Skip to main content

ORIGINAL RESEARCH article

Front. Psychiatry, 09 January 2023
Sec. Psychopathology
This article is part of the Research Topic Community Series in ICD-11 Personality Disorders: Utility and Implications of the New Model, volume II View all 8 articles

Severity in the ICD-11 personality disorder model: Evaluation in a Spanish mixed sample

\r\nFernando Gutirrez,*Fernando Gutiérrez1,2*Anton Aluja,Anton Aluja3,4Claudia RodríguezClaudia Rodríguez1Miguel GrrizMiguel Gárriz5Josep M. PeriJosep M. Peri1Salvador GallartSalvador Gallart6Natalia Calvo,,Natalia Calvo7,8,9Marc Ferrer,,Marc Ferrer7,8,9Alfonso Gutirrez-Zotes,,Alfonso Gutiérrez-Zotes8,10,11Joaquim Soler,Joaquim Soler9,12Juan Carlos Pascual,Juan Carlos Pascual9,12
  • 1Institute of Neuroscience, Hospital Clínic of Barcelona, Barcelona, Spain
  • 2Institut d’Investigacións Biomèdiques August Pi i Sunyer (IDIBAPS), Barcelona, Spain
  • 3Lleida Institute for Biomedical Research Dr. Pifarré Foundation, Lleida, Spain
  • 4Department of Psychology, University of Lleida, Lleida, Spain
  • 5Neuropsychiatry and Drug Addiction Institute, Parc de Salut Mar, Barcelona, Spain
  • 6Department of Psychiatry, Mental Health, and Addiction, GSS–Hospital Santa Maria, Lleida, Spain
  • 7Department of Psychiatry, Vall d’Hebron University Hospital, Barcelona, Spain
  • 8Network Centre for Biomedical Research in Mental Health (CIBERSAM), Barcelona, Spain
  • 9Psychiatry and Legal Medicine Department, Autonomous University of Barcelona, Barcelona, Spain
  • 10Pere Virgili Health Research Institute (IISPV), CERCA, Reus, Spain
  • 11Pere Mata Psychiatric University Hospital, Reus, Spain
  • 12Hospital de la Santa Creu i Sant Pau, Barcelona, Spain

Severity is the main component of the ICD-11 personality disorder (PD) classification, but pertinent instruments have only recently been developed. We analyzed the psychometric properties of the ICD-11 Personality Disorder Severity scale (PDS-ICD-11) in a mixed sample of 726 community and clinical subjects. We also examined how the different components of the ICD-11 PD system —five trait domains, the borderline pattern specifier, and severity, all of them measured through self-reports— are interconnected and operate together. PDS-ICD-11 properties were adequate and similar to those of the original instrument. However, regressions and factor analyses showed a considerable overlap of severity with the five personality domains and the borderline specifier (72.6%). Bifactor modeling resulted in a general factor of PD (g-PD) that was not equivalent to severity nor improved criterion validity. The whole ICD-11 PD system, i.e., five personality domains, borderline, and severity, explained an average of 43.6% of variance of external measures of well-being, disability, and clinical problems, with severity contributing 4.8%. Suggestions to further improve the ICD-11 PD taxonomy include remodeling the present definition of severity to give more weight to the real-life consequences of traits.

1. Introduction

Severity is the most important component of personality disorder (PD) diagnosis in the newer International Classification of Diseases, 11th Revision (ICD-11) (1). The diagnostic process first involves rating the subject into five levels of disturbance, from none to severe PD. The most prominent personality traits can then be optionally described according to five domains — negative affectivity, detachment, dissociality, disinhibition, and anankastia— plus a borderline pattern specifier based on the Diagnostic and Statistical Manual of Mental Disorders, 5th Edition (DSM-5) (2).

Whereas the relevance of PD severity —termed personality dysfunction in the DSM-5—, has been fostered for 30 years (35), its definition has been changing all the while (6). Some definitions have relied upon the consequences of traits, such as impairment in several life areas, subjective distress, risk of serious self-injury or death, risk to others, or service utilization (7, 8). Other definitions have revolved around symptomatic complexity, including the total number of PD criteria or disorders, the involvement of more than one cluster, or the presence of particular diagnoses, mostly borderline (3, 9, 10). In this case, severity is assumed to capture comorbidity between individual disorders, and is tightly related to the notion of a general factor that underlies all specific PDs (g-PD) (1115). Still other definitions, rooted in the psychoanalytic literature, have focused on dysfunction of the self, such as compromised reality testing, primitive defense mechanisms, and identity diffusion (5, 16). For the time being, there is insufficient evidence on how these diverse conceptions of severity relate to each other or whether any of them should be preferred over the rest (7, 8). In fact, whereas the initial ICD-11 proposal accentuated major problems in occupational, social, and/or personal relationships (17), the final version incorporated self-dysfunction in order to bring itself into line with the DSM-5 (1820), and both approaches are now pretty similar (2123).

Among the instruments developed to measure severity, the Standardized Assessment of Severity of Personality Disorder (SASPD) (24) was a premature attempt, as it did not yet reflect the last-minute changes in the definition (21). An updated proposal are the still unnamed scales by Clark et al. (25) that, despite acceptable psychometric properties, may need additional improvements before clinical use, and are deemed preliminary by the authors. Finally, the ICD-11 Personality Disorder Severity scale (PDS-ICD-11) (26), which is the focus of the present study, reflects in a one-to-one basis the ICD-11 requirements for severity and has worked well in its original version. However, its properties have been tested on community subjects, except for limited analyses in 87 clinical subjects (26, 27).

It is clear from the above that many critical aspects of ICD-11 severity remain to be tested. The psychometric properties of the PDS-ICD-11 need to be replicated in clinical samples and in different cultures and languages. Among these properties, careful consideration should be given to dimensionality. Although severity is usually depicted as a unidimensional construct reflecting the dysfunctional features of any PD (18, 2830), it actually encompasses very disparate or even antithetic features. For example, the PDS-ICD-11 conceives severity as an aggregate of either too low or too high sense of self-worth, insufficient or excessive goal orientation, imperious or no need for close relationships, too lax or too tight control over emotions, and so on. It is improbable that all these aspects and their opposites are equally relevant for maladaptation, so a more fine-grained analysis is warranted. Furthermore, it would be also unlikely for all personalities to show the same way of being dysfunctional. For example, fearful, psychopathic, or asocial personalities could be equally severe at the topmost level of abstraction, but may be related to quite different “types of severity” at lower levels (31). Thus, understanding severity will require careful examination at the item level.

Another underexplored aspect is the joint functioning of the whole ICD-11 PD system, that is, how its different components —personality domains, borderline specifier, and severity— relate to each other and operate together. Concerning the architecture of the diagnostic system, a twofold model that separates personality traits from severity/functioning has been gaining ground in current classifications (5, 8, 18, 3235). This model, however, requires personality and severity to be two different things in the first place, which is currently far from clear. This separation could not be proved in the case of DSM-5 personality functioning, which massively overlaps with traits (23, 3642), and this might be the case of the ICD-11 as well. Indeed, the scant evidence suggests that self-dysfunction is factorially inseparable of negative affectivity and disinhibition, and that interpersonal dysfunction is mixed with negative affectivity and detachment (25). Similar findings have been reported using diverse measures of personality and functioning (36, 43, 44), so the relationships between the descriptive and valuative components of the ICD-11 classification need further examination.

Finally, few data exist on the diagnostic utility of severity within the ICD-11 system. Previous operationalization of this construct have generally shown to be strong predictors of maladjustment, comorbid psychopathology, well-being, and treatment outcome (10, 13, 18, 22, 28, 4547), and this could also be the case of the PDS-ICD-11 (26). The point is, however, whether the same information could be provided more parsimoniously by personality traits, either individually or in the form of a g-PD factor, making severity superfluous. To date, the contribution of severity seems modest at best, both in predicting categorial PDs (31, 38, 39, 41, 4850) and external dysfunction criteria (51).

It is apparent from the above that further research is needed regarding some aspects of the ICD-11 classification of PDs. We still lack a sufficiently established measure of severity, as well as a clear idea of how the ICD-11 diagnostic system works as a whole. Our study aims 1) to analyze the psychometric properties of the Spanish version of the PDS-ICD-11 in a mixed community and clinical sample, with particular emphasis on dimensionality, 2) to examine how the different components of the ICD-11 PD system —the five trait domains, the borderline pattern specifier, and severity— interact with each other, and 3) to test how these components operate together as predictors of external measures of psychosocial impairment.

2. Materials and methods

2.1. Participants

The community sample consisted of 436 volunteers, 50.5% women, with mean age 46.3 years (SD = 18.0, range 18-87). Participants were undergraduates and their relatives and acquaintances recruited from a university in Spain. This sample was representative of the general Spanish population in terms of age (43.4 years)1 and level of education: about 17% had completed primary and lower secondary education, 37% upper secondary and post-secondary education, and the remaining 46% tertiary education. The clinical sample consisted of 290 outpatients, 67.9% women, with mean age 41.2 years (SD = 14.8, range 18–80). They were consecutively referred to the mental health units of six hospitals in Catalonia, Spain. Patients were clinically diagnosed at their respective centers, with the main diagnoses including mild to moderate affective disorders (25.2%), anxiety or phobic disorder (20.9%), mixed affective and anxious disorder (28.8%), eating disorder (1.8%), substance-related disorder (0.7%), and other disorders (22.6%) each with a frequency below 2%. No categorical diagnoses of personality disorder were made. With α = 0.05 and 1-β = 0.80, the combined sample allowed detecting correlations of 0.11. The study was approved by the ethical committees of the respective centers.

