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CORRECTION article

Front. Oncol.
Sec. Pharmacology of Anti-Cancer Drugs
Volume 14 - 2024 | doi: 10.3389/fonc.2024.1494738

Frontiers Corrigendum Template for Authors

Provisionally accepted
Xuehua Zhang Xuehua Zhang 1Leilei Zhao Leilei Zhao 1Jinlong Xiao Jinlong Xiao 1Yudi Wang Yudi Wang 1Yunmeng Li Yunmeng Li 1Chaoqun Zhu Chaoqun Zhu 2He Zhang He Zhang 3Yurui Zhang Yurui Zhang 1Xiao Zhu Xiao Zhu 2Yucui Dong Yucui Dong 1*
  • 1 Department of Immunology, Binzhou Medical University, Yantai, China
  • 2 School of Computer and Control Engineering, Yantai University, Yantai, Shandong Province, China
  • 3 Department of Immunology, Qiqihar Medical University,, Qiqihar,, China

This article is a correction to:

  1. 5-Demethylnobiletin mediates cell cycle arrest and apoptosis via the ERK1/2/AKT/STAT3 signaling pathways in glioblastoma cells

    1. Read original article

The final, formatted version of the article will be published soon.

    Keywords: Glioblastoma, 5-Demethylnobiletin, cell cycle arrest, cell apoptosis, ERK1/2, Akt, stat3

    Received: 11 Sep 2024; Accepted: 16 Sep 2024.

    Copyright: © 2024 Zhang, Zhao, Xiao, Wang, Li, Zhu, Zhang, Zhang, Zhu and Dong. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

    * Correspondence: Yucui Dong, Department of Immunology, Binzhou Medical University, Yantai, China

    Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.