Shu-yi Tan
Jin-xiang Jiang
Hui-xian Huang
Xiu-ping Mo
Jing-ru Feng1
Yu Chen
Li Yang
Cheng Long
95% of researchers rate our articles as excellent or good
Learn more about the work of our research integrity team to safeguard the quality of each article we publish.
Find out more
- 1School of Life Sciences, South China Normal University, Guangzhou, China
- 2School of Life Sciences, Guangzhou University, Guangzhou, China
- 3South China Normal University-Panyu Central Hospital Joint Laboratory of Translational Medical Research, Panyu Central Hospital, Guangzhou, China
A corrigendum on
Neural mechanism underlies CYLD modulation of morphology and synaptic function of medium spiny neurons in dorsolateral striatum
by Tan, S.-y., Jiang, J.-x., Huang, H.-x., Mo, X.-p., Feng, J.-r., Chen, Y., Yang, L., and Long, C. (2023). Front. Mol. Neurosci. 16:1107355. doi: 10.3389/fnmol.2023.1107355
In the published article, there was an error in the legend for “Figure 1” as published. “(H) Bar graphs showing more stubby spines and varicosity, but fewer mushroom spines and filopodia, in Cyld−/− MSNs than in Cyld+/+ MSNs.” The corrected legend appears below.
“(H) Bar graphs showing more stubby spines and varicosity, but fewer mushroom spines, in Cyld−/− MSNs than in Cyld+/+ MSNs.”
In the published article, there was an error. “P-value error for the main effect of distance in Figure 1E.”
A correction has been made to section 3. Results, “3.1. CYLD affects the morphology and physiological features of MSNs,” [Paragraph 1]. This sentence previously stated:
“main effect of distance, F(3.202,153.3) = 202.200, p = 0.188”
The corrected sentence appears below:
“main effect of distance, F(3.202,153.3) = 202.200, p < 0.0001”
In the published article, there was an error. “Misspelling on a word “GluA2”.”
A correction has been made to section 4. Discussion, “The last paragraph.” This sentence previously stated:
“CYLD deficiency causes an increase in K63-linked ubiquitination of GluA1 and GluA2, resulting in reduced GluA1 and GluA1 surface levels and therefore reduced AMPAR-dependent synaptic transmission in MSNs, which is associated with altered DHPG-and HFS-LTD.”
The corrected sentence appears below:
“CYLD deficiency causes an increase in K63-linked ubiquitination of GluA1 and GluA2, resulting in reduced GluA1 and GluA2 surface levels and therefore reduced AMPAR-dependent synaptic transmission in MSNs, which is associated with altered DHPG-and HFS-LTD.”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Publisher's note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Keywords: CYLD, AMPAR, GluA1, GluA2, K63-linked ubiquitination, synaptic transmission, long-term depression, dorsolateral striatum
Citation: Tan S-y, Jiang J-x, Huang H-x, Mo X-p, Feng J-r, Chen Y, Yang L and Long C (2023) Corrigendum: Neural mechanism underlies CYLD modulation of morphology and synaptic function of medium spiny neurons in dorsolateral striatum. Front. Mol. Neurosci. 16:1162275. doi: 10.3389/fnmol.2023.1162275
Received: 09 February 2023; Accepted: 13 February 2023;
Published: 23 February 2023.
Edited and reviewed by: Victor Anggono, The University of Queensland, Australia
Copyright © 2023 Tan, Jiang, Huang, Mo, Feng, Chen, Yang and Long. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Li Yang, 
eWFuZ19saUBnemh1LmVkdS5jbg==; Cheng Long, bG9uZ2NoZW5nQG0uc2NudS5lZHUuY24=