AUTHOR=Roberts Timothy P. L. , Bloy Luke , Blaskey Lisa , Kuschner Emily , Gaetz Leah , Anwar Ayesha , Ku Matt , Dipiero Marissa , Bennett Amanda , Edgar J. Christopher TITLE=A MEG Study of Acute Arbaclofen (STX-209) Administration JOURNAL=Frontiers in Integrative Neuroscience VOLUME=13 YEAR=2019 URL=https://www.frontiersin.org/journals/integrative-neuroscience/articles/10.3389/fnint.2019.00069 DOI=10.3389/fnint.2019.00069 ISSN=1662-5145 ABSTRACT=
Several electrophysiological parameters, including the auditory evoked response component M50/M100 latencies and the phase synchrony of transient and steady-state gamma-band oscillations have been implicated as atypical (to various extents) in autism spectrum disorder (ASD). Furthermore, some hypotheses suggest that an underlying neurobiological mechanism for these observations might be atypical local circuit function indexed by atypical levels of inhibitory neurotransmitter, GABA. This study was a randomized, placebo-controlled, double-blind, escalating-dose, acute investigation conducted in 25 14–18 year-old adolescents with ASD. The study assessed the sensitivity of magnetoencephalography (MEG) and MEGAPRESS “GABA” magnetic resonance spectroscopy (MRS) to monitor dose-dependent acute effects, as well as seeking to define properties of the pre-drug “baseline” electrophysiological and GABA signatures that might predict responsiveness to the GABA-B agonist, arbaclofen (STX-209). Overall, GABA levels and gamma-band oscillatory activity showed no acute changes at either low (15 mg) or high (30 mg) dose. Evoked M50 response latency measures tended to shorten (normalize), but there was heterogeneity across the group in M50 latency response, with only a subset of participants (