Inflammatory pain is one of the most common complaints that can seriously lead to loss of function and reduced life quality. Typically, the tissue damage of inflammatory pain is responsible for activating inflammatory mediators associated with worsening pain. When acute pain develops into chronic pain through central sensitization, inflammatory pain may persist and become chronic pain. The pain-inducing mechanisms in inflammatory conditions have been widely developed; however, the research investigating the possible mechanisms of pain relief is still in its infancy.
The inflammatory cascade is complex, but the cessation of the inflammatory process represents a potential aim to alleviate pain. The management of inflammatory pain includes both pharmacological and non-pharmacological strategies. Further research into exploring the assessment and the mechanisms of pharmacological and non-pharmacological interventions of inflammatory pain is required in the future.
The aim of this Research Topic is to gather recent findings that report the mechanisms, assessment, and intervention of inflammatory pain. We welcome both human studies and animal studies. We encourage studies making use of molecular biology. We are interested in manuscripts underlying mechanisms of all kinds of effective pharmacological and non-pharmacological interventions for inflammatory pain, including medications, exercise, neuromodulations (eg, transcranial magnetic stimulation (TMS), transcranial direct current stimulation (tDCS), etc.), acupuncture, and psychological treatments.
Potential topics include but are not limited to the following:
• Original Research of the mechanisms, assessment, or intervention of inflammatory pain
• Reviews and meta-analysis of the mechanisms, assessment, or intervention of inflammatory pain
• Consensus statement and guidelines of the mechanisms, assessment, or intervention of inflammatory pain
• Peripheral or central neuro-immune signaling and neuroinflammatory mechanisms of inflammatory pain
• Inflammatory mediators/ biomarkers of inflammatory pain
• Microglia, macrophage, proinflammatory or anti-inflammatory cytokines of inflammatory pain
• Studies of one specific intervention for inflammatory pain
• The roles of non-coding RNA (miRNAs, lncRNAs, circRNAs, etc.) in regulating the inflammatory response in inflammatory pain
• The long-term effect of the intervention on inflammatory mechanisms of inflammatory pain
Please note that manuscripts should be submitted through Frontiers in Molecular Neuroscience or Frontiers in Neurology.
Inflammatory pain is one of the most common complaints that can seriously lead to loss of function and reduced life quality. Typically, the tissue damage of inflammatory pain is responsible for activating inflammatory mediators associated with worsening pain. When acute pain develops into chronic pain through central sensitization, inflammatory pain may persist and become chronic pain. The pain-inducing mechanisms in inflammatory conditions have been widely developed; however, the research investigating the possible mechanisms of pain relief is still in its infancy.
The inflammatory cascade is complex, but the cessation of the inflammatory process represents a potential aim to alleviate pain. The management of inflammatory pain includes both pharmacological and non-pharmacological strategies. Further research into exploring the assessment and the mechanisms of pharmacological and non-pharmacological interventions of inflammatory pain is required in the future.
The aim of this Research Topic is to gather recent findings that report the mechanisms, assessment, and intervention of inflammatory pain. We welcome both human studies and animal studies. We encourage studies making use of molecular biology. We are interested in manuscripts underlying mechanisms of all kinds of effective pharmacological and non-pharmacological interventions for inflammatory pain, including medications, exercise, neuromodulations (eg, transcranial magnetic stimulation (TMS), transcranial direct current stimulation (tDCS), etc.), acupuncture, and psychological treatments.
Potential topics include but are not limited to the following:
• Original Research of the mechanisms, assessment, or intervention of inflammatory pain
• Reviews and meta-analysis of the mechanisms, assessment, or intervention of inflammatory pain
• Consensus statement and guidelines of the mechanisms, assessment, or intervention of inflammatory pain
• Peripheral or central neuro-immune signaling and neuroinflammatory mechanisms of inflammatory pain
• Inflammatory mediators/ biomarkers of inflammatory pain
• Microglia, macrophage, proinflammatory or anti-inflammatory cytokines of inflammatory pain
• Studies of one specific intervention for inflammatory pain
• The roles of non-coding RNA (miRNAs, lncRNAs, circRNAs, etc.) in regulating the inflammatory response in inflammatory pain
• The long-term effect of the intervention on inflammatory mechanisms of inflammatory pain
Please note that manuscripts should be submitted through Frontiers in Molecular Neuroscience or Frontiers in Neurology.