2.2. Instruments and procedure

Questionnaires were delivered to community participants by undergraduates taking part in a personality research and practice program. Participants answered anonymously and did not receive any compensation for participating. Clinical subjects filled the questionnaires in their respective hospitals, as a part of their diagnostic procedure. All questionnaires were completed individually, in paper-and-pencil format, and in the same order they are presented below.

The Personality Inventory for ICD-11 (PiCD) (52) is a 60-item Likert-type self-report measuring the five personality domains of the dimensional ICD-11 personality model: negative affectivity, detachment, dissociality, disinhibition, and anankastia (1). Each domain has 12 items rated from 1 (strongly disagree) to 5 (strongly agree). With a similar format, the Borderline Pattern Scale (BPS) (53) includes 12 items and was developed to accommodate the DSM-5 borderline diagnosis (2) into the ICD-11 classification. Both instruments have shown good psychometric properties in their Spanish versions (54, 55).

The ICD-11 Personality Disorder Severity Scale (PDS-ICD-11) (26) is a 14-item measure designed to assess the various components of PD severity in the ICD-11: four items are related to self-dysfunction (identity, self-worth, self-perception, and goals); four to interpersonal dysfunction (interest in relationships, perspective-taking, mutuality, and disagreement management); five to control over emotions, behavior, and cognition; and one measures global psychosocial impairment. The instrument was translated by four Spanish native speakers who were familiar with the constructs being measured and worked independently. The translators agreed on a common version, which was blindly back-translated by an English native speaker and compared with the original. Discrepancies were consensually resolved by the translators and the authors of the instrument.

The Level of Personality Functioning Scale–Brief Form 2.0 (LPFS-BF 2.0) (56) is a 12-item self-report measuring the self-functioning and interpersonal-functioning components of personality dysfunction described in Section III of the DSM-5. It has shown good reliability and validity in different samples and languages (57).

Subjective well-being was measured through the World Health Organization-5 Well-Being Index (WHO-5), a 5-item self-report that has been widely used to measure the impact of mental problems on quality of life (58). It measures levels of mood, energy, and interest for things in a 0-100 scale, with higher scores indicating greater well-being. Disability was measured through the 12-item self-reported World Health Organization Disability Assessment Schedule 2.0 (WHODAS 2.0) (59), which assesses difficulties due to health conditions in six different life areas: understanding and communication, self-care, mobility, interpersonal relationships, work and household roles, and community and civic roles. Scores range from 0 (no disability) to 48 (total disability). The Work and Social Adjustment Scale (WSAS) (60) is a 5-item self-reported measure of general impairment assessing the impact of an identified problem, mental condition in this case, on five areas: work, home management, social leisure, private leisure, and relationships, each rated on a scale of 0 to 8. Higher scores in a 0-40 range denote more disability.

3. Data analysis and results

3.1. PDS-ICD-11 psychometric properties

All analyses were made using R package ‘psych’ (61) and SPSS v. 25 (62) unless stated otherwise. Descriptive statistics for the ICD-11 components and the remaining instruments are shown in Supplementary Table 1. The PDS-ICD-11 showed good internal consistency, with Cronbach’s alpha of α = 0.89 and McDonald’s omega of ω = 0.93 based on the one-factor solution. All corrected item-scale correlations were ri–s > 0.50 except for item 13 (ri–s = 0.44).

An item response theory (IRT) analysis based on Samejima’s graded response model was undertaken using the “ltm” package (63) to analyze the information provided by each item (Supplementary Table 2). Items 9 (emotional control) and 2 (self-worth) showed the highest discrimination ability with a > 2.50, whereas item 13 (harm to others) showed the lowest (0.95). Items 11 (experience of reality), 12 (harm to self), and particularly item 13 (harm to others, b3 = 5.15), showed high difficulty in parameter b3, questioning the utility of the most extreme response option (“I often harm others”) (Supplementary Figure 1). This item also offered the least information (Supplementary Figure 2). The test information function shows that the highest reliability came about at theta values between 0 and 2 (Supplementary Figure 3).

The unidimensionality of the PDS-ICD-11 was tested through confirmatory factor analysis (CFA) in R package “lavaan” (64), using weighted least squares estimation. Results suggested adequate fit, with χ2 = 193.81, df = 77, p < 0.001, Comparative Fit Index CFI = 0.993, Tucker-Lewis Index TLI = 0.992, Root Mean Square Error of Approximation RMSEA = 0.046 and Standardized Root Mean Square Residual SRMR = 0.051 (Supplementary Table 2). All loadings were above 0.60 except for items 11 (experience of reality) and 13 (harm to others). Factor structure was invariant between the community and clinical samples, as given by the “sirt” package (65).

3.2. Structure of the whole ICD-11 system: PiCD, BPS, and PDS-ICD-11

3.2.1. Correlation and regression analyses

Pearson’s and disattenuated intercorrelations between the five personality domains, the borderline specifier, and severity are shown in Table 1. Strong associations were found of severity with borderline (0.83), negative affectivity (0.76), and PiCD total score (0.72), which rose to 0.92, 0.84, and 0.80 after disattenuation for reliability (66). The considerable overlap between severity, negative affectivity, and borderline can be better appreciated through an Euler diagram (Figure 1). Associations were moderate with the remaining personality domains and negligible with anankastia. Accordingly, multiple regression analyses showed that 72.6% of the variance of severity is explained by the five PiCD domains and the borderline specifier. Borderline was the best predictor of severity (beta = 0.527), followed by negative affectivity (Table 1, below). Disinhibition and dissociality showed little or no relationship with severity even after the borderline specifier was excluded, and anankastia showed an inverse association.

TABLE 1
www.frontiersin.org

Table 1. Pearson’s (lower triangle) and disattenuated correlations (upper triangle) between the ICD-11 system components with alphas in the diagonal, and multiple regressions of severity on personality traits.

FIGURE 1
www.frontiersin.org

Figure 1. Euler diagram showing the overlap between negative affectivity, borderline, and severity. Circle sizes are proportional to variances. Due to the geometric impossibility to represent the overlap between more than three variables through a proportionally-built Euler diagram, anankastia and detachment —explaining an additional 2% of the PDS-ICD-11 variance— have been omitted. Drawn at http://eulerr.co/.

3.2.2. Item-level analysis

Additionally, we wanted to clarify whether certain personality traits were associated to specific aspects of severity. To this end, two different scoring procedures for the PDS-ICD-11 were used: Whereas in the standard unipolar scoring the two poles of each item are added together and contribute interchangeably to a single dimension of severity (2-1-0-1-2), we adopted an alternative bipolar procedure in which the two poles pointed in opposite directions (0-1-2-3-4). Under unipolar scoring, Spearman’s correlations showed that all PDS-ICD-11 items were homogeneously correlated with the borderline specifier (mean rs = 0.50), negative affectivity (0.47) and PiCD total score (0.47), although with some advantage for self-worth and psychosocial impairment (Supplementary Table 3). By contrast, associations were more specific under bipolar scoring (Supplementary Table 3, below): negative affectivity and borderline became more associated to low self-worth and emotional dysregulation, whereas detachment was weakly but differentially linked to low interest in relationships (rs = 0.27), anankastia to rigid goals and behavioral overcontrol (0.25 and 0.41), disinhibition to difficulty following goals and lack of behavioral control (0.34 and 0.41), and dissociality to more disagreements and lack of behavioral control (0.28 and 0.27). However, each aspect of severity is measured by a single item in the PDS-ICD-11, and associations were generally not strong.

3.2.3. Factor analysis

The structure of the whole ICD-11 system, operationalized by the PiCD, the BPS, and the PDS-ICD-11, was then examined at the item-level through exploratory factor analysis based on the polychoric matrix (Supplementary Table 4) and unweighted least squares estimation. Kaiser-Meyer-Olkin index was 0.874 and Bartlett’s sphericity test was 7,569.7 (df = 3,655; p = 0.00001), indicating suitability of data for factor analysis. Velicer’s MAP and optimal implementation of parallel test (67) suggested four and five factors, respectively. One to seven factors were successively retained, rotated to Promin, and examined. The four-factor solution (Supplementary Table 5) reproduced the usual structure containing disinhibition-anankastia (r = −0.79 and 0.85 with the original domain scores), dissociality (0.95), and detachment (0.94), along with a broader factor gathering negative affectivity, borderline, and severity together (0.94, 0.94, and 0.87). Solutions of five or more factors produced empty or trivial factors, and were unsuccessful in separating this latter factor into its original components.

