This article is a correction to:
CVB3-Mediated Mitophagy Plays an Important Role in Viral Replication via Abrogation of Interferon Pathways
Soo-Jin Oh
Byung-Kwan Lim2
Jeanho Yun
Ok Sarah Shin- 1BK21 Graduate Program, Department of Biomedical Sciences, College of Medicine, Korea University Guro Hospital, Seoul, South Korea
- 2Department of Biomedical Science, Jungwon University, Goesan-gun, South Korea
- 3Department of Translational Biomedical Sciences, Peripheral Neuropathy Research Center, College of Medicine, Dong-A University, Busan, South Korea
A Corrigendum on
CVB3-Mediated Mitophagy Plays an Important Role in Viral Replication via Abrogation of Interferon Pathways
by Oh S-J, Lim B-K, Yun J and Shin OS (2021). Front. Cell. Infect. Microbiol. 11:704494. doi: 10.3389/fcimb.2021.704494
Missing Funding
In the original article, we neglected to include the funder ** Korea Health Technology R&D Project through the Korea Health Industry Development Institute (KHIDI), funded by the Ministry of Health & Welfare, Republic of Korea **, **(HI21C1252)** to **SJO**.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Error in Figure/Table
In the original article, there was a mistake in **Figure 2** as published. ** The fluorescence laser of Figure 2C were mistakenly mislabeled. The red signal should be labeled as 561 nm and the green signal should be labeled as 488nm**. The corrected **Figure 2** appears below.
FIGURE 2
Figure 2 Induction of mitophagy following CVB3 infection. (A) hNPCs were infected with mock (m) or CVB3 at an MOI of 5 for the indicated time points. The cells were subsequently stained with MitoTracker Green and LysoTracker Red. Scale bar=10 μm. Percentage (%) of cells containing mitolysosomes is quantified and represented as a graph on the right. At least 100 cells were counted per experiment (n=3) (B) HeLa-Parkin cells were transfected with LC3-GFP (green) and mito-dsRED (red) plasmids and infected with CVB3 for 8 h or treated with control DMSO (Ctl) CCCP for 2 h. Scale bar=10 μm. Percentage (%) of cells with LC3 puncta associated with mitochondria is quantified and represented as a graph on the right. (C) HeLa-Parkin cells expressing mt-Keima were either treated with 25 μM CCCP for 2 h or infected with CVB3 for 8 h. The emission signal obtained after excitation with the 488 nm laser is shown in green, and that obtained after excitation with the 561 nm laser is shown in red. Zeiss ZEN software was used to determine changes in pH-dependent fluorescence and % mitophagy is represented in graph. Scale bar =10 μm. Bar graph shows mean ± SD from at least 100 cells/condition compiled from three experiments. *p < 0.05; **p < 0.01; ***p < 0.001 vs. control DMSO-treated cells.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: Coxsackievirus B3 virus (CVB3), mitochondrial dynamics, mitophagy, interferon, neural progenitor cells
Citation: Oh S-J, Lim B-K, Yun J and Shin OS (2021) Corrigendum: CVB3-Mediated Mitophagy Plays an Important Role in Viral Replication via Abrogation of Interferon Pathways. Front. Cell. Infect. Microbiol. 11:757341. doi: 10.3389/fcimb.2021.757341
Received: 12 August 2021; Accepted: 26 August 2021;
Published: 10 September 2021.
Edited and reviewed by:
Chunfu Zheng, University of Calgary, CanadaCopyright © 2021 Oh, Lim, Yun and Shin. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Jeanho Yun, eXVuakBkYXUuYWMua3I=; Ok Sarah Shin, b3NoaW5Aa29yZWEuYWMua3I=
†These authors have contributed equally to this work