and posttraumatic stress among perpetrators of violent crime. Traumatology 22, 104–112.
Chung, M. C., and Hunt, L. J. (2014). Posttraumatic stress symptoms and well-being following relationship dissolution: past trauma, alexithymia, suppression. Psychiatric Q. 85, 155–176. doi: 10.1007/s11126-013-9280-4
Chung, M. C., Rudd, H., and Wall, N. (2012). Posttraumatic stress disorder following asthma attack (post-asthma attack PTSD) and psychiatric co-morbidity: the impact of alexithymia and coping. Psychiatry Res. 197, 246–252. doi: 10.1016/j.psychres.2012.01.008
Chung, M. C., and Wall, N. (2013). Alexithymia and posttraumatic stress disorder following asthma attack. Psychiatric Q. 84, 287–302. doi: 10.1007/s11126-012-9244-0
Corruble, E., Bronnec, M., Falissard, B., and Hardy, P. (2004). Defense styles in depressed suicide attempters. Psychiatry Clin. Neurosci. 58, 285–288. doi: 10.1111/j.1440-1819.2004.01233.x
Davidson, J., Hughes, D., Blazer, D. G., and George, L. K. (1991). Post-traumatic stress disorder in the community: an epidemiological study. Psychol. Med. 21, 713–721. doi: 10.1017/s0033291700022352
Declercq, F., Vanheule, S., and Deheegher, J. (2010). Alexithymia and posttraumatic stress: subscales and symptom clusters. J. Clin. Psychol. 66, 1076–1089. doi: 10.1002/jclp.20715
Diehl, M., Chui, H., Hay, E. L., Lumley, M. A., Grühn, D., and Labouvie-Vief, G. (2014). Change in coping and defense mechanisms across adulthood: longitudinal findings in a European American sample. Dev. Psychol. 50, 634–648. doi: 10.1037/a0033619
Eglinton, R., and Chung, M. C. (2011). The relationship between posttraumatic stress disorder, illness cognitions, defence styles, fatigue severity and psychological well-being in chronic fatigue syndrome. Psychiatry Res. 188, 245–252. doi: 10.1016/j.psychres.2011.04.012
Endler, N. S. (1983). Interactionism: a personality model but not yet a theory. Nebr. Symp. Motiv. 1983, 155–200.
Espié, E., Gaboulaud, V., Baubet, T., Casas, G., Mouchenik, Y., Yun, O., et al. (2009). Trauma-related psychological disorders among palestinian children and adults in Gaza and West Bank, 2005-2008. Intern. J. Ment. Health Syst. 3:21. doi: 10.1186/1752-4458-3-21
Evren, C., Cagil, D., Ulku, M., Ozcetinkaya, S., Gokalp, P., Cetin, T., et al. (2012). Relationship between defense styles, alexithymia, and personality in alcohol-dependent inpatients. Comprehens. Psychiatry 53, 860–867. doi: 10.1016/j.comppsych.2012.01.002
Fang, S., and Chung, M. C. (2018). “The impact of past trauma on psychological distress: the role of alexithymia,” in Current Developments in Alexithymia: A Cognitive and Affective Deficits, eds R. J. Teixeira, B. Bermond, and P. P. Moormann (New York, NY: Nova Science Publishers, Inc).