Additionally, we adopted a pure exploratory bifactor approach (68) implemented in FACTOR 12.01.02 (69) to clarify whether a general factor of personality disorder (g-PD) could be extracted from the ICD-11 personality descriptors —five domains and the borderline specifier— and whether this g-PD would be equivalent to severity. KMO was 0.902 and Bartlett’s statistic 7,842.2 (df = 2,556; P = 0.00001). MAP and parallel test suggested retaining four and five specific factors, respectively. Whereas the former solution had one empty and one uninterpretable factor, the five-factor solution showed a clear g-PD with 72% of items loading over 0.30 and five specific factors roughly representing detachment (r = 0.56 with the original domain), negative affectivity-borderline (0.67 and 0.62), dissociality (0.64), anankastia (0.56), and a factor gathering disinhibition items but with little relation with the original domain (0.29) (Supplementary Table 6). The g-PD was mostly associated to negative affectivity and borderline (0.70 and 0.73) and moderately to the remaining personality domains (0.29 to 0.62), but could not be regarded as exactly equivalent to severity (0.66) or the LPFS-BF (0.69).

3.3. Concurrent and criterion validity of the ICD-11 system

3.3.1. Correlation and regression analyses

We examined the relationships of the ICD-11 components with external indicators of maladaptation: caseness (belonging to the clinical sample, as an indicator of clinically significant problems), as well as three World Health Organization (WHO) scales reflecting well-being and disability. Severity showed the strongest correlation with all indicators (mean r = 0.60), albeit with little advantage over negative affectivity (0.59), borderline (0.57), and the DSM-5 LPFS-BF (0.58), with which severity correlated 0.81. PiCD total and the g-PD showed slightly lower averages (0.52 and 0.49), whereas anankastia showed no relation to maladaptation (Table 2). Due to the extensive overlap among ICD-11 components, the predictive utility of diverse combinations of these components was then analyzed through multiple regression. The whole ICD-11 system —five personality domains (PiCD), the borderline specifier (BPS), and severity (PDS-ICD-11)— predicted 40.6% of the variance of the WHODAS, 45.8% of the WHO-5, 47.9% of the WSAS, and 39.9% of caseness (Table 2, below). The five personality domains alone were better predictors than severity alone. However, both severity and the personality domains added incremental variance to each other (4.8 and 6.3%, respectively). Among personality domains, disinhibition, dissociality, and anankastia were non-significantly or inversely related to maladaptation. The exclusion of the borderline specifier was inconsequential for prediction. Interactions of personality traits with severity, which reflect the additional contribution of having an extreme trait plus high severity, were non-significant too.

TABLE 2
www.frontiersin.org

Table 2. Correlations of the ICD-11 components with external indicators of well-being, disability, and clinical problems, and multiple regressions of indicators on ICD-11 components.

3.3.2. Item-level analysis

However, the best predictions resulted from replacing the PDS-ICD-11 scale with its items in regression analyses. All models invariably retained items 2 and 14, suggesting that self-worth and global psychosocial impairment are particularly relevant aspects of severity. Other items made smaller contributions that fluctuated across models. Mean incremental variance of items over the five personality domains was 7.4% (Table 2). Supplementary Figure 4 shows that most PDS-ICD-11 items are more maladaptive in one of their extremes. Namely, having a weak sense of identity, feeling worthless, perceiving few strengths in oneself, lacking emotional control, losing touch with reality under stress, harming oneself or others, and showing psychosocial impairment in several important areas of life (items 1 to 3, 9, and 11 to 14), are more detrimental than their opposites. By contrast, having too rigid goals and being over-controlled are just as bad as lacking any goal at all and being impulsive (items 4 and 10). Similarly, in the interpersonal domain, being uninterested in relationships, unempathetic, selfish, or adversarial (items 5 to 8) is almost as bad as being afraid of loneliness and disagreements, or being too kind or clingy.

3.3.3. ROC analysis

Finally, the seven components of the ICD-11 PD system were tested as for their ability to predict caseness through receiver operating characteristics (ROC) analysis. Negative affectivity and severity obtained virtually identical areas under the curve (AUC 0.844 and 0.842, respectively), closely followed by borderline (0.812) (Figure 2). The remaining scales showed AUCs between 0.501 and 0.684. Although the Youden’s index suggested a cutpoint of 10 (resulting in 69% sensitivity and 85% specificity), a still lower threshold of 8 would be necessary to detect at least 80% of cases whereas still discarding 73% of non-cases. The proposed cutoff of 17.5 (26, 27) would only detect about 22% of cases.

FIGURE 2
www.frontiersin.org

Figure 2. ROC curves for the components of the ICD-11 PD system predicting caseness.

4. Discussion

The PDS-ICD-11 shows good psychometric properties in its Spanish version, similar to those of the original and the German versions (26, 27). Specifically, it has high internal consistency and a well-fitted unidimensional structure, and it is among the best predictors of external indicators of well-being, disability, and clinical problems. This makes it a suitable instrument for measuring the severity dimension of the ICD-11 PD system.

On the other hand, some unexpected results suggest that there remains scope for improvement. The ICD-11 diagnostic system does not explicitly assume that personality domains and severity are independent constructs. Rather, elevations in severity are expected to be associated to elevations in one or several personality domains, which reflect distinct styles of malfunctioning. However, what is unanticipated is the ample overlap between personality traits and severity/dysfunction which has been repeatedly found regarding both the ICD-11 and DSM-5 systems (25, 36, 38, 70). In our study, almost three quarters of the variance of severity is accounted for by personality descriptors, particularly the negative affectivity domain and the borderline specifier. In fact, these three components cannot be split and are virtually the same thing from a factor-analytic perspective (25, 55). A major consequence is that severity plays a smaller role than expected in predicting maladaptation, and borderline plays no role whatsoever; for this purpose, negative affectivity alone would work almost as well. This result is rather expected for the borderline specifier, which was appended to the system at the eleventh hour and shows little specific variance (55), but it deserves further consideration regarding severity. As a second point, we could not confirm either that severity reflects the maladaptation resulting from a wide range of dysfunctional personality styles. Instead, regression analyses reveal that the PDS-ICD-11 provides little or no coverage of the possible impairments linked to disinhibition, dissociality, and anankastia (Table 1). This has been also the case with the German PDS-ICD-11 regarding the two latter domains (27).

One possible explanation is that, indeed, certain traits are harmful whereas others are innocuous. For example, negative emotionality has proven to be definitely impairing with regard to a broad range of clinical outcomes, whereas all other dimensions are associated to only a few detrimental outcomes or are clinically inconsequential (71). In fact, anankastic features such as perfectionism, workaholism, rigidity, or even the obsessive-compulsive PD, have shown to be fairly harmless (29, 72, 73), or even beneficial in certain areas (71, 74), and the same is true of disinhibition and dissociality (71, 7577). In accordance with this, these domains are also unrelated in our study to either caseness or the WHO scales. A second possibility is that the ICD-11 and DSM-5 unidimensional constructs of severity/dysfunction are pervaded by the frequency and intensity of aversive emotions, which is the defining feature of negative affectivity, but they do not reflect the type of impairment characteristic of most other traits. Only when we take advantage of the bipolar nature of the PDS-ICD-11 items (Supplementary Table 3) we can appreciate that anankastia causes subjects to adhere rigidly to unreachable goals and impulse control, that detachment results in social isolation, that dissociality turns disagreements into major conflicts, and that disinhibition prevents subjects from attaining their predefined goals. Thus, the unidimensional construct of severity/dysfunction may overdetect distress-related problems but be blind to the many other ways of being maladapted. A third possibility is that, whatever it is the domain that is causing problems in the first place, most patients may ultimately seek help because of distress and demoralization, which would thrust both severity and negative affectivity at the same time (78). This is consistent with the fact that all domains (except anankastia) are significantly associated to severity until negative affectivity is controlled for through regression (Table 1). A final possibility is that it is the PDS-ICD-11, not the severity construct in itself, which is biased toward negative affect. In this case, clinician ratings, interviews, or other self-reports available in the near future could be better able to differentiate severity from negative affect, or to capture the maladaptive aspects of disinhibition, dissociality, and anankastia. For example, it has been reported that personality functioning overlaps with traits to a lesser extent when it is measured longitudinally through daily diaries (79). By contrast, the only study using clinician ratings of ICD-11 severity shows results which are very similar to those of self-reports (80). All these possible explanations are not mutually exclusive.

No consensual solution to these shortcomings has been found so far, and the debate is ongoing (28). Given that severity is the pivotal component of the system, and that personality domains are only optional ways to identify particular styles of malfunctioning, it has been proposed that it is domains which should be amended or replaced with normal-range traits to reduce overlap (6, 42, 81). Normal traits, however, such as those of the Five Factor Model, have shown to be equally related to severity, so the problem is left unsolved (6, 39). A more statistically sophisticated solution is the extraction of a general factor of PD (g-PD) that accounts for the common variance across disorders or traits (11, 12, 15). This approach significantly reduces the overlap between domains (82), and the resulting g-PD is a good indicator of maladaptation, thus precluding the need for a separate assessment of severity (13, 14, 30). However, the g-PD does not work well in our study: It is not equivalent to either ICD-11 severity or DSM-5 personality functioning, and it does not bring significant advantage over preexisting variables in predicting well-being and disability. An additional drawback of this approach is that it provides the clinician with a broad factor whose meaning we can barely envisage (8385), and a number of specific factors with no less ambiguous significance. For example, one might wonder what negative emotionality would look like after distress and impairment have been removed, if such a thing exists at all, and what is the point of having a so inert domain in a diagnostic system.

