In order to promote the sustainable development of aquaculture, it is of great importance to better understand fish diseases caused by classic and emerging bacterial pathogens. Strains of classic fish pathogens such as Aeromonas, Photobacterium, Vibrio, Yersinia, Edwardsiella, Flavobacterium, Piscirickettsia, Streptococcus, and Lactococcus have been joined by other emerging bacterial species or new strains of Moritella, Chlamydia, Tenacibaculum, Hafnia, Erysipelothrix, Mycobacterium, Renibacterium, and Weissella, that have begun to cause mortalities of important fish in some parts of the world.
Studies of these fish diseases should focus on better understanding the basis of the interaction between pathogens and their host, both at a cellular and molecular level, with the aim of creating new tools to combat these pathogens. While Cellular Microbiology has grown exponentially in the study of animal and human bacterial pathogens, the study of the interaction of fish bacterial pathogens with cells has made virtually no progress. This is partly due to the lack of well-characterized fish cell lines, and poor homogeneity in studies with infection models.
In this Research Topic, we welcome authors to contribute high-quality Original Research articles as well as Review articles, Mini-reviews, Methods, and Perspectives that include but are not limited to how classic and emerging fish pathogens interact with host cells. Advancing knowledge about this area of research should also encourage collaborations between bacteriologists, veterinarians, immunologists, and cell biologists.
We welcome authors to contribute with articles on these specific topics of interest:
· Use of fish cell lines to study host-bacteria interactions
· Immune responses and immune evasion of bacterial fish pathogens
· Interaction between cells, probiotics and bacterial pathogens
· Molecular mechanisms of host-bacteria interactions during fish infections
· Comparative genome analysis for a better understanding of pathogenic mechanisms and their evolution.
In order to promote the sustainable development of aquaculture, it is of great importance to better understand fish diseases caused by classic and emerging bacterial pathogens. Strains of classic fish pathogens such as Aeromonas, Photobacterium, Vibrio, Yersinia, Edwardsiella, Flavobacterium, Piscirickettsia, Streptococcus, and Lactococcus have been joined by other emerging bacterial species or new strains of Moritella, Chlamydia, Tenacibaculum, Hafnia, Erysipelothrix, Mycobacterium, Renibacterium, and Weissella, that have begun to cause mortalities of important fish in some parts of the world.
Studies of these fish diseases should focus on better understanding the basis of the interaction between pathogens and their host, both at a cellular and molecular level, with the aim of creating new tools to combat these pathogens. While Cellular Microbiology has grown exponentially in the study of animal and human bacterial pathogens, the study of the interaction of fish bacterial pathogens with cells has made virtually no progress. This is partly due to the lack of well-characterized fish cell lines, and poor homogeneity in studies with infection models.
In this Research Topic, we welcome authors to contribute high-quality Original Research articles as well as Review articles, Mini-reviews, Methods, and Perspectives that include but are not limited to how classic and emerging fish pathogens interact with host cells. Advancing knowledge about this area of research should also encourage collaborations between bacteriologists, veterinarians, immunologists, and cell biologists.
We welcome authors to contribute with articles on these specific topics of interest:
· Use of fish cell lines to study host-bacteria interactions
· Immune responses and immune evasion of bacterial fish pathogens
· Interaction between cells, probiotics and bacterial pathogens
· Molecular mechanisms of host-bacteria interactions during fish infections
· Comparative genome analysis for a better understanding of pathogenic mechanisms and their evolution.