Fang, S., and Chung, M. C. (2019). The impact of past trauma on psychological distress among Chinese students: the roles of cognitive distortion and alexithymia. Psychiatry Res. 271, 136–143. doi: 10.1016/j.psychres.2018.11.032
Finzi-Dottan, R., and Karu, T. (2006). From emotional abuse in childhood to psychopathology in adulthood: a path mediated by immature defense mechanisms and self-esteem. J. Nerv. Ment. Dis. 194, 616–621. doi: 10.1097/01.nmd.0000230654.49933.23
Flett, G. L., Besser, A., and Hewitt, P. L. (2005). Perfectionism, ego defense styles, and depression: a comparison of self-reports versus informant ratings. J. Pers. 73, 1355–1396. doi: 10.1111/j.1467-6494.2005.00352.x
Foa, E. B., Steketee, G., and Rothbaum, B. O. (1989). Behavioral/cognitive conceptualizations of post-traumatic stress disorder. Behav. Ther. 20, 155–176. doi: 10.1016/S0005-7894(89)80067-X
Frewen, P. A., Dozois, D. J., Neufeld, R. W., and Lanius, R. A. (2008). Meta-analysis of alexithymia in posttraumatic stress disorder. J. Traum. Stress 21, 243–246. doi: 10.1002/jts.20320
Goldberg, D., and Hillier, V. F. (1979). A scaled version of the general health questionnaire. Psychol. Med. 9, 139–145. doi: 10.1017/S0033291700021644
Gross, J. J. (1998). The emerging field of emotion regulation: an integrative review. Rev. Gen. Psychol. 2, 271–299. doi: 10.1037/1089-2680.2.3.271
Hayes, A. F. (2016). Introduction to Mediation Moderation and Conditional Process Analysis: A Regression-Based Approach. New York, NY: The Guilford Press.
Heim, C., and Nemeroff, C. B. (2001). The role of childhood trauma in the neurobiology of mood and anxiety disorders: preclinical and clinical studies. Biol. Psychiatry 49, 1023–1039. doi: 10.1016/s0006-3223(01)01157-x
Helmes, E., McNeill, P. D., Holden, R. R., and Jackson, C. (2008). The construct of alexithymia: associations with defense mechanisms. J. Clin. Psychol. 64, 318–331. doi: 10.1002/jclp.20461
Hill, J. (2003). Childhood trauma and depression. Curr. Opin. Psychiatry 16, 3–6. doi: 10.1097/01.yco.0000049394.00317.77
Honkalampi, K., Hintikka, J., Tanskanen, A., Lehtonen, J., and Viinamäki, H. (2000). Depression is strongly associated with alexithymia in the general population. J. Psychosom. Res. 48, 99–104. doi: 10.1016/S0022-3999(99)00083-5
Horowitz, M. J. (1986). Stress-response syndromes: a review of posttraumatic and adjustment disorders. Hosp. Commun. Psychiatry 37, 241–249. doi: 10.1176/ps.37.3.241
Horowitz, M. J., Wilner, N., and Alvarez, W. (1979). Impact of event scale: a measure of subjective stress. Psychosom. Med. 41, 209–218. doi: 10.1097/00006842-197905000-00004
Hu, L.-T., and Bentler, P. M. (1998). Fit indices in covariance structure modeling: sensitivity to underparameterized model misspecification. Psychol. Methods 3:424.
Jun, J. Y., Lee, Y.-J. G., Lee, S.-H., Yoo, S. Y., Song, J., and Kim, S. J. (2015). Association between defense mechanisms and psychiatric symptoms in North Korean Refugees. Comprehens. Psychiatry 56, 179–187. doi: 10.1016/j.comppsych.2014.10.001
Karukivi, M., Hautala, L., Kaleva, O., Haapasalo-Pesu, K.-M., Liuksila, P.-R., Joukamaa, M., et al. (2010). Alexithymia is associated with anxiety among adolescents. J. Affect. Disord. 125, 383–387.