Thus, psychometric refinement may not be sufficient to break this deadlock, mainly caused by the fact that severity and negative affectivity are largely the same. Another proposed solution has been the elimination of severity in its current form (38, 40). However, severity is upheld not only on empirical but also on theoretical grounds, and so its conceptual foundations need previous clarification and discussion. Impairments in self- and interpersonal functioning have been emphasized in many widely accepted models of PD, as those of Kernberg, Livesley, Parker, or Cloninger (16, 8688). A central assumption underpinning these models and, by extension, the ICD-11 and DSM-5 systems, is that the main components of severity/dysfunction —self-direction, sense of identity, empathy, intimacy— are of a different nature than personality traits. Concretely, they have been considered to be core features, basic psychological capacities, meta-constructs, key components of an intrapsychic system needed to fulfill universal life tasks, or even the cornerstone of humanness (5, 28, 86, 8891). These appellations do not clarify, however, what exactly makes them different from all remaining traits (92). For example, there is no reason to think that these features reflect capacities or serve universal life tasks to a greater extent than any other trait. In fact, all traits reflect the variation of brain systems that enable and impel us to perform essential life-sustaining tasks: detecting and managing threats, exploring the surroundings, striving for incentives, gaining power or status, becoming attached to others, deterring rivals, and dozens of equally —if no more— important pursuits (9397). We also lack evidence to assert that identity or empathy are either more universal or consequential, or are more central to PDs, than all others. And certainly, we do not know which —if any— is the cornerstone of humanness. In essence, self- and interpersonal dysfunctions may simply reflect the fact that most PDs are underlain by neuroticism and disagreeableness (98, 99). A second, closely related assumption of current classifications is that the components of severity/dysfunction are inherently maladaptive. For example, whereas negative affectivity, disinhibition, or dissociality need an additional criterion to be considered pathological in the ICD-11 system, the deleterious nature of low empathy or self-directedness is taken for granted. In practice, this leads to a tautological diagnostic process in which the severity of some traits is determined on the basis of other —or even the same— traits. On empirical grounds, this is at odds with the well-known fact that negative affectivity is the most maladaptive trait ever found (71, 100, 101). By contrast, the premise that self-complexity, low self-directedness, aloofness, or lack of empathy are dysfunctional in themselves has no comparable support (71, 7577, 102104). Although the abovementioned assumptions are difficult to prove or refute at this time, they certainly warrant in-depth examination and debate with a view to future revisions of the taxonomy.

A promising alternative may be the replacement —or the complementation— of the current severity construct with an assessment of the negative consequences of traits (33, 71, 92, 105108). Negative consequences have been categorized in many ways, but they generally include difficulties or failures in a number of key areas: education/work, e.g., inability to finish studies, hold a paid job, or achieve financial independence; interpersonal functioning, e.g., trouble finding or maintaining romantic relationships, chronic conflict with family, or lack of a support network; social functioning, e.g., difficulties for life in society, breaking rules of coexistence, or harming others; physical health and longevity, e.g., problems for self-care, harming oneself, or putting oneself at risk of death; and psychological health, e.g., chronic suffering, substance abuse, psychopathology, requiring specialized care or hospitalization, or being unable to attain acceptable levels of well-being (71, 109111). This is a redress rather than a turning point, as the PDS-ICD-11 already encompasses outcomes such as harm to self, harm to others, or psychosocial impairment. However, the focus is moved further from theory-driven intrapsychic constructs towards concrete outcomes in the real world, which may bring a number of interwoven advantages. First, life outcomes are clearly different from traits, which reduces conceptual confusion and redundancy. Second, this approach recovers a more pragmatic view of PDs as extreme traits causing suffering or functional impairment (112), rather than as pathological entities characterized by poorly known intrapsychic processes. Third, it puts the accent on functional status, the most difficult aspect to attain and maintain in severe PD patients, and then a primary goal for intervention (113). Fourth, it gives us the chance of exploring from scratch what traits change what aspects of our lives for the worse (or for the better). As a final advantage, it excludes the possibility of diagnosing a PD in the absence of negative consequences in the real world (33), and thus the risk of pathologizing normal behavior. In doing so, it offsets the increasing trend toward assuming that PDs are defects, illnesses, or dysfunctions sensu Wakefield: the failure of a mental mechanism to perform its evolved function (34). On the contrary, a life-outcome perspective focuses on whether traits are harmful —negative or undesirable by social standards (34, 114117)— and then fits in better with the fact that, at present, no evidence supports a dysfunction model for PDs. Whereas we know what normal lungs and hearts look like and what they are expected to do, this is not the case of “normal” personality (118). On the other hand, what is normal in nature, from insects to higher primates and humans, is the coexistence of different personalities, often maintained by balancing selection (119, 120). For example, certain traits may damage important biological goals while promoting others, or may be globally beneficial or detrimental depending on environmental circumstances, thus remaining in the population at evolutionary equilibrium. The very existence of a single “normal” personality would then be an evolutionary anomaly (121, 122).

Whereas this approach may lay the foundations for a less conjectural and more pragmatic taxonomy of difficult personalities, some caution is needed. On the one hand, it is not as objective as it may seem, as we cannot establish which life outcomes are undesirable and which constitute a ‘good life’ without a significant amount of subjectivity and theorizing (114). On the other, personality functioning and psychosocial impairment are different constructs. Whereas the former is about what do all PDs have in common (e.g., low self-directedness, impaired mentalizing, the g-PD), and then about the mechanisms of personality pathology (28, 123), the latter refers to the impact of traits on the life of individuals. It is argued, therefore, that the consequences of disease are essential in clinical decision making but are not the disease itself, and cannot be part of diagnosis (28). In turn, this assumes that PDs are diseases rather than disliked traits, a point on which we are far from a consensus (114117). Ultimately, even if both approaches are deemed complementary, life outcomes have been relatively overlooked in current classifications and deserve greater consideration they have received so far.

Some caveats on the scope of this study are in order. First, the instruments are not the model, so that the shortcomings identified in the ICD-11 diagnostic system could be circumscribed to specific questionnaires such as the PiCD, the BPS or the PDS-ICD-11. Moreover, all instruments are self-reported, which may produce common-method bias and overestimate overlapping (124). Therefore, the generalizability or our results depends on their eventual confirmation using different tools, preferably interviews or clinician rating forms, when they are developed. Second, the same is true of criterion variables. Particularly, caseness cannot be considered equivalent to PD diagnosis, but it is a general indicator of clinically significant problems. Other criteria suggestive of disordered personality need to be tested in upcoming studies. Third, certain parts or our analysis are performed at the item level. Even if severity is a complex construct and its components need to be examined separately, it should be taken into account that items are less reliable than scales, and these results should be interpreted cautiously. Finally, our sample included less than 300 patients. This is enough to reach our study’s aims but did not allow deeper examination of different levels of severity, which is an important feature of the ICD-11 system.

With these objections in mind, we conclude that the PDS-ICD-11 has proven adequate properties as a measure of severity. However, the ICD-11 system as a whole is conditioned by important limiting factors. Whereas the adoption of a dimensional taxonomy has meant a significant improvement, the overlap between personality domains —one major reason for the abandonment of traditional categories (35)— remains a problem (81, 82). Furthermore, while the conceptual separation between personality traits and severity/dysfunction is widely accepted and helpful, the existing operationalizations of severity lack solid theoretical justification and are too close to negative affectivity to serve diagnostic purposes. Irrespective of whether this is due to personality traits containing variance of impairment (29) or the other way around (38, 39), the result is a redundant and conceptually confusing diagnostic system. In contrast, the types of impairment resulting from disinhibition, dissociality, and anankastia are not well reflected by severity, or these traits are not significantly harmful, as suggested by previous research (71, 74). We take up previous suggestions that a model with refined, truly independent domains (82), followed by a list of undesired life consequences would be more parsimonious, feasible, and theoretically clearer than the current approach (33, 106).

Data availability statement

The datasets presented in this study can be found in online repositories. The names of the repository/repositories and accession number(s) can be found below: https://osf.io/mvyug/?view_only=94d52cde2dd540b09665d775a9eea52e.

Ethics statement

The studies involving human participants were reviewed and approved by Comité de Ética de Investigación con Medicamentos (CEIm, Drug Research Ethics Committee), Hospital Clínic, Barcelona, Spain. The patients/participants provided their written informed consent to participate in this study.

Author contributions

FG, AA, CR, MG, and JMP contributed to the conception and design of the study. FG and CR performed the statistical analyses and wrote the first draft of the manuscript. CR, SG, NC, MF, AGZ, JS, and JCP made amendments to the manuscript and rewrote parts of it. All authors recruited samples in their respective centers, assessed outpatients, organized the database, contributed to the manuscript revision, and approved the submitted version.

