miR-193a-3p Mediates Placenta Accreta Spectrum Development by Targeting EFNB2 via Epithelial-Mesenchymal Transition Pathway Under Decidua Defect Conditions
- 1Department of Obstetrics and Gynecology, Shengjing Hospital of China Medical University, Shenyang, China
- 2Key Laboratory of Maternal-Fetal Medicine of Liaoning Province, Key Laboratory of Obstetrics and Gynecology of Higher Education of Liaoning Province, Benxi, China
by Li, N., Hou, R., Yang, T., Liu, C., and Wei, J. (2021). Front. Mol. Biosci. 7:613802. doi:10.3389/fmolb.2020.613802
In the original article, there was a mistake in Figure 4 as published. The picture of “Control” in Figure 4C was wrong when we upload the pictures. The corrected Figure 4 appears below.
FIGURE 4. EFNB2 downregulation promotes the migration and invasion of HTR-8/SVneo cells. (A) EFNB2 mRNA expression examined after transfection of HTR-8/SVneo cells with siRNA for EFNB2. (B) Levels of EFNB2 and EMT-related proteins detected after 48-h transfection. (C,D) Transwell migration and invasion assays showing higher numbers of migrated and invaded cells in the si-EFNB2 group than in the NC group (×200). All data are presented as the mean ± standard deviation. EFNB2, Ephrin-B2; Si, siRNA; NC, negative control; EMT, epithelial-mesenchymal transition. *P < 0.05, **P < 0.01, ***P < 0.001, and ****P < 0.0001.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: miR-193a-3p, EFNB2, epithelial-mesenchymal transition, decidua defect, placenta accreta spectrum
Citation: Li N, Hou R, Yang T, Liu C and Wei J (2022) Corrigendum: miR-193a-3p Mediates Placenta Accreta Spectrum Development by Targeting EFNB2 via Epithelial-Mesenchymal Transition Pathway Under Decidua Defect Conditions. Front. Mol. Biosci. 9:839235. doi: 10.3389/fmolb.2022.839235
Received: 19 December 2021; Accepted: 24 January 2022;
Published: 15 February 2022.
Edited and reviewed by:
Yasuhito Ishigaki, Kanazawa Medical University, JapanCopyright © 2022 Li, Hou, Yang, Liu and Wei. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Caixia Liu, liucxshimen@163.com; Jun Wei, weij@sj-hospital.org