Keane, T. M., Marshall, A. D., and Taft, C. T. (2006). Posttraumatic stress disorder: etiology, epidemiology, and treatment outcome. Annu. Rev. Clin. Psychol. 2, 161–197. doi: 10.1146/annurev.clinpsy.2.022305.095305
Kennedy, B. L., Schwab, J. J., and Hyde, J. A. (2001). Defense styles and personality dimensions of research subjects with anxiety and depressive disorders. Psychiatric Q. 72, 251–262. doi: 10.1023/a:1010353116016
Kessler, R. C., Berglund, P., Demler, O., Jin, R., Merikangas, K. R., and Walters, E. E. (2005). Lifetime prevalence and age-of-onset distributions of DSM-IV disorders in the national comorbidity survey replication. Archiv. Gen. Psychiatry 62, 593–602. doi: 10.1001/archpsyc.62.6.593
Kessler, R. C., Sonnega, A., Bromet, E., Hughes, M., and Nelson, C. B. (1995). Posttraumatic stress disorder in the national comorbidity survey. Archiv. Gen. Psychiatry 52, 1048–1060. doi: 10.1001/archpsyc.1995.03950240066012
Kilpatrick, D. G., Resnick, H. S., Milanak, M. E., Miller, M. W., Keyes, K. M., and Friedman, M. J. (2013). National estimates of exposure to traumatic events and PTSD prevalence using DSM-IV and DSM-5 criteria. J. Traum. Stress 26, 537–547. doi: 10.1002/jts.21848
Kooiman, C., Spinhoven, P., Trijsburg, R., and Rooijmans, H. (1998). Perceived parental attitude, alexithymia and defense style in psychiatric outpatients. Psychother. Psychosom. 67, 81–87. doi: 10.1159/000012264
Kupchik, M., Strous, R. D., Erez, R., Gonen, N., Weizman, A., and Spivak, B. (2007). Demographic and clinical characteristics of motor vehicle accident victims in the community general health outpatient clinic: a comparison of PTSD and non PTSD subjects. Depress. Anxiety 24, 244–250. doi: 10.1002/da.20189
Kwon, P., and Lemon, K. E. (2000). Attributional style and defense mechanisms: a synthesis of cognitive and psychodynamic factors in depression. J. Clin. Psychol. 56, 723–735. doi: 10.1002/(sici)1097-4679(200006)56:6<723::aid-jclp3>3.0.co;2-3
Langher, V., Marchini, F., Brandimarte, D., Giacchetta, A., and Caputo, A. (2019). Trauma e riparazione in una comunità colpita dal sisma: una lettura clinica dinamica per l’intervento post-emergenziale. Psicol. Della Salute 24, 98–121.
Lumley, M. A., Neely, L. C., and Burger, A. J. (2007). The assessment of alexithymia in medical settings: implications for understanding and treating health problems. J. Pers. Assess. 89, 230–246. doi: 10.1080/00223890701629698
Macdonald, A., Greene, C. J., Torres, J. G., Frueh, B. C., and Morland, L. A. (2013). Concordance between clinician-assessed and self-reported symptoms of posttraumatic stress disorder across three ethnoracial groups. Psychol. Trauma 5:401.
MacKinnon, D. P., Lockwood, C. M., and Williams, J. (2004). Confidence limits for the indirect effect: Distribution of the product and resampling methods. Multivar. Behav. Res. 39, 99–128. doi: 10.1207/s15327906mbr3901_4
Marchini, F., Caputo, A., Napoli, A., Balonan, J. T., Martino, G., Nannini, V., et al. (2018). Chronic illness as loss of good self: underlying mechanisms affecting diabetes adaptation. Mediterran. J. Clin. Psychol. 6:1981.
Marsella, A. J., Friedman, M. J., Gerrity, E. T., and Scurfield, R. M. (1996). “Ethnocultural aspects of PTSD: Some closing thoughts,” in Ethnocultural Aspects Of Posttraumatic Stress Disorder: Issues Research And Clinical Applications, eds A. J. Marsella, M. J. Friedman, E. T. Gerrity, and R. M. Scurfield (New York, NY: American Psychological Association), 529–538.
Martino, G., Bellone, F., Langher, V., Caputo, A., Catalano, A., Quattropani, M. C., et al. (2019a). Alexithymia and psychological distress affect perceived quality of life in patients with type 2 diabetes mellitus. Mediterran. J. Clin. Psychol. 7:81.
