AUTHOR=Jiao Kexin , Dong Jihong , Luo Sushan , Yu Liqiang , Ke Qing , Wang Zhiqiang , Luan Xinghua , Zhang Xiaojie , Guo Junhong , Chen Yan , Li Xihua , Tan Song , Qian Fangyuan , Jiang Jianming , Yu Xuen , Yue Dongyue , Liu Changxia , Luo Lijun , Li Jianping , Qu Yanzhou , Chen Lan , Tu Jianglong , Sun Chong , Yan Chong , Song Jie , Xi Jianying , Lin Jie , Lu Jiahong , Zhao Chongbo , Zhu Wenhua , Fang Qi TITLE=High-risk screening of late-onset Pompe disease: A different early portrait in China JOURNAL=Frontiers in Neurology VOLUME=13 YEAR=2022 URL=https://www.frontiersin.org/journals/neurology/articles/10.3389/fneur.2022.965207 DOI=10.3389/fneur.2022.965207 ISSN=1664-2295 ABSTRACT=Introduction

The lack of knowledge regarding the differences between Chinese and other ethnicities in the early manifestation of late-onset Pompe disease (LOPD) prohibits the development of an effective screening strategy. We conducted a multicenter screening study to determine LOPD prevalence in high-risk populations and define the early manifestation of LOPD in China.

Methods

Between August 2020 and April 2021, the participants were prospectively identified through medical examination at 20 centers from inpatient departments and outpatient neuromuscular clinics in China. The inclusion criteria were as follows: (1) age ≥ 1 year and (2) either one of the following conditions: (a) persistent hyperCKemia, (b) muscle weakness of the axial and/or limb-girdle muscles, or (c) unexplained restrictive respiratory insufficiency (RI). Enzymatic activity of acid α-glucosidase (GAA) was measured in a dried blood spot (DBS) using a tandem mass spectrometry (MS/MS) assay. Next-generation sequencing (NGS) was used to evaluate all samples with decreased GAA activity, searching for GAA mutations and pseudodeficiency alleles.

Results

Among the 492 cases, 26 positive samples (5.3%) were detected in the DBS test. Molecular studies confirmed a diagnosis of LOPD in eight cases (1.6%). Using MS/MS assay, GAA activities in individuals with pseudodeficiency could be distinguished from those in patients with LOPD. The median interval from the onset of symptoms to diagnosis was 5 years. All patients also showed RI, with a mean forced vital capacity (FVC) of 48%, in addition to axial/proximal muscle weakness. The creatine kinase (CK) level ranged from normal to no more than 5-fold the upper normal limit (UNL). LOPD with isolated hyperCKemia was not identified.

Conclusion

Less frequent hyperCKemia and predominant RI depict a different early portrait of adult Chinese patients with LOPD. A modified high-risk screening strategy should be proposed for the early diagnosis of Chinese patients with LOPD.