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Tianwen Huang1,2,3†
Yaoyu Pu1,2,3†
Xiangpeng Wang1,2,3
Yanhong Li1,2,3
Hang Yang1,2,3
Yubin Luo1,2,3*‡
Yi Liu1,2,3*‡- 1Department of Rheumatology and Immunology, West China Hospital, Sichuan University, Chengdu, China
- 2Rare Diseases Center, West China Hospital, Sichuan University, Chengdu, China
- 3Institute of Immunology and Inflammation, Frontiers Science Center for Disease Related Molecular Network, West China Hospital, Chengdu, China
A corrigendum on
Metabolomic analysis in spondyloarthritis: A systematic review
by Huang, T., Pu, Y., Wang, X., Li, Y., Yang, H., Luo, Y., and Liu, Y. (2022). Front. Microbiol. 13:965709. doi: 10.3389/fmicb.2022.965709
In the published article, there was an error. We mistakenly described the trend of changes in the levels of some metabolites after treatment of SpA patients.
A correction has been made to Results, “Dynamic alterations in the metabolic profile before and after treatment of spondyloarthritis,” Paragraphs 2 and 4. These sentences previously stated:
“Kapoor et al. (2013) and Bogunia-Kubik et al. (2021) found elevated levels of isobutyrate and acetone in AS as well as elevated levels of acetate in PsA after TNFi therapy. Decreased levels of amino acids, including histidine, leucine and phenylalanine, were also found in AS patients after therapy. Another study including PsA patients receiving TNFi therapy showed higher levels of glutamine than those observed at baseline. Additionally, the two studies both found decreased creatine and creatinine levels in SpA patients after treatment.”
And
“In addition, in the serum of JIA patients, MTX therapy increased the level of omega-3 unsaturated fatty acids (docosahexanoic acid and linoleic acid), which are anti-inflammatory mediators.”
The corrected sentence appears below:
“Kapoor et al. (2013) and Bogunia-Kubik et al. (2021) found decreased levels of isobutyrate and acetone in AS as well as decreased levels of acetate in PsA after TNFi therapy. Elevated levels of amino acids, including histidine, leucine and phenylalanine, were also found in AS patients after therapy. Another study including PsA patients receiving TNFi therapy showed lower levels of glutamine than those observed at baseline. Additionally, the two studies both found elevated creatine and creatinine levels in SpA patients after treatment.”
And
“In addition, in the serum of JIA patients, MTX therapy reduced the level of omega-3 unsaturated fatty acids (docosahexanoic acid and linoleic acid), which are anti-inflammatory mediators.”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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References
Bogunia-Kubik, K., Wojtowicz, W., Swierkot, J., Mielko, K. A., Qasem, B., Wielinska, J., et al. (2021). Disease differentiation and monitoring of anti-TNF treatment in rheumatoid arthritis and spondyloarthropathies. Int. J. Mol. Sci. 22:7389. doi: 10.3390/ijms22147389
Keywords: spondyloarthritis, ankylosing spondylitis, metabolomics, biomarkers, dysbiosis
Citation: Huang T, Pu Y, Wang X, Li Y, Yang H, Luo Y and Liu Y (2022) Corrigendum: Metabolomic analysis in spondyloarthritis: A systematic review. Front. Microbiol. 13:1100290. doi: 10.3389/fmicb.2022.1100290
Received: 16 November 2022; Accepted: 30 November 2022;
Published: 12 December 2022.
Edited and reviewed by: M. Pilar Francino, Fundación para el Fomento de la Investigación Sanitaria y Biomédica de la Comunitat Valenciana (FISABIO), Spain
Copyright © 2022 Huang, Pu, Wang, Li, Yang, Luo and Liu. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Yubin Luo, 
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†These authors have contributed equally to this work
‡ORCID: Yubin Luo orcid.org/0000-0002-7669-1579
Yi Liu orcid.org/0000-0002-9258-3594