Martino, G., Catalano, A., Bellone, F., Russo, G. T., Vicario, C. M., Lasco, A., et al. (2019b). As time goes by: anxiety negatively affects the perceived quality of life in patients with type 2 diabetes of long duration. Front. Psychol. 10:1779. doi: 10.3389/fpsyg.2019.01779
Martino, G., Caputo, A., Bellone, F., Quattropani, M. C., and Vicario, C. M. (2020). Going beyond the visible in type 2 diabetes mellitus: defense mechanisms and their associations with depression and health-related quality of life. Front. Psychol. 11:267. doi: 10.3389/fpsyg.2019.0267
Mazzeo, S. E., and Espelage, D. L. (2002). Association between childhood physical and emotional abuse and disordered eating behaviors in female undergraduates: an investigation of the mediating role of alexithymia and depression. J. Counsel. Psychol. 49, 86–100.
Mazzeo, S. E., Mitchell, K. S., and Williams, L. J. (2008). Anxiety, alexithymia, and depression as mediators of the association between childhood abuse and eating disordered behavior in African American and European American women. Psychol. Women Q. 32, 267–280.
Meganck, R., Vanheule, S., and Desmet, M. (2013). Affective processing and affect regulation: A clinical interview study. J. Am. Psychoanal. Assoc. 61, N12–N16. doi: 10.1177/0003065113516365
Miller, M. W., Kaloupek, D. G., Dillon, A. L., and Keane, T. M. (2004). Externalizing and internalizing subtypes of combat-related PTSD: a replication and extension using the PSY-5 scales. J. Abnorm. Psychol. 113, 636–645. doi: 10.1037/0021-843X.113.4.636
Nemiah, J. C., and Sifneos, P. E. (1970). Psychosomatic illness: a problem in communication. Psychother. Psychosom. 18, 154–160. doi: 10.1159/000286074
Nickel, R., and Egle, U. T. (2006). Psychological defense styles, childhood adversities and psychopathology in adulthood. Child Abuse Neglect. 30, 157–170. doi: 10.1016/j.chiabu.2005.08.016
Norris, F. H. (1992). Epidemiology of trauma: frequency and impact of different potentially traumatic events on different demographic groups. J. Consult. Clin. Psychol. 60:409. doi: 10.1037//0022-006x.60.3.409
Norris, F. H., Perilla, J. L., Ibañez, G. E., and Murphy, A. D. (2001). Sex differences in symptoms of posttraumatic stress: does culture play a role? J. Traum. Stress 14, 7–28. doi: 10.1023/A:1007851413867
Parker, J. D. A., Taylor, G. J., and Bagby, R. M. (1998). Alexithymia: relationship with ego defense and coping styles. Comprehens. Psychiatry 39, 91–98. doi: 10.1016/S0010-440X(98)90084-0
Pennebaker, J. W., and Beall, S. K. (1986). Confronting a traumatic event: toward an understanding of inhibition and disease. J. Abnorm. Psychol. 95:274. doi: 10.1037//0021-843x.95.3.274
Sayar, K., Kirmayer, L. J., and Taillefer, S. S. (2003). Predictors of somatic symptoms in depressive disorder. Gen. Hosp. Psychiatry 25, 108–114. doi: 10.1016/S0163-8343(02)00277-3
Schafer, J. L., and Graham, J. W. (2002). Missing data: our view of the state of the art. Psychol. Methods 7, 147–177.
Schottenbauer, M. A., Glass, C. R., Arnkoff, D. B., and Gray, S. H. (2008). Contributions of psychodynamic approaches to treatment of PTSD and trauma: a review of the empirical treatment and psychopathology literature. Psychiatry Interpers. Biol. Process. 71, 13–34. doi: 10.1521/psyc.2008.71.1.13
Silverstein, R. (1996). Combat-related trauma as measured by ego developmental indices of defenses and identity achievement. J. Genet. Psychol. 157, 169–179. doi: 10.1080/00221325.1996.9914855
Sinha, B. K., and Watson, D. C. (1999). Predicting personality disorder traits with the Defense Style Questionnaire in a normal sample. J. Pers. Disord. 13, 281–286. doi: 10.1521/pedi.1999.13.3.281
Smyth, J. M., Hockemeyer, J. R., Heron, K. E., Wonderlich, S. A., and Pennebaker, J. W. (2008). Prevalence, type, disclosure, and severity of adverse life events in college students. J. Am. Coll. Health 57, 69–76. doi: 10.3200/JACH.57.1.69-76
Sterling, M. (2011). General health questionnaire – 28 (GHQ-28). J. Physiother. 57:259. doi: 10.1016/S1836-9553(11)70060-1
Taylor, G. J., Bagby, R. M., and Parker, J. D. (1991). The alexithymia construct: a potential paradigm for psychosomatic medicine. Psychosomatics 32, 153–164. doi: 10.1016/S0033-3182(91)72086-0
Taylor, G. J., Bagby, R. M., and Parker, J. D. (1997). Disorders of Affect Regulation: Alexithymia in Medical and Psychiatric Illness. Cambridge, England: Cambridge University Press.
