AUTHOR=Bordet Elise , Blanc Fany , Tiret Mathieu , Crisci Elisa , Bouguyon Edwige , Renson Patricia , Maisonnasse Pauline , Bourge Mickael , Leplat Jean-Jacques , Giuffra Elisabetta , Jouneau Luc , Schwartz-Cornil Isabelle , Bourry Olivier , Bertho Nicolas TITLE=Porcine Reproductive and Respiratory Syndrome Virus Type 1.3 Lena Triggers Conventional Dendritic Cells 1 Activation and T Helper 1 Immune Response Without Infecting Dendritic Cells JOURNAL=Frontiers in Immunology VOLUME=9 YEAR=2018 URL=https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2018.02299 DOI=10.3389/fimmu.2018.02299 ISSN=1664-3224 ABSTRACT=
Porcine Reproductive and Respiratory Syndrome virus (PRRSV) is an arterivirus responsible for highly contagious infection and huge economic losses in pig industry. Two species, PRRSV-1 and PRRSV-2 are distinguished, PRRSV-1 being more prevalent in Europe. PRRSV-1 can further be divided in subtypes. PRRSV-1.3 such as Lena are more pathogenic than PRRSV-1.1 such as Lelystad or Flanders13. PRRSV-1.3 viruses trigger a higher Th1 response than PRRSV-1.1, although the role of the cellular immune response in PRRSV clearance remains ill defined. The pathogenicity as well as the T cell response inductions may be differentially impacted according to the capacity of the virus strain to infect and/or activate DCs. However, the interactions of PRRSV with