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CORRECTION article

Front. Oncol., 07 September 2021
Sec. Molecular and Cellular Oncology

Corrigendum: Loss of TARBP2 Drives the Progression of Hepatocellular Carcinoma via miR-145-SERPINE1 Axis

Li-Man Li&#x;Li-Man Li1†Chang Chen&#x;Chang Chen2†Ruo-Xi Ran&#x;Ruo-Xi Ran1†Jing-Tao Huang,Jing-Tao Huang1,3Hui-Lung SunHui-Lung Sun4Chang ZengChang Zeng2Zhou ZhangZhou Zhang2Wei Zhang,*&#x;Wei Zhang2,5*‡Song-Mei Liu,*&#x;Song-Mei Liu1,6*‡
  • 1Department of Clinical Laboratory, Center for Gene Diagnosis, and Program of Clinical Laboratory Medicine, Zhongnan Hospital of Wuhan University, Wuhan, China
  • 2Department of Preventive Medicine, Northwestern University Feinberg School of Medicine, Chicago, IL, United States
  • 3Department of Clinical Laboratory, Renmin Hospital, Wuhan University, Wuhan, China
  • 4Department of Chemistry and Institute for Biophysical Dynamics, Howard Hughes Medical Institute, The University of Chicago, Chicago, IL, United States
  • 5Institute of Precision Medicine, Jining Medical University, Jining, China
  • 6Hubei Province Key Laboratory of Allergy and Immunology, Wuhan, China

A Corrigendum on
Loss of TARBP2 Drives the Progression of Hepatocellular Carcinoma via miR-145-SERPINE1 Axis

By Li L-M, Chen C, Ran R-X, Huang J-T, Sun H-L, Zeng C, Zhang Z, Zhang W, Liu S-M (2021). Front. Oncol. 11:620912. doi:  10.3389/fonc.2021.620912

In the original article, there were mistakes in Figure 5 and Figure 7 as published. In Figure 5A and Figure 7A, CTL should be sh-Ctrl to keep consistence of the abbreviation throughout of the manuscript. In Figure 5B, a layer was not removed due to our less carefulness during PS operation. The corrected Figures appear below.

FIGURE 5
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Figure 5 SERPINE1 is a downstream player of TARBP2. (A, B) The mRNA and protein expression of TARBP2 and SERPINE1 in the stable sh-TARBP2 HepG2 cells after treatment with scramble control and sh-SERPINE1. (C) Cell migration; (D) Invasion; and (E) Proliferation assays of sh-TARBP2 HepG2 cells treated with scramble control and sh-SERPINE1. *p-value < 0.05; **p-value < 0.01.

FIGURE 7
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Figure 7 miR-145 mediates SERPINE1 to affect the role of TARBP2 in HCC progression. (A) Levels of miR-145 in sh-TARBP2 HepG2 cells after treatment with Mimic NC and Mimic miR-145. (B) Protein levels of SERPINE1 and TARBP2 in the stable sh-TARBP2 HepG2 cells after treatment with Mimic NC and Mimic miR-145. (C) Cell migration; (D) Invasion; and (E) Proliferation of sh-TARBP2 HepG2 cells treated with Mimic NC and Mimic miR-145. *p-value < 0.05; **p-value < 0.01; NC, negative control.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s Note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: HCC, TARBP2, miR-145, SERPINE1, progression

Citation: Li L-M, Chen C, Ran R-X, Huang J-T, Sun H-L, Zeng C, Zhang Z, Zhang W and Liu S-M (2021) Corrigendum: Loss of TARBP2 Drives the Progression of Hepatocellular Carcinoma via miR-145-SERPINE1 Axis. Front. Oncol. 11:746958. doi: 10.3389/fonc.2021.746958

Received: 25 July 2021; Accepted: 13 August 2021;
Published: 07 September 2021.

Edited and reviewed by:

Guohui Wan, Sun Yat-sen University, China

Copyright © 2021 Li, Chen, Ran, Huang, Sun, Zeng, Zhang, Zhang and Liu. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Song-Mei Liu, smliu@whu.edu.cn; Wei Zhang, wei.zhang1@northwestern.edu

These authors have contributed equally to this work

These authors share senior authorship

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.