Funding

Open access publication fees were financed by the Hospital Clínic de Barcelona (Spain).

Conflict of interest

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Supplementary material

The Supplementary Material for this article can be found online at: https://www.frontiersin.org/articles/10.3389/fpsyt.2022.1015489/full#supplementary-material

Footnotes

  1. ^ www.ine.es

References

1. World Health Organization [WHO]. ICD-11 clinical descriptions and diagnostic guidelines for mental and behavioural disorders. Geneva: World Health Organization (2019).

Google Scholar

2. American Psychiatric Association [APA]. Diagnostic and statistical manual of mental disorders. 5th ed. Arlington, VA: American Psychiatric Association (2013).

Google Scholar

3. Tyrer P, Johnson T. Establishing the severity of personality disorder. Am J Psychiatry. (1996) 153:1593–7. doi: 10.1176/ajp.153.12.1593

PubMed Abstract | CrossRef Full Text | Google Scholar

4. Tyrer P. The problem of severity in the classification of personality disorder. J Pers Disord. (2005) 19:309–31. doi: 10.1521/pedi.2005.19.3.309

PubMed Abstract | CrossRef Full Text | Google Scholar

5. Morey L, Berghuis H, Bender D, Verheul R, Krueger R, Skodol A. Toward a model for assessing level of personality functioning in DSM–5, part II: empirical articulation of a core dimension of personality pathology. J Pers Assess. (2011) 93:347–53. doi: 10.1080/00223891.2011.577853

PubMed Abstract | CrossRef Full Text | Google Scholar

6. Hopwood C, Malone J, Ansell E, Sanislow C, Grilo C, McGlashan T, et al. Personality assessment in DSM-5: empirical support for rating severity, style, and traits. J Pers Disord. (2011) 25:305–20. doi: 10.1521/pedi.2011.25.3.305

PubMed Abstract | CrossRef Full Text | Google Scholar

7. Crawford M, Koldobsky N, Mulder R, Tyrer P. Classifying personality disorder according to severity. J Pers Disord. (2011) 25:321–30. doi: 10.1521/pedi.2011.25.3.321

PubMed Abstract | CrossRef Full Text | Google Scholar

8. Zimmerman M, Morgan T, Stanton K. The severity of psychiatric disorders. World Psychiatry. (2018) 17:258–75. doi: 10.1002/wps.20569

PubMed Abstract | CrossRef Full Text | Google Scholar

9. Soeteman D, Verheul R, Busschbach J. The burden of disease in personality disorders: diagnosis-specific quality of life. J Pers Disord. (2008) 22:259–68. doi: 10.1521/pedi.2008.22.3.259

PubMed Abstract | CrossRef Full Text | Google Scholar

10. Clark L, Nuzum H, Ro E. Manifestations of personality impairment severity: comorbidity, course/prognosis, psychosocial dysfunction, and ‘borderline’ personality features. Curr Opin Psychol. (2018) 21:117–21. doi: 10.1016/j.copsyc.2017.12.004

PubMed Abstract | CrossRef Full Text | Google Scholar

11. Oltmanns J, Smith G, Oltmanns T, Widiger T. General factors of psychopathology, personality, and personality disorder: across domain comparisons. Clin Psychol Sci. (2018) 6:581–9. doi: 10.1177/2167702617750150

PubMed Abstract | CrossRef Full Text | Google Scholar

12. Sharp C, Wright A, Fowler J, Frueh B, Allen J, Oldham J, et al. The structure of personality pathology: both general (‘g’) and specific (‘s’) factors? J Abnorm Psychol. (2015) 124:387–98. doi: 10.1037/abn0000033

PubMed Abstract | CrossRef Full Text | Google Scholar

13. Conway C, Hammen C, Brennan P. Optimizing prediction of psychosocial and clinical outcomes with a transdiagnostic model of personality disorder. J Pers Disord. (2016) 30:545–66. doi: 10.1521/pedi_2015_29_218

CrossRef Full Text | Google Scholar

14. Williams T, Scalco M, Simms L. The construct validity of general and specific dimensions of personality pathology. Psychol Med. (2018) 48:834–48. doi: 10.1017/S0033291717002227

PubMed Abstract | CrossRef Full Text | Google Scholar

15. McCabe G, Oltmanns J, Widiger T. The general factors of personality disorder, psychopathology, and personality. J Pers Disord. (2022) 36:129–56. doi: 10.1521/pedi_2021_35_530

CrossRef Full Text | Google Scholar

16. Kernberg O. Severe personality disorders: psychotherapeutic strategies. New Haven: Yale University Press (1984).

Google Scholar

17. Tyrer P, Crawford M, Mulder R, Blashfield R, Farnam A, Fossati A, et al. The rationale for the reclassification of personality disorder in the 11th revision of the International Classification of Diseases (ICD-11). Personal Ment Health. (2011) 5:246–59. doi: 10.1002/pmh.190

CrossRef Full Text | Google Scholar

18. Tyrer P, Mulder R, Kim Y, Crawford M. The development of the ICD-11 classification of personality disorders: an amalgam of science, pragmatism, and politics. Annu Rev Clin Psychol. (2019) 15:481–502. doi: 10.1146/annurev-clinpsy-050718-095736

PubMed Abstract | CrossRef Full Text | Google Scholar

19. Mulder RT. ICD-11 personality disorders: utility and implications of the new model. Front Psychiatry. (2021) 12:655548. doi: 10.3389/fpsyt.2021.655548

PubMed Abstract | CrossRef Full Text | Google Scholar

20. Bach B, Anderson J. Patient-reported ICD-11 Personality disorder severity and DSM-5 level of personality functioning. J Pers Disord. (2020) 34:231–49. doi: 10.1521/pedi_2018_32_393

CrossRef Full Text | Google Scholar

21. Zimmermann J, Müller S, Bach B, Hutsebaut J, Hummelen B, Fischer F. A common metric for self-reported severity of personality disorder. Psychopathology. (2020) 53:168–78. doi: 10.1159/000507377

CrossRef Full Text | Google Scholar

22. Bach B, Simonsen S. How does level of personality functioning inform clinical management and treatment? Implications for ICD-11 classification of personality disorder severity. Curr Opin Psychiatry. (2021) 34:54–63. doi: 10.1097/YCO.0000000000000658

PubMed Abstract | CrossRef Full Text | Google Scholar

23. McCabe G, Widiger TA. A comprehensive comparison of the ICD-11 and DSM-5 section III personality disorder models. Psychol Assess. (2020) 32:72–84. doi: 10.1037/pas0000772

PubMed Abstract | CrossRef Full Text | Google Scholar

24. Olajide K, Munjiza J, Moran P, O’Connell L, Newton-Howes G, Bassett P, et al. Development and psychometric properties of the standardized assessment of severity of personality disorder (SASPD). J Pers Disord. (2018) 32:44–56. doi: 10.1521/pedi_2017_31_285

CrossRef Full Text | Google Scholar

25. Clark L, Corona-Espinosa A, Khoo S, Kotelnikova Y, Levin-Aspenson H, Serapio-García G, et al. Preliminary scales for ICD-11 personality disorder: self and interpersonal dysfunction plus five personality disorder trait domains. Front Psychol. (2021) 12:668724. doi: 10.3389/fpsyg.2021.668724

PubMed Abstract | CrossRef Full Text | Google Scholar

26. Bach B, Brown T, Mulder R, Newton-Howes G, Simonsen E, Sellbom M. Development and initial evaluation of the ICD-11 personality disorder severity scale: PDS-ICD-11. Personal Ment Health. (2021) 15:223–36. doi: 10.1002/pmh

CrossRef Full Text | Google Scholar

27. Zimmermann J, Falk C, Wendt L, Spitzer C, Fischer F, Bach B, et al. Validating the German version of the personality disorder severity-ICD-11 scale using nominal response models. Psychol Assess. (2022). doi: 10.1037/pas0001199 [Epub ahead of print].

