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CORRECTION article

Front. Pharmacol., 06 January 2022
Sec. Ethnopharmacology

Corrigendum: Sanguisorba officinalis L. Suppresses Triple-Negative Breast Cancer Metastasis by Inhibiting Late-Phase Autophagy via Hif-1α/Caveolin-1 Signaling

  • 1The Research Center for Integrative Medicine, School of Basic Medical Sciences, Guangzhou University of Chinese Medicine, Guangzhou, China
  • 2Integrative Research Laboratory of Breast Cancer, The Second Clinical College, Guangzhou University of Chinese Medicine, Guangzhou, China
  • 3Guangdong Provincial Key Laboratory of Clinical Research on Traditional Chinese Medicine Syndrome, Guangdong Provincial Academy of Chinese Medical Sciences, Guangdong Provincial Hospital of Chinese Medicine, Guangzhou, China

A corrigendum on
Sanguisorba officinalis L. Suppresses Triple-Negative Breast Cancer Metastasis by Inhibiting Late-Phase Autophagy via Hif-1α/Caveolin-1 Signaling

by Wang, N., Muhetaer, G., Zhang, X., Yang, B., Wang, C., Zhang, Y., Wang, X., Zhang, J., Wang, S., Zheng, Y., Zhang, F., and Wang, Z. (2020). Front. Pharmacol. 10:591400. doi:10.3389/fphar.2020.591400

In the original article, there were mistakes in Figures 2B, 6E as published. In Figure 2B, the invasion image of 100 μg/ml SA in MDA-MB-231 panel was mistakenly uploaded. In Figure 6E, certain invasion images were unintentionally misused during picture assembly and image processing. The corrected Figures 2B, 6E appear below.

FIGURE 2
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FIGURE 2. SA suppresses migration and invasion in metastatic cells. (B) Representative images of decreased cell number in transwell chambers with or without SA (0–200 μg/ml).

FIGURE 6
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FIGURE 6. SA suppresses autophagy-mediated metastatic processes during starvation or hypoxia. (E) wound healing assays as well as transwell invasion assays reflected the influences of SA on cell growth or invasiveness under starvation (EBSS) or hypoxia (cobalt chloride).

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s Note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: breast-cancer metastasis, Sanguisorba officinalis L., late-phase autophagic regulation, Cav-1, HIF-1α

Citation: Wang N, Muhetaer G, Zhang X, Yang B, Wang C, Zhang Y, Wang X, Zhang J, Wang S, Zheng Y, Zhang F and Wang Z (2022) Corrigendum: Sanguisorba officinalis L. Suppresses Triple-Negative Breast Cancer Metastasis by Inhibiting Late-Phase Autophagy via Hif-1α/Caveolin-1 Signaling. Front. Pharmacol. 12:810621. doi: 10.3389/fphar.2021.810621

Received: 07 November 2021; Accepted: 29 November 2021;
Published: 06 January 2022.

Edited and Reviewed by:

Rong-Rong He, Jinan University, China

Copyright © 2022 Wang, Muhetaer, Zhang, Yang, Wang, Zhang, Wang, Zhang, Wang, Zheng, Zhang and Wang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Zhiyu Wang, wangzhiyu976@126.com; Fengxue Zhang, zhangfengxue@gzucm.edu.cn

These authors have contributed equally to this work

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.