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Yuanyuan Wu1,2†
Lili Huang3†
Wenli Sai4†Fei Chen3Yu Liu3
Cheng Han3Joanna M. Barker5
Zinah D. Zwaini6Mark P. Lowe5
Nigel J. Brunskill2,3
Bin Yang2,3*- 1Department of Pathology, Medical School of Nantong University, Nantong, China
- 2Department of Cardiovascular Sciences, College of Life Sciences, University of Leicester, University Hospitals of Leicester NHS Trust, Leicester, United Kingdom
- 3Nantong-Leicester Joint Institute of Kidney Science, Nephrology, Affiliated Hospital of Nantong University, Nantong, China
- 4Research Center of Clinical Medicine, Affiliated Hospital of Nantong University, Nantong, China
- 5School of Chemistry, University of Leicester, Leicester, United Kingdom
- 6Department of Respiratory Sciences, College of Life Sciences, University of Leicester, Leicester, United Kingdom
By Wu Y, Huang L, Sai W, Chen F, Liu Y, Han C, Barker JM, Zwaini ZD, Lowe MP, Brunskill NJ and Yang B (2023) Front. Immunol. 14:1183768. doi: 10.3389/fimmu.2023.1183768
In the published article, an author name was incorrectly written as Weili Sai. The correct spelling is Wenli Sai.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated
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Keywords: HBSP, innate repair receptor, ischaemia-reperfusion injury, phagocytosis, properdin, repair, tubular epithelial cells
Citation: Wu Y, Huang L, Sai W, Chen F, Liu Y, Han C, Barker JM, Zwaini ZD, Lowe MP, Brunskill NJ and Yang B (2023) Corrigendum: HBSP improves kidney ischemia-reperfusion injury and promotes repair in properdin deficient mice via enhancing phagocytosis of tubular epithelial cells. Front. Immunol. 14:1242436. doi: 10.3389/fimmu.2023.1242436
Received: 19 June 2023; Accepted: 20 June 2023;
Published: 29 June 2023.
Approved by:
Frontiers Editorial Office, Frontiers Media SA, SwitzerlandCopyright © 2023 Wu, Huang, Sai, Chen, Liu, Han, Barker, Zwaini, Lowe, Brunskill and Yang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Bin Yang, Ynk1QGxlLmFjLnVr
†These authors have contributed equally to this work and share first authorship