PubMed Abstract | CrossRef Full Text | Google Scholar

28. Sharp C, Wall K. DSM-5 level of personality functioning: refocusing personality disorder on what it means to be human. Annu Rev Clin Psychol. (2021) 17:313–37. doi: 10.1146/annurev-clinpsy-081219-105402

PubMed Abstract | CrossRef Full Text | Google Scholar

29. Morey L. Thoughts on the assessment of the DSM–5 alternative model for personality disorders: comment on sleep et al. (2019). Psychol Assess. (2019) 31:1192–9. doi: 10.1037/pas0000710

PubMed Abstract | CrossRef Full Text | Google Scholar

30. Hopwood C, Good E, Morey L. Validity of the DSM–5 levels of personality functioning scale-self report. J Pers Assess. (2018) 100:650–9. doi: 10.1080/00223891.2017.1420660

PubMed Abstract | CrossRef Full Text | Google Scholar

31. Anderson J, Sellbom M. Evaluating the DSM-5 Section III personality disorder impairment criteria. Personal Disord. (2018) 9:51–61. doi: 10.1037/per0000217

PubMed Abstract | CrossRef Full Text | Google Scholar

32. Pincus A. Some comments on nomology, diagnostic process, and narcissistic personality disorder in the DSM-5 proposal for personality and personality disorders. Personal Disord. (2011) 2:41–53. doi: 10.1037/a0021191

PubMed Abstract | CrossRef Full Text | Google Scholar

33. Leising D, Zimmermann J. An integrative conceptual framework for assessing personality and personality pathology. Rev Gen Psychol. (2011) 15:317–30. doi: 10.1037/a0025070

CrossRef Full Text | Google Scholar

34. Wakefield J. Disorder as harmful dysfunction: a conceptual critique of DSM-III-R’s definition of mental disorder. Psychol Rev. (1992) 99:232–47. doi: 10.1037/0033-295x.99.2.232

PubMed Abstract | CrossRef Full Text | Google Scholar

35. Widiger T, Trull T. Plate tectonics in the classification of personality disorder: shifting to a dimensional model. Am Psychol. (2007) 62:71–83. doi: 10.1037/0003-066X.62.2.71

PubMed Abstract | CrossRef Full Text | Google Scholar

36. Clark L, Ro E. Three-pronged assessment and diagnosis of personality disorder and its consequences: personality functioning, pathological traits, and psychosocial disability. Personal Disord. (2014) 5:55–69. doi: 10.1037/per0000063

PubMed Abstract | CrossRef Full Text | Google Scholar

37. Fossati A, Borroni S, Somma A, Markon K, Krueger R. Testing relationships between DSM-5 Section III maladaptive traits and measures of self and interpersonal impairment in Italian community dwelling adults. Personal Disord. (2017) 8:275–80. doi: 10.1037/per0000192

PubMed Abstract | CrossRef Full Text | Google Scholar

38. Sleep C, Lynam D, Widiger T, Crowe M, Miller J. An evaluation of DSM-5 section III personality disorder criterion a (impairment) in accounting for psychopathology. Psychol Assess. (2019) 31:1181–91. doi: 10.1037/pas0000620

PubMed Abstract | CrossRef Full Text | Google Scholar

39. Sleep C, Weiss B, Lynam D, Miller J. The DSM-5 section III personality disorder criterion A in relation to both pathological and general personality traits. Personal Disord. (2020) 11:202–12. doi: 10.1037/per0000383

PubMed Abstract | CrossRef Full Text | Google Scholar

40. Sleep C, Lynam D. The problems with criterion A: a comment on Morey et al. (2022). Personal Disord. (2022) 3:325–7. doi: 10.1037/per0000585

PubMed Abstract | CrossRef Full Text | Google Scholar

41. Widiger T, Bach B, Chmielewski M, Clark L, DeYoung C, Hopwood C, et al. Criterion a of the AMPD in HiTOP. J Pers Assess. (2019) 101:345–55. doi: 10.1080/00223891.2018.1465431

PubMed Abstract | CrossRef Full Text | Google Scholar

42. Morey L, McCredie M, Bender D, Skodol A. Criterion A: level of personality functioning in the alternative DSM-5 model for personality disorders. Personal Disord. (2022) 13:305–15. doi: 10.1037/per0000551

PubMed Abstract | CrossRef Full Text | Google Scholar

43. Farmer R, Goldberg L. Brain modules, personality layers, planes of being, spiral structures, and the equally implausible distinction between TCI-R “temperament” and “character” scales: a reply to Cloninger (2008). Psychol Assess. (2008) 20:300–4. doi: 10.1037/a0012932

PubMed Abstract | CrossRef Full Text | Google Scholar

44. Farmer R, Goldberg LR. A psychometric evaluation of the revised temperament and character inventory (TCI-R) and the TCI-140. Psychol Assess. (2008) 20:281–91. doi: 10.1037/a0012934

PubMed Abstract | CrossRef Full Text | Google Scholar

45. Sharp C. Fulfilling the promise of the LPF: comment on Morey et al. (2022). Personal Disord. (2022) 13:316–20. doi: 10.1037/per0000567

PubMed Abstract | CrossRef Full Text | Google Scholar

46. Buer Christensen T, Eikenaes I, Hummelen B, Pedersen G, Nysćter T, Bender D, et al. Level of personality functioning as a predictor of psychosocial functioning-concurrent validity of criterion A. Personal Disord. (2020) 11:79–90. doi: 10.1037/per0000352

PubMed Abstract | CrossRef Full Text | Google Scholar

47. Veenstra M, van Dijk S, Bouman R, van Alphen S, van Asselt A, van den Brink R, et al. Impact of personality functioning and pathological traits on mental wellbeing of older patients with personality disorders. BMC Psychiatry. (2022) 22:214. doi: 10.1186/s12888-022-03857-8

PubMed Abstract | CrossRef Full Text | Google Scholar

48. Few L, Miller J, Rothbaum A, Meller S, Maples J, Terry D, et al. Examination of the section III DSM-5 diagnostic system for personality disorders in an outpatient clinical sample. J Abnorm Psychol. (2013) 122:1057–69. doi: 10.1037/a0034878

PubMed Abstract | CrossRef Full Text | Google Scholar

49. Zimmermann J, Kerber A, Rek K, Hopwood C, Krueger RF. A brief but comprehensive review of research on the alternative DSM-5 model for personality disorders. Curr Psychiatry Rep. (2019) 21:92. doi: 10.1007/s11920-019-1079-z

PubMed Abstract | CrossRef Full Text | Google Scholar

50. Nysaeter T, Hummelen B, Christensen T, Eikenaes I, Selvik S, Pedersen G, et al. The incremental utility of criteria A and B of the DSM-5 alternative model for personality disorders for predicting DSM-IV/DSM-5 section II personality disorders. J Pers Assess. (2022) 14:1–10. doi: 10.1080/00223891.2022.2039166

PubMed Abstract | CrossRef Full Text | Google Scholar

51. Roche M, Jaweed S. Comparing measures of criterion A to better understand incremental validity in the alternative model of personality disorders. Assessment. (2021). doi: 10.1177/10731911211059763 [Epub ahead of print].

PubMed Abstract | CrossRef Full Text | Google Scholar

52. Oltmanns J, Widiger TA. A self-report measure for the ICD-11 dimensional trait model proposal: the personality inventory for ICD-11. Psychol Assess. (2018) 30:154–69. doi: 10.1037/pas0000459

PubMed Abstract | CrossRef Full Text | Google Scholar

53. Oltmanns J, Widiger T. Evaluating the assessment of the ICD-11 personality disorder diagnostic system. Psychol Assess. (2019) 31:674–84. doi: 10.1037/pas0000693

PubMed Abstract | CrossRef Full Text | Google Scholar

54. Gutiérrez F, Aluja A, Ruiz J, García L, Gárriz M, Gutiérrez-Zotes A, et al. Personality disorders in the ICD-11: Spanish validation of the PiCD and the SASPD in a mixed community and clinical sample. Assessment. (2021) 28:759–72. doi: 10.1177/1073191120936357

PubMed Abstract | CrossRef Full Text | Google Scholar

55. Gutiérrez F, Aluja A, Ruiz Rodríguez J, Peri J, Gárriz M, Garcia L, et al. Borderline, where are you? a psychometric approach to the personality domains in the International Classification of Diseases, 11th Revision (ICD-11). Personal Disord. (2022). doi: 10.1037/per0000592 [Epub ahead of print].

PubMed Abstract | CrossRef Full Text | Google Scholar

56. Weekers L, Hutsebaut J, Kamphuis J. The level of personality functioning scale-brief form 2.0: update of a brief instrument for assessing level of personality functioning. Personal Ment Health. (2019) 13:3–14. doi: 10.1002/pmh.1434

PubMed Abstract | CrossRef Full Text | Google Scholar

57. Birkhölzer M, Schmeck K, Goth K. Assessment of criterion A. Curr Opin Psychol. (2021) 37:98–103. doi: 10.1016/j.copsyc.2020.09.009

PubMed Abstract | CrossRef Full Text | Google Scholar

58. Topp C, Østergaard S, Søndergaard S, Bech P. The WHO-5 well-being index: a systematic review of the literature. Psychother Psychosom. (2015) 84:167–76. doi: 10.1159/000376585

PubMed Abstract | CrossRef Full Text | Google Scholar

59. Rehm J, Üstün T, Saxena S, Nelson C, Chatterji S, Ivis F. On the development and psychometric testing of the WHO screening instrument to assess disablement in the general population. Int J Methods Psychiatr Res. (1999) 8:110–22. doi: 10.1002/mpr.61

CrossRef Full Text | Google Scholar

60. Mundt J, Marks I, Shear M, Greist J. The Work and social adjustment scale: a simple measure of impairment in functioning. Br J Psychiatry. (2002) 180:461–4. doi: 10.1192/bjp.180.5.461

PubMed Abstract | CrossRef Full Text | Google Scholar

61. Revelle W. Psych: procedures for psychological, psychometric, and personality research. R package version 2.2.5. Evanston, IL: Northwestern University (2022).

