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BRIEF RESEARCH REPORT article
Front. Cardiovasc. Med.
Sec. Atherosclerosis and Vascular Medicine
Volume 12 - 2025 |
doi: 10.3389/fcvm.2025.1492768
Inducing Hypertension in Myh11 R247C/R247C Mice Triggers Aortic Dissections with Increased Focal Adhesion Kinase Signaling
Provisionally accepted- 1 University of Texas Health Science Center at Houston, Houston, United States
- 2 McGill University, Montreal, Quebec, Canada
Objective: We sought to determine if hypertension in combination with a “variant of uncertain significance” that disrupts protein function, MYH11 p.Arg247Cys, would induce aortic dissections in a mouse model. Approach and Results: Administration of L-NAME via drinking water and a high salt diet increased blood pressure in WT and Myh11R247C/R247C mice and triggered type A dissections with cardiac tamponade in 20% of the Myh11R247C/R247C mice. Myh11R247C/R247C aortas have aberrant smooth muscle contractile unit-elastin connections by transmission electron microscopy, along with increased focal adhesion signaling at baseline, which further increases with hypertension.Conclusion: Gene-environment interactions trigger aortic dissections in Myh11R247C/R247C mice.
Keywords: myosin heavy chain, focal adhesion, elastin-contractile unit, thoracic aortic dissection Subject code: vascular disease, aortic dissection, Smooth muscle cell (SMC)
Received: 07 Sep 2024; Accepted: 31 Jan 2025.
Copyright: © 2025 Kwartler, Wang, Zhou, Guan, Yu, Duan, Zhang, Chen, Davis and Milewicz. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
* Correspondence:
Callie Kwartler, University of Texas Health Science Center at Houston, Houston, United States
Dianna Milewicz, University of Texas Health Science Center at Houston, Houston, United States
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