Huiyun Yang
Yuliang Pan2
Long Jin
Xi Zhang
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- 1Department of Oncology, Xiangya Hospital, Central South University, Changsha, China
- 2Department of Oncology, Third Xiangya Hospital, Central South University, Changsha, China
By Yang H, Pan Y, Zhang J, Jin L and Zhang X (2021). Front. Oncol. 11:715635. doi: 10.3389/fonc.2021.715635
In the published article, there was an error in Figure 5 due to incorrect image editing in Figure 5D as published. The corrected Figure 5 appear below.
Figure 5
Figure 5 YBX1 reversed the proliferation, migration and invasion induced by knockdown of lncRNA FOXD3-AS1. (A) The expression of YBX1 was upregulated by transient transfection in cells silencing FOXD3-AS1 (shF-1), which was detected by western blot. (B, C) That YBX1 reversed the proliferation induced by knockdown of lncRNA FOXD3-AS1 was demonstrated by CCK8 assays (B) and colony forming assays (C) in HONE1 and SUNE1 cells. Colonies of colony forming assays were counted and compared by histogram (C, right). (D, E) That YBX1 reversed migration (D) and invasion (E) induced by knockdown of FOXD3-AS1 was validated by transwell assays in HONE1 and SUNE1 cells. Cells of transwell assays were calculated and compared by histograms [(D, E), right]. *P < 0.05, **P < 0.01.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: FOXD3-AS1, nasopharyngeal carcinoma, YBX1, H3K27ac, lnc RNA
Citation: Yang H, Pan Y, Zhang J, Jin L and Zhang X (2024) Corrigendum: LncRNA FOXD3-AS1 promotes the malignant progression of nasopharyngeal carcinoma through enhancing the transcription of YBX1 by H3K27Ac modification. Front. Oncol. 14:1436609. doi: 10.3389/fonc.2024.1436609
Received: 22 May 2024; Accepted: 03 June 2024;
Published: 11 June 2024.
Edited and Reviewed by:
Kwai Fung Hui, The University of Hong Kong, Hong Kong SAR, ChinaCopyright © 2024 Yang, Pan, Zhang, Jin and Zhang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Xi Zhang, eHkzemhhbmd4aUBjc3UuZWR1LmNu