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REVIEW article
Front. Cell. Infect. Microbiol.
Sec. Extra-intestinal Microbiome
Volume 14 - 2024 |
doi: 10.3389/fcimb.2024.1518811
This article is part of the Research Topic Skin Microbiome: Microbiological, Immunological and Cellular aspects for therapies to control Antimicrobial Resistance and Skin Repair View all articles
Skin microbiota: Pathogenic roles and implications in atopic dermatitis
Provisionally accepted- 1 Department of Dermatology, Peking University Shenzhen Hospital, Shenzhen CN, China
- 2 Department of Obstetrics and Gynecology, Peking University Shenzhen Hospital, Shenzhen CN, China
Atopic dermatitis (AD) is a chronic and inflammatory skin disorder characterized by impaired barrier function and imbalanced immunity. Recent advances have revealed that dysbiosis of skin microbiota plays important roles in the pathogenesis and development of AD. Meanwhile, endogenous and external factors contribute to the dysbiosis of skin microbiota in AD. Additionally, various treatments, including topical treatments, phototherapy, and systemic biologics, have demonstrated positive impacts on the clinical outcomes, alongside with the modulations of cutaneous microbiota in AD patients. Importantly, therapeutics or products regulating skin microbiota homeostasis have demonstrated potential for AD treatment in early clinical studies. In this review, we underline changes of the skin microbiota correlated with AD. Meanwhile, we provide an overview of the skin microbiota regarding its roles in the 2 pathogenesis and development of AD. Finally, we summarize therapeutic strategies restoring the skin microbial homeostasis in AD management.
Keywords: atopic dermatitis, Skin microbiota, Staphylococcus aureus, Pathogenesis, Microbiota-based therapy
Received: 30 Oct 2024; Accepted: 27 Dec 2024.
Copyright: © 2024 Huang, Zhuo, Guo, Wang, Zhang, Li, Dai, Dou and Yu. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
* Correspondence:
Cong Huang, Department of Dermatology, Peking University Shenzhen Hospital, Shenzhen CN, China
Xia Dou, Department of Dermatology, Peking University Shenzhen Hospital, Shenzhen CN, China
Bo Yu, Department of Dermatology, Peking University Shenzhen Hospital, Shenzhen CN, China
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