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CORRECTION article

Front. Immunol., 23 January 2024
Sec. Autoimmune and Autoinflammatory Disorders : Autoimmune Disorders

Corrigendum: The immune factors have complex causal regulation effects on inflammatory bowel disease

  • Department of Gastric and Colorectal Surgery, General Surgery Center, The First Hospital of Jilin University, Changchun, China

A Corrigendum on
The immune factors have complex causal regulation effects on inflammatory bowel disease

By Qiu B, Zhang T, Qin X, Ma S and Wang Q (2024) Front. Immunol. 14:1322673. doi: 10.3389/fimmu.2023.1322673

In the published article, there were some errors. Because of our carelessness and inattention when we first undertook manuscript writing, we made these errors.

A correction has been made to Abstract, Results. This sentence previously stated:

“Subsequent meta-integration of the two datasets provided evidence of solid causal associations between 20 immune phenotypes and IBD and its subtypes. Nominal causal associations were also identified in the remaining six immune phenotypes and IBD and its subtypes.”

The corrected sentence appears below:

“Subsequent meta-integration of the two datasets provided evidence of solid causal associations between 18 immune phenotypes and IBD and its subtypes. Nominal causal associations were also identified in the remaining eight immune phenotypes and IBD and its subtypes.”

A correction has also been made to Abstract, Conclusion. This sentence previously stated:

“Our study confirms causal solid associations between 20 immune phenotypes and IBD, thus guiding future clinical studies.”

The corrected sentence appears below:

“Our study confirms causal solid associations between 18 immune phenotypes and IBD, thus guiding future clinical studies.”

Finally, a correction has also been made to Discussion, paragraph 1. This sentence previously stated:

“The results of the study indicate that 24 immune phenotypes may be causally linked to IBD and its different subtypes. Combining the results from two datasets, we discovered that 20 immune phenotypes remain strongly causally linked to IBD and its subtypes.”

The corrected sentence appears below:

“The results of the study indicate that 26 immune phenotypes may be causally linked to IBD and its different subtypes. Combining the results from two datasets, we discovered that 18 immune phenotypes remain strongly causally linked to IBD and its subtypes.”

The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: inflammatory bowel disease, causal relationship, Mendelian randomization, Crohn’s disease, ulcerative colitis

Citation: Qiu B, Zhang T, Qin X, Ma S and Wang Q (2024) Corrigendum: The immune factors have complex causal regulation effects on inflammatory bowel disease. Front. Immunol. 15:1368428. doi: 10.3389/fimmu.2024.1368428

Received: 10 January 2024; Accepted: 15 January 2024;
Published: 23 January 2024.

Edited and Reviewed by:

Die Wang, Genentech, United States

Copyright © 2024 Qiu, Zhang, Qin, Ma and Wang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Quan Wang, wquan@jlu.edu.cn

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.