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ORIGINAL RESEARCH article
Front. Immunol.
Sec. T Cell Biology
Volume 15 - 2024 |
doi: 10.3389/fimmu.2024.1531294
This article is part of the Research Topic Enhancing T Cell Function: Innovations in Cancer Immunotherapy View all 9 articles
A self-activated and protective (SAP) module enhances the preclinical performance of allogeneic anti-CD70 CAR-T cells
Provisionally accepted- 1 Nanjing Normal University, Nanjing, China
- 2 Affiliated Hospital, Inner Mongolia Medical University, Hohhot, Inner Mongolia Autonomous Region, China
- 3 Jiangsu Province Geriatric Hospital, Nanjing, Jiangsu Province, China
- 4 Nanjing CalmHome Institute of Cell and Gene Engineering, Nanjing, China
Allogeneic chimeric antigen receptor T (CAR-T) therapy, also known as universal CAR-T (UCAR-T) therapy, offers broad applicability, high production efficiency, and reduced costs, enabling quicker access for patients. However, clinical application remains limited by challenges such as immune rejection, and issues with potency and durability. To address these limitations, we developed an 'all-in-one' self-activated and protective (SAP) module, integrated into the CAR scaffold. The SAP module consists of the CD47 extracellular domain, a mutant interleukin 7 receptor alpha (IL7Rα) transmembrane domain, and the IL7Rα intracellular domain, designed to protect UCAR-T cells from host immune attacks and enhance their survival. As anticipated, SAP UCAR-T cells experienced reduced immune rejection from the innate immune system, showed improved persistence and functionality both in vitro and in vivo, and raised no safety concerns. Moreover, the process stability of SAP UCAR-T cells was demonstrated in scale-up production. Overall, our findings highlight the SAP module as a promising strategy for the preclinical development of anti-CD70 UCAR-T, paving the way for an 'off-the-shelf' cell therapy product.
Keywords: chimeric antigen receptor T (CAR-T), allogeneic CAR-T, cancer immunotherapy, Solid tumor, CD70-positive cancer
Received: 20 Nov 2024; Accepted: 30 Dec 2024.
Copyright: © 2024 Zhang, Zhao, Huang, Chen, Zhao, Liang, Ao, Jia, Kang, Kong, Jing, Hu, Gu, Pan, Hu, He, Zhou, Chen and Guo. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
* Correspondence:
Lianfeng Zhao, Nanjing Normal University, Nanjing, China
Tinghui Huang, Nanjing Normal University, Nanjing, China
Zhengliang Chen, Nanjing Normal University, Nanjing, China
Yaoyao Zhao, Nanjing Normal University, Nanjing, China
Junqing Liang, Affiliated Hospital, Inner Mongolia Medical University, Hohhot, Inner Mongolia Autonomous Region, China
Xudong Ao, Affiliated Hospital, Inner Mongolia Medical University, Hohhot, Inner Mongolia Autonomous Region, China
Xiaoqiong Jia, Affiliated Hospital, Inner Mongolia Medical University, Hohhot, Inner Mongolia Autonomous Region, China
Lei Kang, Affiliated Hospital, Inner Mongolia Medical University, Hohhot, Inner Mongolia Autonomous Region, China
Qi Jing, Affiliated Hospital, Inner Mongolia Medical University, Hohhot, Inner Mongolia Autonomous Region, China
Jianhua Hu, Jiangsu Province Geriatric Hospital, Nanjing, 210024, Jiangsu Province, China
Lili Gu, Nanjing CalmHome Institute of Cell and Gene Engineering, Nanjing, China
Muya Zhou, Nanjing Normal University, Nanjing, China
Jiannan Chen, Nanjing Normal University, Nanjing, China
Zhigang Guo, Nanjing Normal University, Nanjing, China
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