Jiao Chen1*
Yanting Shao1Zhibin Cheng1
Guanghui Li2Fen Wan1Chenyan Gao1Danqin Wu3Dandan Wei4,5
Yang Liu4,5*Rong Li6*- 1School of Laboratory Medicine, Nanchang Medical College, Nanchang, China
- 2School of Information Engineering, East China Jiaotong University, Nanchang, China
- 3Neurology Intensive Care Unit (ICU), First Affiliated Hospital of Nanchang University, Nanchang, China
- 4Department of Clinical Microbiology, the First Affiliated Hospital of Nanchang University, Nanchang, China
- 5Clinical Laboratory, China-Japan Friendship Jiang Xi Hospital, Nanchang, China
- 6Department of Clinical Laboratory & Jiangxi Province Key Laboratory of Immunology and Inflammation, Jiangxi Provincial People’s Hospital & The first Affiliated Hospital of Nanchang Medical College, Nanchang, China
by Chen J, Shao Y, Cheng Z, Li G, Wan F, Gao C, Wu D, Wei D, Liu Y and Li R (2025). Front. Cell. Infect. Microbiol. 15:1549940. doi: 10.3389/fcimb.2025.1549940
In the published article, there was an error in the Funding statement. The original text:
“The author(s) declare financial support was received for the research, authorship, and/or publication of this article. This research was financially supported by the National Natural Science Foundation of China (82200194, 82102411,32370195,62362034), the Project of Science and Technology Innovation Talents in Jiangxi (JXSQ2019201102), the Clinical Research Nurture Project of the First Affiliated Hospital of Nanchang University (YFYLCYJPY202001), Natural Science Foundation of Jiangxi Province of China (20232ACB202010), the Jiangxi Provincial Health Commission-Health Commission science and technology project (SKJP220226762, 202310458), and Institutional Research Project of Nanchang medical college (NYXJ-2024-040).”
The correct Funding statement appears below.
“The author(s) declare financial support was received for the research, authorship, and/or publication of this article. This research was financially supported by the National Natural Science Foundation of China (82200194, 82102411,32370195,62362034), the Project of Science and Technology Innovation Talents in Jiangxi (JXSQ2019201102), the Clinical Research Nurture Project of the First Affiliated Hospital of Nanchang University (YFYLCYJPY202001), Natural Science Foundation of Jiangxi Province of China (20232ACB202010), the Jiangxi Provincial Health Commission-Health Commission science and technology project (SKJP220226762, 202310458), Project of Innovation Team on Inflammation and Cellular Immunity, and Institutional Research Project of Nanchang medical college (NYXJ-2024-040).”
In the published article, there was an error in the Abstract, Results. The P-value of biofilm formation between the ST2 and non-ST2 groups is incorrect. It should be 0.035.
This section previously stated:
“Results: The 30-day mortality rate of 67 patients with BSIs was 55.22%. Patients in the death group had significantly lower platelet counts and higher CRP levels than those in the survival group. Additionally, higher rates of antibiotic use (≥2 classes) and greater carbapenem exposure were observed. Among the isolates, CRAb accounted for 80.6%, ST2 accounted for 76.12%, and KL2/3/7/77/160 accounted for 65.67%. The predominant KL type was KL3, found in 19.4% of the isolates. All ST2 and KL2/3/7/77/160 isolates were CRAb. Among the isolates, 90.7% of the CRAb isolates coharbored blaOXA-23 and blaOXA-66, while one coharbored blaNDM-1 and blaOXA-23. Compared with non-ST2 and non KL2/3/7/77/160 infections, ST2 and KL2/3/7/77/160 infections had higher mortality rates (66.0% vs. 23.5%, P=0.002; 65.90% vs. 34.78%, P=0.015). Patients with ST2 and KL2/3/7/77/160 infections underwent more invasive procedures, received two or more antibiotics and carbapenem therapy before isolation, and had lower serum albumin levels. These isolates exhibited significantly higher resistance to antimicrobial agents. No significant differences in virulence phenotypes were observed between the two groups, except for biofilm formation between the ST2 and non-ST2 groups (P=0.002). However, these isolates harbored more virulence genes related to iron uptake and biofilm formation.”
The corrected section appears below:
“Results: The 30-day mortality rate of 67 patients with BSIs was 55.22%. Patients in the death group had significantly lower platelet counts and higher CRP levels than those in the survival group. Additionally, higher rates of antibiotic use (≥2 classes) and greater carbapenem exposure were observed. Among the isolates, CRAb accounted for 80.6%, ST2 accounted for 76.12%, and KL2/3/7/77/160 accounted for 65.67%. The predominant KL type was KL3, found in 19.4% of the isolates. All ST2 and KL2/3/7/77/160 isolates were CRAb. Among the isolates, 90.7% of the CRAb isolates coharbored blaOXA-23 and blaOXA-66, while one coharbored blaNDM-1 and blaOXA-23. Compared with non-ST2 and non KL2/3/7/77/160 infections, ST2 and KL2/3/7/77/160 infections had higher mortality rates (66.0% vs. 23.5%, P=0.002; 65.90% vs. 34.78%, P=0.015). Patients with ST2 and KL2/3/7/77/160 infections underwent more invasive procedures, received two or more antibiotics and carbapenem therapy before isolation, and had lower serum albumin levels. These isolates exhibited significantly higher resistance to antimicrobial agents. No significant differences in virulence phenotypes were observed between the two groups, except for biofilm formation between the ST2 and non-ST2 groups (P=0.035). However, these isolates harbored more virulence genes related to iron uptake and biofilm formation.”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: Acinetobacter baumannii, bloodstream infection, carbapenem-resistant Acinetobacter baumannii, sequence type, capsular type
Citation: Chen J, Shao Y, Cheng Z, Li G, Wan F, Gao C, Wu D, Wei D, Liu Y and Li R (2025) Corrigendum: Exploring the clinical outcomes and molecular characteristics of Acinetobacter baumannii bloodstream infections: a study of sequence types, capsular types, and drug resistance in China. Front. Cell. Infect. Microbiol. 15:1585728. doi: 10.3389/fcimb.2025.1585728
Received: 01 March 2025; Accepted: 11 March 2025;
Published: 19 March 2025.
Edited and Reviewed by:
Mithun Rudrapal, Vignan’s Foundation for Science, Technology and Research, IndiaCopyright © 2025 Chen, Shao, Cheng, Li, Wan, Gao, Wu, Wei, Liu and Li. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Jiao Chen, Y2hlbmppYW9AbmNtYy5lZHUuY24=; Yang Liu, bHkxMzc2NzE2MDQ3NEBzaW5hLmNvbQ==; Rong Li, bGlyb25nanhAb3V0bG9vay5jb20=