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MINI REVIEW article
Front. Cardiovasc. Med.
Sec. Lipids in Cardiovascular Disease
Volume 11 - 2024 |
doi: 10.3389/fcvm.2024.1510148
This article is part of the Research Topic Evidence of Atherogenic Lipoproteins: what we gain from in vitro and in vivo research View all 10 articles
Frontiers in Cardiovascular Medicine-Collection: "Evidence of Atherogenic Lipoproteins: what we gain from in vitro and in vivo research" Mini-Review Bile acids and incretins as modulators of obesity-associated atherosclerosis
Provisionally accepted- Division of Physiology and Pathophysiology, Otto Loewi Research Center, Medical University of Graz, Graz, Austria
Obesity is one of the major global health concerns of the 21 st century, associated with many comorbidities such as type 2 diabetes mellitus (T2DM), metabolic associated steatotic liver disease and early and aggressive atherosclerotic cardiovascular disease, which is the leading cause of death worldwide. Bile acids (BAs) and incretins are gut hormones involved in digestion and absorption of fatty acids, and insulin secretion, respectively. In recent years BAs and incretins are increasingly recognized as key signaling molecules, which target multiple tissues and organs, beyond the gastro-intestinal system. Moreover, incretin-based therapy has revolutionized the treatment of T2DM and obesity. This mini review highlights the current knowledge about dysregulations in BA homeostasis in obesity with a special focus on atherosclerosis as well as athero-modulating roles of incretins and currently available incretinbased therapies.
Keywords: cardiovascular disease, Obesity, gut hormones, Lipoproteins, Atherosclerosis
Received: 12 Oct 2024; Accepted: 17 Dec 2024.
Copyright: © 2024 Kirsch, Gindlhuber, Zabini and Osto. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
* Correspondence:
Elena Osto, Division of Physiology and Pathophysiology, Otto Loewi Research Center, Medical University of Graz, Graz, Austria
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