Thomas Sithara
K. B. Arun1
H. P. Syama
T. R. Reshmitha
P. Nisha
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- 1Agro Processing and Technology Division, National Institute for Interdisciplinary Science and Technology (CSIR), Thiruvananthapuram, India
- 2Academy of Scientific and Innovative Research, New Delhi, India
by Sithara T, Arun KB, Syama HP, Reshmitha TR and Nisha P (2017). Front. Pharmacol. 8:640. doi: 10.3389/fphar.2017.00640
In the published article, there was an error in the text values.
A correction has been made to Results, Detection of Cell Apoptosis Using Flow Cytometry. This sentence previously stated:
“The mean percentage of cells in the early apoptotic population and the late apoptotic population on treatment with 150 µM morin for 48 h was 17.26 ± 0.75 and 9.26 ± 0.40, respectively. It was further increased to 11.56 ± 0.37, 15.83 ± 0.41 and 36.3 ± 1.35, 48.76 ± 0.2 after exposure to 200 and 250 µM morin, respectively, which was significantly different from control cell population (early apoptotic: 1.4 ± 0.2 and late apoptotic: 0.3 ± 0.1). The increment in the late apoptotic population on morin treatment (250 µM) was significantly higher than that of the positive control, camptothecin (early apoptotic: 14.8 ± 0.12 and late apoptotic: 30.2 ± 0.18)”.
The corrected sentence appears below:
“The mean percentage of cells in the early apoptotic population and the late apoptotic population on treatment with 150 µM morin for 48 h was 17.26 ± 0.75 and 15.83 ± 0.41, respectively. It was further changed to 9.26 ± 0.40 and 11.56 ± 0.37 and 36.3 ± 0.42, 48.76 ± 0.20, respectively, after exposure to 200 and 250 µM morin, which was significantly different from control cell population (early apoptotic: 1.4 ± 0.2 and late apoptotic: 0.3 ± 0.1). The increment in the late apoptotic population on morin treatment (200 µM and 250 µM) were significantly higher than that of the positive control, camptothecin (early apoptotic: 30.2 ± 0.3 and late apoptotic: 14.8 ± 0.35).”
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
Publisher’s note
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Keywords: colorectal cancer, reactive oxygen species, apoptosis, Warburg effect, energetic stress
Citation: Sithara T, Arun KB, Syama HP, Reshmitha TR and Nisha P (2025) Corrigendum: Morin inhibits proliferation of SW480 colorectal cancer cells by inducing apoptosis mediated by reactive oxygen species formation and uncoupling of Warburg effect. Front. Pharmacol. 16:1580299. doi: 10.3389/fphar.2025.1580299
Received: 20 February 2025; Accepted: 11 March 2025;
Published: 27 March 2025.
Edited and reviewed by:
Anna Rita Migliaccio, Campus Bio-Medico University, ItalyCopyright © 2025 Sithara, Arun, Syama, Reshmitha and Nisha. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: P. Nisha, YnAubmlzaGFAeWFob28uY29t, cG5pc2hhQG5paXN0LnJlcy5pbg==