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EDITORIAL article

Front. Endocrinol.
Sec. Reproduction
Volume 15 - 2024 | doi: 10.3389/fendo.2024.1517200
This article is part of the Research Topic Environmental Threats to Human Reproduction View all 13 articles

Environmental threats to human reproduction

Provisionally accepted
Roland Eghoghosoa Akhigbe Roland Eghoghosoa Akhigbe *
  • Ladoke Akintola University of Technology, Ogbomosho, Nigeria

The final, formatted version of the article will be published soon.

    These environmental stressors acts via multiple pathways. First, they disrupt the endocrine system by mimicking or blocking sex hormones (Akhigbe et al., 2022). They may also induce oxidative stress by upregulating the generation of free radicals and suppressing antioxidants (Kumar et al., 2018;Akhigbe et al., 2021), trigger inflammation and immune response (Li et al., 2021;Ashonibare et al., 2024b), and promote genotoxicity (Choudhuri et al., 2021) Although SARS-CoV-2 remains quite novel, convincing evidences have been provided on its possible link with infertility (Akhigbe and Hamed, 2020;Adeyemi et al., 2022)

    Keywords: Environmental toxicant, environmental stressors, Oxidtive stress, epigenetics, Inflammation, Apoptosis

    Received: 25 Oct 2024; Accepted: 21 Nov 2024.

    Copyright: © 2024 Akhigbe. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

    * Correspondence: Roland Eghoghosoa Akhigbe, Ladoke Akintola University of Technology, Ogbomosho, Nigeria

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