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CORRECTION article

Front. Vet. Sci., 12 August 2024
Sec. Comparative and Clinical Medicine

Corrigendum: Classification of feline hypertrophic cardiomyopathy-associated gene variants according to the American College of Medical Genetics and Genomics guidelines

  • 1Laboratory Animal Genetics, Department of Veterinary and Biosciences, Faculty of Veterinary Medicine, Ghent University, Merelbeke, Belgium
  • 2Univ Lyon, VetAgro Sup, Marcy-l'Etoile, France & Institut NeuroMyoGène INMG-PNMG, CNRS UMR5261, INSERM U1315, Faculté de Médicine, Rockefeller, Université Claude Bernard Lyon 1, Lyon, France
  • 3Wisdom Panel, Mars Petcare Science & Diagnostics, Helsinki, Finland
  • 4Osservatorio Veterinario Italiano Cardiopatie, Azzano San Paolo, Italy
  • 5Bis Clinica Veterinaria Orobica Anicura, Bergamo, Italy
  • 6The Animal Medical Center, New York, NY, United States
  • 7Department of Biomedical Sciences and Cornell Veterinary Biobank, College of Veterinary Medicine, Cornell University, Ithaca, NY, United States
  • 8Department of Clinical Sciences, Faculty of Veterinary Medicine and Animal Science, Swedish University of Agricultural Sciences, Uppsala, Sweden
  • 9Wisdom Panel, Mars Petcare Science & Diagnostics, Leicestershire, United Kingdom
  • 10Veterinary Information Network and School of Veterinary Medicine and Epidemiology, University of California, Davis, Davis, CA, United States
  • 11Department of Clinical Sciences, Faculty of Veterinary Medicine, Utrecht University, Utrecht, Netherlands
  • 12Department of Veterinary Medicine and Surgery, College of Veterinary Medicine, University of Missouri, Columbia, MO, United States
  • 13Department of Veterinary Medicine and Animal Sciences, University of Milan, Lodi, Italy
  • 14Department of Animal Breeding and Genetics, Faculty of Veterinary Medicine and Animal Science, Swedish University of Agricultural Sciences, Uppsala, Sweden
  • 15Antagene, La Tour-de-Salvagny, France
  • 16Small Animal Department, Ghent University, Merelbeke, Belgium
  • 17Genefast srl, Forlì, Italy

A Corrigendum on
Classification of feline hypertrophic cardiomyopathy-associated gene variants according to the American College of Medical Genetics and Genomics guidelines

by Boeykens, F., Abitbol, M., Anderson, H., Dargar, T., Ferrari, P., Fox, P. R., Hayward, J. J., Häggström, J., Davison, S., Kittleson, M. D., van Steenbeek, F., Ljungvall, I., Lyons, L. A., Longeri, M., Ohlsson, Å., Peelman, L., Dufaure de Citres, C., Smets, P., Turba, M. E., and Broeckx, B. J. G. (2024). Front. Vet. Sci. 11:1327081. doi: 10.3389/fvets.2024.1327081

In the published article, there was an error: the wording in the phenotype subsection was imprecise.

A correction has been made to 2 Materials and Methods, 2.5 Phenotyping. These sentences previously stated:

“A left ventricular wall thickness during end-diastole below 5 mm is regarded as within the physiological norm, and a thickness equal to or exceeding 6 mm is indicative of hypertrophy. It is noteworthy that reference ranges, incorporating considerations of body weight, were applied as an additional parameter (1). Cats with a wall thickness measurement between 5.5 and 5.9 mm outside the body weight-based reference interval, and with normal left atrial size, were classified as equivocal.”

The corrected sentences appear below:

“In the realm of feline cardiology, there is no universally accepted standard for determining the normal thickness of the left ventricular wall during diastole. It is overly simplistic to assume that a single numerical value can reliably distinguish between a healthy left ventricular wall and one affected by hypertrophy across all cats. The thickness of the left ventricular wall increases nonlinearly with body weight and is influenced by physiological factors such as hydration level and heart rate. Conditions like hyperthyroidism and systemic hypertension, which are common in older cats, can further impact left ventricular wall thickness, and the presence of these conditions were examined in cats, if suspected, before inclusion.

Nevertheless, for most cats that are not hyperthyroid or hypertensive, of average size and with a normal body condition (weighing between 3.5–5 kg), a left ventricular diastolic wall thickness of 5 mm or less is typically considered within the normal range, while a thickness of 6 mm or more suggests concentric hypertrophy. Cats with a left ventricular wall thickness falling between 5 and 6 mm were termed equivocal and were excluded from analysis. Cats outside this weight range were managed on a case to case basis with the aid of previously published 95% prediction intervals (1).”

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher's note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: cardiac disease, feline genetics, variant classification, ACMG guidelines, genetic diversity

Citation: Boeykens F, Abitbol M, Anderson H, Dargar T, Ferrari P, Fox PR, Hayward JJ, Häggström J, Davison S, Kittleson MD, van Steenbeek F, Ljungvall I, Lyons LA, Longeri M, Ohlsson Å, Peelman L, Dufaure de Citres C, Smets P, Turba ME and Broeckx BJG (2024) Corrigendum: Classification of feline hypertrophic cardiomyopathy-associated gene variants according to the American College of Medical Genetics and Genomics guidelines. Front. Vet. Sci. 11:1458433. doi: 10.3389/fvets.2024.1458433

Received: 02 July 2024; Accepted: 26 July 2024;
Published: 12 August 2024.

Edited and reviewed by: Carlo Guglielmini, University Hospital of Padua, Italy

Copyright © 2024 Boeykens, Abitbol, Anderson, Dargar, Ferrari, Fox, Hayward, Häggström, Davison, Kittleson, van Steenbeek, Ljungvall, Lyons, Longeri, Ohlsson, Peelman, Dufaure de Citres, Smets, Turba and Broeckx. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Fréderique Boeykens, frederique.boeykens@ugent.be; Bart J. G. Broeckx, Bart.broeckx@ugent.be

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.