CORRECTION article

Front. Pharmacol., 15 August 2024

Sec. Gastrointestinal and Hepatic Pharmacology

Volume 15 - 2024 | https://doi.org/10.3389/fphar.2024.1395496

Corrigendum: YKL-40 promotes chemokine expression following drug-induced liver injury via TF-PAR1 pathway in mice

  • 1. Department of Pharmacy, Shengjing Hospital of China Medical University, Shenyang, China

  • 2. Department of the Second Clinical Pharmacy, School of Pharmacy, China Medical University, Shenyang, China

In the published article, there was an error Figure 3A and its legend as published. The TF was inadvertently misused during the final assembly of Figure 3A. The corrected Figure 3 its caption appears below.

FIGURE 3

In the published article, there was an error Figure 7A and its legend as published. The TF was inadvertently misused during the final assembly of Figure 7A. The corrected Figure 7 its caption appears below.

FIGURE 7

In the published article, there was an error. The anti-TF antibody’s manufacturer and batch number are incorrect.

A correction has been made to 2. Materials and methods, 2.1 Animal experiments, Paragraph Number 1. This sentence previously stated:

“In the experimental groups, mice were immediately injected with recombinant Chi3l1 (500 ng, Sino Biological, 50929-M08H), anti-Chi3l1 antibody (500 ng, Sino Biological, 50929-RP01), or anti-TF antibody (1/1,000, 500 μg, ProteinTech, 17435-1-AP) after receiving ConA (Shan et al., 2018).”

The corrected sentence appears below:

“In the experimental groups, mice were immediately injected with recombinant Chi3l1 (500 ng, Sino Biological, 50929-M08H), anti-Chi3l1 antibody (500 ng, Sino Biological, 50929-RP01), or anti-TF antibody (1/1,000, 500 μg, Bioss, bs-4690R) after receiving ConA (Shan et al., 2018).”

In the published article, there was an error. The anti-TF antibody’s manufacturer and batch number are incorrect.

A correction has been made to 2. Materials and methods, 2.4 Western blot analysis, Paragraph Number 1. This sentence previously stated:

“The membrane was then blocked with a rapid blocking solution and incubated with the specific primary antibodies against TF (ProteinTech, 17435-1-AP, 1/3,000), PAR1 (Solarbio, K009690P, 1/1,500), CCL2/MCP-1 (ProteinTech, 25542-1-AP, 1/2,000), and CXCL10/IP-10 (ProteinTech, 10937-1-AP, 1/500).”

The corrected sentence appears below:

“The membrane was then blocked with a rapid blocking solution and incubated with the specific primary antibodies against TF (Bioss, bs-4690R, 1/1,000), PAR1 (Solarbio, K009690P, 1/1,500), CCL2/MCP-1 (ProteinTech, 25542-1-AP, 1/2,000), and CXCL10/IP-10 (ProteinTech, 10937-1-AP, 1/500).”

The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Statements

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

YKL-40, TF-PAR1 pathway, inflammation, liver injury, CCL2, IP-10

Citation

Jing-Lun Z, Shuang C, Li-Mei Z and Xiao-Dong L (2024) Corrigendum: YKL-40 promotes chemokine expression following drug-induced liver injury via TF-PAR1 pathway in mice. Front. Pharmacol. 15:1395496. doi: 10.3389/fphar.2024.1395496

Received

04 March 2024

Accepted

05 August 2024

Published

15 August 2024

Volume

15 - 2024

Edited by

Patricia Moriel, State University of Campinas, Brazil

Reviewed by

Ning Zhang, Fudan University, China

Updates

Copyright

*Correspondence: Liu Xiao-Dong,

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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