Google Scholar

62. IBM Corp. IBM SPSS statistics for windows, version 25.0. Armonk, NY: IBM Corp (2017).

Google Scholar

63. Rizopoulos D. Ltm: an R package for latent variable modelling and item response theory analyses. J Stat Softw. (2006) 17:1–25. doi: 10.18637/jss.v017.i05

CrossRef Full Text | Google Scholar

64. Rosseel Y. Lavaan: an R package for structural equation modeling. J Stat Softw. (2012) 48:1–36. doi: 10.18637/jss.v048.i02

CrossRef Full Text | Google Scholar

65. Robitzsch A. Sirt: supplementary item response theory models. R package version 3.12-66. (2022). Available online at: https://CRAN.R-project.org/package=sirt (accesed December 15, 2022).

Google Scholar

66. Osborne J. Effect sizes and the disattenuation of correlation and regression coefficients: lessons from educational psychology. Pract Assess Res Eval. (2003) 8:1–7. doi: 10.7275/0k9h-tq64

CrossRef Full Text | Google Scholar

67. Timmerman M, Lorenzo-Seva U. Dimensionality assessment of ordered polytomous items with parallel analysis. Psychol Methods. (2011) 16:209–20. doi: 10.1037/a0023353

PubMed Abstract | CrossRef Full Text | Google Scholar

68. Lorenzo-Seva U, Ferrando PJ. A general approach for fitting pure exploratory bifactor models. Multivar Behav Res. (2019) 54:15–30. doi: 10.1080/00273171.2018.1484339

PubMed Abstract | CrossRef Full Text | Google Scholar

69. Ferrando P, Lorenzo-Seva U. Program FACTOR at 10: origins, development and future directions. Psicothema. (2017) 29:236–40. doi: 10.7334/psicothema2016.304

PubMed Abstract | CrossRef Full Text | Google Scholar

70. Sleep C, Lynam D, Widiger T, Crowe M, Miller J. Difficulties with the conceptualization and assessment of criterion A in the DSM-5 alternative model of personality disorder: a reply to Morey (2019). Psychol Assess. (2019) 31:1200–5. doi: 10.1037/pas0000758

PubMed Abstract | CrossRef Full Text | Google Scholar

71. Vall G, Gutiérrez F, Peri J, Gárriz M, Ferraz L, Baillés E, et al. Seven basic dimensions of personality pathology and their clinical consequences: are all personalities equally harmful? Br J Clin Psychol. (2015) 54:450–68. doi: 10.1111/bjc.12091

PubMed Abstract | CrossRef Full Text | Google Scholar

72. Cruitt P, Boudreaux M, King H, Oltmanns J, Oltmanns T. Examining criterion A: DSM-5 level of personality functioning as assessed through life story interviews. Personal Disord. (2019) 10:224–34. doi: 10.1037/per0000321

PubMed Abstract | CrossRef Full Text | Google Scholar

73. Peri J, Muñoz-Champel A, Torrubia R, Gutiérrez F. The general criteria for personality disorders assessed by interview: do they still have a role to play? J Pers Disord. (2019) 33:515–32. doi: 10.1521/pedi_2018_32_356

CrossRef Full Text | Google Scholar

74. Ullrich S, Farrington D, Coid J. Dimensions of DSM-IV personality disorders and life-success. J Pers Disord. (2007) 21:657–63. doi: 10.1521/pedi.2007.21.6.657

PubMed Abstract | CrossRef Full Text | Google Scholar

75. Vall G, Gutiérrez F, Peri J, Gárriz M, Baillés E, Garrido M, et al. Seven dimensions of personality pathology are under sexual selection in modern Spain. Evol Hum Behav. (2016) 37:169–78. doi: 10.1016/j.evolhumbehav.2015.10.004

CrossRef Full Text | Google Scholar

76. Gutiérrez F, Peri J, Baillès E, Sureda B, Gárriz M, Vall G, et al. A double-track pathway to fast strategy in humans and its personality correlates. Front Psychol. (2022) 13:889730. doi: 10.3389/fpsyg.2022.889730

PubMed Abstract | CrossRef Full Text | Google Scholar

77. Međedović J, Petrović B, Želeskov-Đorić J, Savić M. Interpersonal and affective psychopathy traits can enhance human fitness. Evol Psychol Sci. (2017) 3:306–15. doi: 10.1007/s40806-017-0097-5

CrossRef Full Text | Google Scholar

78. Noordhof A, Kamphuis J, Sellbom M, Eigenhuis A, Bagby R. Change in self-reported personality during major depressive disorder treatment: a reanalysis of treatment studies from a demoralization perspective. Pers Disord. (2018) 9:93–100. doi: 10.1037/per0000238

PubMed Abstract | CrossRef Full Text | Google Scholar

79. Roche M. Examining the alternative model for personality disorder in daily life: evidence for incremental validity. Pers Disord. (2018) 9:574–83. doi: 10.1037/per0000295

PubMed Abstract | CrossRef Full Text | Google Scholar

80. Brown T, Sellbom M. Examining the reliability and validity of the ICD-11 personality disorder severity diagnosis. Aust N Z J Psychiatry. (2022). doi: 10.1177/00048674221136457 [Epub ahead of print].

PubMed Abstract | CrossRef Full Text | Google Scholar

81. Morey L. On parsimony in personality disorder classification: rejoinder to Sleep et al. (2019). Psychol Assess. (2019) 31:1206–7. doi: 10.1037/pas0000774

PubMed Abstract | CrossRef Full Text | Google Scholar

82. Gutiérrez F, Peri J, Gárriz M, Vall G, Arqué E, Ruiz L, et al. Integration of the ICD-11 and DSM-5 dimensional systems for personality disorders into a unified taxonomy with non-overlapping traits. Front Psychiatry. (2021) 12:591934. doi: 10.3389/fpsyt.2021.591934

PubMed Abstract | CrossRef Full Text | Google Scholar

83. Hengartner M, van der Linden D, Dunkel C. Establishing the substantive interpretation of the GFP by considering evidence from research on personality disorders and animal personality. Front Psychol. (2017) 8:1771. doi: 10.3389/fpsyg.2017.01771

PubMed Abstract | CrossRef Full Text | Google Scholar

84. Van der Linden D, Dunkel C, Wu P. The applied relevance of the general factor of personality: advancements in the occupational and clinical context. Jpn J Pers. (2022) 30:199–211. doi: 10.2132/personality.30.3.13

PubMed Abstract | CrossRef Full Text | Google Scholar

85. Smith G, Atkinson E, Davis H, Riley E, Oltmanns J. The general factor of psychopathology. Annu Rev Clin Psychol. (2020) 16:75–98. doi: 10.1146/annurev-clinpsy-071119-115848

PubMed Abstract | CrossRef Full Text | Google Scholar

86. Livesley W, Jang K. Differentiating normal, abnormal, and disordered personality. Eur J Pers. (2005) 19:257–68. doi: 10.1002/per.559

CrossRef Full Text | Google Scholar

87. Parker G, Hadzi-Pavlovic D, Both L, Kumar S, Wilhelm K, Olley A. Measuring disordered personality functioning: to love and to work reprised. Acta Psychiatr Scand. (2004) 110:230–9. doi: 10.1111/j.1600-0447.2004.00312.x

PubMed Abstract | CrossRef Full Text | Google Scholar

88. Cloninger CR. A practical way to diagnosis personality disorder: a proposal. J Pers Disord. (2000) 14:99–108. doi: 10.1521/pedi.2000.14.2.99

PubMed Abstract | CrossRef Full Text | Google Scholar

89. Bender D, Morey L, Skodol A. Toward a model for assessing level of personality functioning in DSM-5, part I: a review of theory and methods. J Pers Assess. (2011) 93:332–46. doi: 10.1080/00223891.2011.583808

PubMed Abstract | CrossRef Full Text | Google Scholar

90. Livesley W, Jang K. Toward an empirically based classification of personality disorder. J Pers Disord. (2000) 14:137–51. doi: 10.1521/pedi.2000.14.2.137

PubMed Abstract | CrossRef Full Text | Google Scholar

91. Fonagy P, Luyten P, Allison E. Epistemic petrification and the restoration of epistemic trust: a new conceptualization of borderline personality disorder and its psychosocial treatment. J Pers Disord. (2015) 29:575–609. doi: 10.1521/pedi.2015.29.5.575

PubMed Abstract | CrossRef Full Text | Google Scholar

92. Zimmermann J. Beyond defending or abolishing criterion A: comment on Morey et al. (2022). Personal Disord. (2022) 13:321–4. doi: 10.1037/per0000561

PubMed Abstract | CrossRef Full Text | Google Scholar

93. Bowlby J. Attachment and loss: vol. 1. Attachment. 2nd ed. New York, NY: Basic Books (1982).

Google Scholar

94. MacDonald K. Evolution, the five-factor model, and levels of personality. J Pers. (1995) 63:525–67. doi: 10.1111/j.1467-6494.1995.tb00505.x

CrossRef Full Text | Google Scholar

95. Davis K, Panksepp J. The emotional foundations of personality: a neurobiological and evolutionary approach. London: W. W. Norton & Company (2018).

