A small molecule inhibitor of Notch1 modulates stemness and suppresses breast cancer cell growth
- 1Texas A&M University, College Station, TX, United States
- 2Penn State Cancer Institute, College of Medicine, The Pennsylvania State University, Hershey, PA, United States
A Corrigendum on
A small molecule inhibitor of Notch1 modulates stemness and suppresses breast cancer cell growth
by Saran U, Chandrasekaran B, Tyagi A, Shukla V, Singh A, Sharma AK and Damodaran C (2023). Front. Pharmacol. 14:1150774. doi: 10.3389/fphar.2023.1150774
In the published article, there was an error in Supplementary Data Sheet S1. The western blot image for Figure 5A did not match the corresponding full western blot image in Data Sheet 1. The correct Western blot image for Figure 5A has been updated.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: breast cancer, breast cancer stem cell (BCSC), ASR490, autophagy, Notch1
Citation: Saran U, Chandrasekaran B, Tyagi A, Shukla V, Singh A, Sharma AK and Damodaran C (2023) Corrigendum: A small molecule inhibitor of Notch1 modulates stemness and suppresses breast cancer cell growth. Front. Pharmacol. 14:1207589. doi: 10.3389/fphar.2023.1207589
Received: 17 April 2023; Accepted: 24 April 2023;
Published: 03 May 2023.
Edited and reviewed by:
Eswar Shankar, The Ohio State University, United StatesCopyright © 2023 Saran, Chandrasekaran, Tyagi, Shukla, Singh, Sharma and Damodaran. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Chendil Damodaran, chendamodar@tamu.edu
†These authors have contributed equally to this work