Vaillant, G. E. (1971). Theoretical hierarchy of adaptive ego mechanisms: a 30-year follow-up of 30 men selected for psychological health. Archiv. Gen. Psychiatry 24, 107–118. doi: 10.1001/archpsyc.1971.01750080011003
Vaillant, G. E. (1992). Ego Mechanisms Of Defense: A Guide For Clinicans And Researchers. Washington, DC: American Psychiatric Pub.
Vaillant, G. E. (2011). Involuntary coping mechanisms: a psychodynamic perspective. Dialog. Clin. Neurosci. 13:366.
Van der Kolk, B. A., and Ducey, C. P. (1989). The psychological processing of traumatic experience: rorschach patterns in PTSD. J. Traum. Stress 2, 259–274.
Verhaeghe, P., and Vanheule, S. (2005). Actual neurosis and PTSD: the impact of the other. Psychoanal. Psychol. 22, 493–507. doi: 10.1037/0736-9735.22.4.493
Vicario, C., Salehinejad, M. A., Felmingham, K., Martino, G., and Nitsche, M. (2019). A systematic review on the therapeutic effectiveness of non-invasive brain stimulation for the treatment of anxiety disorders. Neurosci. Biobehav. Rev. 96, 219–231. doi: 10.1016/j.neubiorev.2018.12.012
Vrana, S., and Lauterbach, D. (1994). Prevalence of traumatic events and posttraumatic psychological symptoms in a nonclinical sample of college students. J. Traum. Stress 7, 289–302. doi: 10.1002/jts.2490070209
Walburg, V., and Chiaramello, S. (2015). Link between early maladaptive schemas and defense mechanisms. Eur. Rev. Appl. Psychol. 65, 221–226.
Watson, D. C. (2002). Predicting psychiatric symptomatology with the defense style questionnaire-40. Intern. J. Stress Manag. 9, 275–287.
Weathers, F. W., Litz, B. T., Keane, T. M., Palmieri, P., Marx, B., and Schnurr, P. (2013). The PTSD Checklist for DSM-5 (PCL-5). Boston MA: National Center for PTSD.
Wise, T. N., Mann, L. S., and Epstein, S. (1991). Ego defensive styles and alexithymia: a discriminant validation study. Psychother. Psychosom. 56, 141–145. doi: 10.1159/000288547
Wise, T. N., Mann, L. S., and Hill, B. (1990). Alexithymia and depressed mood in the psychiatric patient. Psychother. Psychosom. 54, 26–31. doi: 10.1159/000288373
Keywords: past trauma, posttraumatic stress disorder, alexithymia, defense mechanisms, psychological distress
Citation: Fang S, Chung MC and Wang Y (2020) The Impact of Past Trauma on Psychological Distress: The Roles of Defense Mechanisms and Alexithymia. Front. Psychol. 11:992. doi: 10.3389/fpsyg.2020.00992
Received: 06 January 2020; Accepted: 21 April 2020;
Published: 21 May 2020.
Edited by:
Gabriella Martino, University of Messina, ItalyReviewed by:
Andrea Caputo, Sapienza University of Rome, ItalySzabolcs Keri, University of Szeged, Hungary
Copyright © 2020 Fang, Chung and Wang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Yabing Wang, yabing.wang@link.cuhk.edu.hk