Google Scholar

96. DeYoung C. Cybernetic big five theory. J Res Pers. (2015) 56:33–58. doi: 10.1016/j.jrp.2014.07.004

CrossRef Full Text | Google Scholar

97. Harkness A, Reynolds S, Lilienfeld SO. A review of systems for psychology and psychiatry: adaptive systems, personality psychopathology five (PSY-5), and the DSM-5. J Pers Assess. (2014) 96:121–39. doi: 10.1080/00223891.2013.823438

PubMed Abstract | CrossRef Full Text | Google Scholar

98. Saulsman L, Page A. The five-factor model and personality disorder empirical literature: a meta-analytic review. Clin Psychol Rev. (2004) 23:1055–85. doi: 10.1016/j.cpr.2002.09.001

PubMed Abstract | CrossRef Full Text | Google Scholar

99. Samuel D, Widiger TA. A meta-analytic review of the relationships between the five-factor model and DSM-IV-TR personality disorders: a facet level analysis. Clin Psychol Rev. (2008) 28:1326–42. doi: 10.1016/j.cpr.2008.07.002

PubMed Abstract | CrossRef Full Text | Google Scholar

100. Lahey B. Public health significance of neuroticism. Am Psychol. (2009) 64:241–56. doi: 10.1037/a0015309

PubMed Abstract | CrossRef Full Text | Google Scholar

101. Widiger T, Oltmanns J. Neuroticism is a fundamental domain of personality with enormous public health implications. World Psychiatry. (2017) 16:144–5. doi: 10.1002/wps.20411

PubMed Abstract | CrossRef Full Text | Google Scholar

102. Linville P. Self-complexity as a cognitive buffer against stress-related illness and depression. J Pers Soc Psychol. (1987) 52:663–76. doi: 10.1037//0022-3514.52.4.663

CrossRef Full Text | Google Scholar

103. Gutiérrez F, Gárriz M, Peri J, Ferraz L, Sol D, Navarro J, et al. Fitness costs and benefits of personality disorder traits. Evol Hum Behav. (2013) 34:41–8. doi: 10.1016/j.evolhumbehav.2012.09.001

CrossRef Full Text | Google Scholar

104. Reynolds J, McCrea S. Environmental constraints on the functionality of inhibitory self-control: sometimes you should eat the donut. Self Identity. (2019) 18:60–86. doi: 10.1080/15298868.2017.1354066

CrossRef Full Text | Google Scholar

105. McCrae R, Löckenhoff C, Costa PT. A step toward DSM-V: cataloguing personality-related problems in living. Eur J Pers. (2005) 19:269–86. doi: 10.1002/per.564

CrossRef Full Text | Google Scholar

106. Mullins-Sweatt S, Widiger T. Personality-related problems in living: an empirical approach. Personal Disord. (2010) 1:230–8. doi: 10.1037/a0018228

PubMed Abstract | CrossRef Full Text | Google Scholar

107. Gutiérrez F, Gárriz M, Peri J, Vall G, Torrubia R. How temperament and character affect our career, relationships, and mental health. Compr Psychiatry. (2016) 70:181–9. doi: 10.1016/j.comppsych.2016.07.014

PubMed Abstract | CrossRef Full Text | Google Scholar

108. Sleep C, Lynam D, Miller J. Personality impairment in the DSM-5 and ICD-11: current standing and limitations. Curr Opin Psychiatry. (2021) 34:39–43. doi: 10.1097/YCO.0000000000000657

PubMed Abstract | CrossRef Full Text | Google Scholar

109. Wessels N, Zimmermann J, Leising D. Toward a shared understanding of important consequences of personality. Rev Gen Psychol. (2016) 20:426–36. doi: 10.1037/gpr0000088

CrossRef Full Text | Google Scholar

110. Ozer D, Benet-Martínez V. Personality and the prediction of consequential outcomes. Annu Rev Psychol. (2006) 57:401–21. doi: 10.1146/annurev.psych.57.102904.190127

PubMed Abstract | CrossRef Full Text | Google Scholar

111. Roberts B, Kuncel N, Shiner R, Caspi A, Goldberg L. The power of personality: the comparative validity of personality traits, socioeconomic status, and cognitive ability for predicting important life outcomes. Perspect Psychol Sci. (2007) 2:313–45. doi: 10.1111/j.1745-6916.2007.00047.x

PubMed Abstract | CrossRef Full Text | Google Scholar

112. Schneider K. Psychopathic personalities. New York, NY: Grune & Stratton (1958).

Google Scholar

113. Biskin R. The lifetime course of borderline personality disorder. Can J Psychiatry. (2015) 60:303–8. doi: 10.1177/070674371506000702

PubMed Abstract | CrossRef Full Text | Google Scholar

114. Leising D, Rogers K, Ostner J. The undisordered personality: normative assumptions underlying personality disorder diagnoses. Rev Gen Psychol. (2009) 13:230–41. doi: 10.1037/a0017139

CrossRef Full Text | Google Scholar

115. Leising D, Scherbaum S, Packmohr P, Zimmermann J. Substance and evaluation in personality disorder diagnoses. J Pers Disord. (2018) 32:766–83. doi: 10.1521/pedi_2017_31_324

CrossRef Full Text | Google Scholar

116. Hertler S. The biologically-based bias of personality disorder diagnosis. Front Psychol. (2016) 7:1293. doi: 10.3389/fpsyg.2016.01293

PubMed Abstract | CrossRef Full Text | Google Scholar

117. Zachar P, Kendler K. Psychiatric disorders: a conceptual taxonomy. Am J Psychiatry. (2007) 164:557–65. doi: 10.1176/ajp.2007.164.4.557

PubMed Abstract | CrossRef Full Text | Google Scholar

118. Wakefield J. The perils of dimensionalization: challenges in distinguishing negative traits from personality disorders. Psychiatr Clin North Am. (2008) 31:379–93. doi: 10.1016/j.psc.2008.03.009

PubMed Abstract | CrossRef Full Text | Google Scholar

119. Penke L, Denissen J, Miller G. The evolutionary genetics of personality. Eur J Pers. (2007) 21:549–87. doi: 10.1002/per.629

CrossRef Full Text | Google Scholar

120. Dingemanse N, Réale D. What is the evidence for natural selection maintaining animal personality variation? In: C Carere, D Maestripieri editors. Animal personalities: behavior, physiology, and evolution. Chicago, IL: University of Chicago Press (2013). p. 201–20.

Google Scholar

121. Del Giudice M. Evolutionary psychopathology: a unified approach. New York, NY: Oxford University Press (2018).

Google Scholar

122. Wolf M, Weissing F. Animal personalities: consequences for ecology and evolution. Trends Ecol Evol. (2012) 27:452–61. doi: 10.1016/j.tree.2012.05.001

PubMed Abstract | CrossRef Full Text | Google Scholar

123. Wright A, Hopwood C, Skodol A, Morey L. Longitudinal validation of general and specific structural features of personality pathology. J Abnorm Psychol. (2016) 125:1120–34. doi: 10.1037/abn0000165

PubMed Abstract | CrossRef Full Text | Google Scholar

124. Podsakoff P, MacKenzie S, Podsakoff N. Sources of method bias in social science research and recommendations on how to control it. Ann Rev Psychol. (2012) 63:539–69. doi: 10.1146/annurev-psych-120710-100452

PubMed Abstract | CrossRef Full Text | Google Scholar

125. Mittlböck M, Schemper M. Explained variation for logistic regression. Stat Med. (1996) 15:1987–97. doi: 10.1002/(SICI)1097-0258(19961015)15:193.0.CO;2-9

CrossRef Full Text | Google Scholar

126. Hellevik O. Linear versus logistic regression when the dependent variable is a dichotomy. Qual Quant. (2009) 43:59–74. doi: 10.1007/s11135-007-9077-3

CrossRef Full Text | Google Scholar

Keywords: personality disorder, personality pathology, severity, ICD-11, PDS-ICD-11

Citation: Gutiérrez F, Aluja A, Rodríguez C, Gárriz M, Peri JM, Gallart S, Calvo N, Ferrer M, Gutiérrez-Zotes A, Soler J and Pascual JC (2023) Severity in the ICD-11 personality disorder model: Evaluation in a Spanish mixed sample. Front. Psychiatry 13:1015489. doi: 10.3389/fpsyt.2022.1015489

Received: 09 August 2022; Accepted: 16 December 2022;
Published: 09 January 2023.

Edited by:

Jared Keeley, Virginia Commonwealth University, United States

Reviewed by:

Mark Waugh, Oak Ridge National Laboratory (DOE), United States
Bo Bach, Psychiatry Region Zealand, Denmark

Copyright © 2023 Gutiérrez, Aluja, Rodríguez, Gárriz, Peri, Gallart, Calvo, Ferrer, Gutiérrez-Zotes, Soler and Pascual. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Fernando Gutiérrez, www.frontiersin.org Zmd1dGlAY2xpbmljLmNhdA==

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.