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CORRECTION article
Front. Pharmacol. , 13 May 2024
Sec. Experimental Pharmacology and Drug Discovery
Volume 15 - 2024 | https://doi.org/10.3389/fphar.2024.1394151
This article is a correction to:
BM-MSCs alleviate diabetic nephropathy in male rats by regulating ER stress, oxidative stress, inflammation, and apoptotic pathways
A Corrigendum on
BM-MSCs alleviate diabetic nephropathy in male rats by regulating ER stress, oxidative stress, inflammation, and apoptotic pathways
by Khamis T, Abdelkhalek A, Abdellatif H, Dwidar N, Said A, Ahmed R, Wagdy K, Elgarhy R, Eltahan R, Mohamed H, Said Amer E, Hanna M, Ragab T, Kishk A, Wael J, Sarhan E, Saweres L, Reda M, Elkomy S, Mohamed A, Samy A, Khafaga A, Shaker Y, Yehia H, Alanazi A, Alassiri M, Tîrziu E, Bucur IM and Arisha AH (2023). Front. Pharmacol. 14:1265230. doi: 10.3389/fphar.2023.1265230
In the published article, there was an error in Figure 1 as published. In the published version of the manuscript, there was an unintentional duplication of Figure 1E, which resulted in Figure 1D being covered. This duplication occurred due to a technical error during the formatting process. The corrected Figure 1 and its caption appear below.
Figure 1. Identification and homing of BM-MSCs in diabetic rat renal tissue (A–J). (A) BM-MSC isolation on the third day of culture; (B) BM-MSC isolation on the seventh day of culture (C–I). Flow cytometric detection of BM-MSCs: (C) BM-MSC cell populations were +ve for CD105; (D) BM-MSCS cell populations were +ve for CD90; (E) BM-MSC cell populations were +ve for CD73; (F) BM-MSC cell populations were +ve for CD44; (G) BM-MSC cell populations were −ve for CD45; and (H) BM-MSC cell populations were −ve for CD34. (I) BM-MSC cell populations were −ve for CD14, and (J) PKH26 was used to identify BM-MSC homing in renal tissue.
In the published article, the Supplementary Table S2 was mistakenly not included in the publication. The missing material contains the raw data of the article. This is now published alongside the original article.
The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.
Keywords: diabetic nephropathy, mesenchymal stem cells, bone marrow-derived mesenchymal stem cells, diabetes, apoptosis, ER stress, inflammation, intermediate filament proteins
Citation: Khamis T, Abdelkhalek A, Abdellatif H, Dwidar N, Said A, Ahmed R, Wagdy K, Elgarhy R, Eltahan R, Mohamed H, Said Amer E, Hanna M, Ragab T, Kishk A, Wael J, Sarhan E, Saweres L, Reda M, Elkomy S, Mohamed A, Samy A, Khafaga A, Shaker Y, Yehia H, Alanazi A, Alassiri M, Tîrziu E, Maria Bucur I and Arisha AH (2024) Corrigendum: BM-MSCs alleviate diabetic nephropathy in male rats by regulating ER stress, oxidative stress, inflammation, and apoptotic pathways. Front. Pharmacol. 15:1394151. doi: 10.3389/fphar.2024.1394151
Received: 01 March 2024; Accepted: 30 April 2024;
Published: 13 May 2024.
Edited and reviewed by:
Md Abdul Hye Khan, University of Missouri, United StatesCopyright © 2024 Khamis, Abdelkhalek, Abdellatif, Dwidar, Said, Ahmed, Wagdy, Elgarhy, Eltahan, Mohamed, Said Amer, Hanna, Ragab, Kishk, Wael, Sarhan, Saweres, Reda, Elkomy, Mohamed, Samy, Khafaga, Shaker, Yehia, Alanazi, Alassiri, Tîrziu, Maria Bucur and Arisha. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Tarek Khamis, dC5raGFtaXNAdmV0Lnp1LmVkdS5lZw==; Iulia Maria Bucur, aXVsaWEuYnVjdXJAdXNhYi10bS5ybw==; Ahmed H. Arisha, dmV0YWhtZWRoYW1lZEB6dS5lZHUuZWc=
Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.
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