Negatively Regulated by miR-29c-3p, MTFR1 Promotes the Progression and Glycolysis in Lung Adenocarcinoma via the AMPK/mTOR Signalling Pathway
- 1Department of Thoracic Surgery, Qilu Hospital, Cheeloo College of Medicine, Shandong University, Jinan, China
- 2Department of Anesthesiology, Qilu Children’s Hospital of Shandong University, Jinan, China
A Corrigendum on
Negatively regulated by miR-29c-3p, MTFR1 promotes the progression and glycolysis in lung adenocarcinoma via the AMPK/mTOR signalling pathway
by Li Y, Liu Y, Jin K, Dong R, Gao C, Si L, Feng Z, Zhang H and Tian H (2021). Front. Cell Dev. Biol. 9:771824. doi: 10.3389/fcell.2021.771824
In the original article, there was a mistake in Figure 9D as published. When we were arranging the panel, we misused the HE staining pictures by using the wrong folder. The corrected Figure 9D appears below.
FIGURE 9. MTFR1 promoted the progression of LUAD cells in vivo. (A,B) Tumour images, tumour growth curves and tumour weights of each group. (C) Haematoxylin–eosin (HE) staining and immunohistochemical analysis of the Ki-67 and MTFR1 proteins of tumours in each group. (D) Photographs (left) and HE-stained images (right) of the lungs in each group; the black arrowheads denote lung metastasis nodules (**p < 0.01, ***p < 0.001, ****p < 0.0001).
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: lung cancer, microRNA, mitochondrial fission regulator 1, warburg effect, proliferation, invasion, migration
Citation: Li Y, Liu Y, Jin K, Dong R, Gao C, Si L, Feng Z, Zhang H and Tian H (2022) Corrigendum: Negatively regulated by miR-29c-3p, MTFR1 promotes the progression and glycolysis in lung adenocarcinoma via the AMPK/mTOR signalling pathway. Front. Cell Dev. Biol. 10:870313. doi: 10.3389/fcell.2022.870313
Received: 06 February 2022; Accepted: 09 August 2022;
Published: 31 August 2022.
Edited and reviewed by:
Jiayi Wang, Shanghai Jiao Tong University, ChinaCopyright © 2022 Li, Liu, Jin, Dong, Gao, Si, Feng, Zhang and Tian. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Hui Tian, tianhuiql@126.com