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CORRECTION article

Front. Mol. Biosci., 23 July 2024
Sec. Molecular Diagnostics and Therapeutics

Corrigendum: CMTM6 as a candidate risk gene for cervical cancer: comprehensive bioinformatics study

Xiaoting Huang,&#x;Xiaoting Huang1,2Wei Liu,&#x;Wei Liu1,2Chunshan Liu,&#x;Chunshan Liu1,2Jijie HuJijie Hu3Baiyao Wang,Baiyao Wang1,2Anbang Ren,Anbang Ren1,2Xiaona HuangXiaona Huang4Yawei Yuan,
Yawei Yuan1,2*Jinquan Liu,
Jinquan Liu1,2*Mingyi Li,
Mingyi Li1,2*
  • 1Department of Radiation Oncology, Affiliated Cancer Hospital & Institute of Guangzhou Medical University, Guangzhou, China
  • 2State Key Laboratory of Respiratory Diseases, Guangzhou Institute of Respiratory Disease, Affiliated Cancer Hospital & Institute of Guangzhou Medical University, Guangzhou, China
  • 3Department of Orthopaedics and Traumatology, Nanfang Hospital, Southern Medical University, Guangzhou, China
  • 4TCM Hospital of Liwan District, Guangzhou, China

A Corrigendum on
CMTM6 as a candidate risk gene for cervical cancer: Comprehensive bioinformatics study

by Huang X, Liu W, Liu C, Hu J, Wang B, Ren A, Huang X, Yuan Y, Liu J and Li M (2022). Front. Mol. Biosci. 9:983410. doi: 10.3389/fmolb.2022.983410

In the published article, there was an error in Figure 6 as published. In Figure 6A, in the Western blot bands labeled “ERK” and “JNK,” the use of ERK was incorrect and caused an overlap. The corrected Figure 6 and its caption appear below.

Figure 6
www.frontiersin.org

Figure 6. Effect of inhibited MAPK signaling on CMTM6-regulated cell invasion and migration. (A, B) Western blotting showing the levels of MAPK-related proteins in each group. Data were normalized to the level of GAPDH. (C–F) Cell metastasis and migration were assessed using Transwell assays in which CMTM6 acted as an inhibitor of the MAPK signaling pathway. *p < 0.05; **p < 0.01; ***p < 0.001.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: CMTM6, cervical cancer, proliferation, migration, MAPK signaling

Citation: Huang X, Liu W, Liu C, Hu J, Wang B, Ren A, Huang X, Yuan Y, Liu J and Li M (2024) Corrigendum: CMTM6 as a candidate risk gene for cervical cancer: comprehensive bioinformatics study. Front. Mol. Biosci. 11:1452740. doi: 10.3389/fmolb.2024.1452740

Received: 21 June 2024; Accepted: 04 July 2024;
Published: 23 July 2024.

Edited and reviewed by:

Matteo Becatti, University of Firenze, Italy

Copyright © 2024 Huang, Liu, Liu, Hu, Wang, Ren, Huang, Yuan, Liu and Li. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Yawei Yuan, yuanyawei@gzhmu.edu.cn; Jinquan Liu, ccgmuliujinquan@163.com; Mingyi Li, huangxiaoting5960@163.com

These authors have contributed equally to this work and share first authorship

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.