Identification of the first congenital ichthyosis case caused by a homozygous deletion in the ALOX12B gene due to chromosome 17 mixed uniparental disomy
- 1Precision Medical Center, Wuhan Children’s Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science & Technology, Wuhan, China
- 2Dermatology Department, Wuhan Children’s Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science & Technology, Wuhan, China
- 3Otolaryngology Department, Wuhan Children’s Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science & Technology, Wuhan, China
- 4Neonatology Department, Wuhan Children’s Hospital (Wuhan Maternal and Child Healthcare Hospital), Tongji Medical College, Huazhong University of Science & Technology, Wuhan, China
A Corrigendum on
Identification of the first congenital ichthyosis case caused by a homozygous deletion in the ALOX12B gene due to chromosome 17 mixed uniparental disomy
by Zhang L, Hu Y, Lu J, Zhao P, Zhang X, Tan L, Li J, Xiao C, Zeng L and He X (2022). Front. Genet. 13:931833. doi: 10.3389/fgene.2022.931833
In the published article, there was an error in affiliations 1, 2, and 3.
In all three affiliations, “Huazhong University of Science & Technology, Wuhan, China”, should be “Wuhan Children’s Hospital (Wuhan Maternal and Child Healthcare), Tongji Medical College, Huazhong University of Science & Technology, Wuhan, China”.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Keywords: ARCI, ALOX12B, whole-exome sequencing, mixed UPD (mixUPD), microtia
Citation: Zhang L, Hu Y, Lu J, Zhao P, Zhang X, Tan L, Li J, Xiao C, Zeng L and He X (2022) Corrigendum: Identification of the first congenital ichthyosis case caused by a homozygous deletion in the ALOX12B gene due to chromosome 17 mixed uniparental disomy. Front. Genet. 13:1036144. doi: 10.3389/fgene.2022.1036144
Received: 04 September 2022; Accepted: 28 September 2022;
Published: 21 October 2022.
Edited and reviewed by:
Lidia Larizza, Research Lab of Medical Cytogenetics and Molecular Genetics, Italian Auxological Institute (IRCCS), ItalyCopyright © 2022 Zhang, Hu, Lu, Zhao, Zhang, Tan, Li, Xiao, Zeng and He. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Xuelian He, aGV4dWVsaWFuMjAxM0Bob3RtYWlsLmNvbQ==; Cuiping Xiao, NDU5NTE4MjQwQHFxLmNvbQ==; Linkong Zeng, ZnJlZW1hbjMxNUAxNjMuY29t
†These authors have contributed equally to this work