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CORRECTION article

Front. Oncol., 30 September 2024
Sec. Pharmacology of Anti-Cancer Drugs

Corrigendum: 5-Demethylnobiletin mediates cell cycle arrest and apoptosis via the ERK1/2/AKT/STAT3 signaling pathways in glioblastoma cells

Xuehua ZhangXuehua Zhang1Leilei ZhaoLeilei Zhao1Jinlong XiaoJinlong Xiao1Yudi WangYudi Wang1Yunmeng LiYunmeng Li1Chaoqun ZhuChaoqun Zhu2He ZhangHe Zhang3Yurui ZhangYurui Zhang1Xiao Zhu*Xiao Zhu2*Yucui Dong*Yucui Dong1*
  • 1Department of Immunology, Binzhou Medical University, Yantai, China
  • 2School of Computer and Control Engineering, Yantai University, Yantai, China
  • 3Department of Immunology, Qiqihar Medical University, Qiqihar, China

A Corrigendum on
5-Demethylnobiletin mediates cell cycle arrest and apoptosis via the ERK1/2/AKT/STAT3 signaling pathways in glioblastoma cells

By Zhang X, Zhao L, Xiao J, Wang Y, Li Y, Zhu C, Zhang H, Zhang Y, Zhu X and Dong Y (2023). Front. Oncol. 13:1143664. doi: 10.3389/fonc.2023.1143664

In the published article, there was an error in Figure 1D as published. We regret in Figure 1D the pictures of A172 cells (25 μM) and U251 cells (50 μM) partially overlapped, when we sorted out the raw data. The picture of U251 cells (50 μM) is wrong. In the correct figure below, the picture representing U251 (50 μM) group in Figure 1D has been replaced. We used the data from the original images for statistical analysis. The corrected Figure 1 and its caption appear below.

Figure 1
www.frontiersin.org

Figure 1. Inhibitory effects of 5-DMN on the viability, migration and invasion of GBM cells. (A) Cell viability was determined using the MTT assay after cultured U87-MG, A172, and U251 cells were incubated with various doses of 5-DMN for 48 h. (B) The merged image of live cells (green) and dead cells (red) in GBM cells treated with different concentrations of 5-DMN. Scale bar = 100 μm. (C) Wound healing assays were used to determine the migration ability of U87-MG, A172, and U251 cells after 48 h of incubation with 5-DMN (0, 25, 50 μM). (D) The invasion ability of GBM cells treated with or without 5-DMN (25, 50 μM) for 48 h was detected by transwell assays. Values are the means ± SD of three independent experiments. *p<0.05, **p<0.01, ***p<0.001 vs. cells in the untreated control group. ns, no significance.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: glioblastoma, 5-Demethylnobiletin, cell cycle arrest, cell apoptosis, ERK1/2, AKT, STAT3

Citation: Zhang X, Zhao L, Xiao J, Wang Y, Li Y, Zhu C, Zhang H, Zhang Y, Zhu X and Dong Y (2024) Corrigendum: 5-Demethylnobiletin mediates cell cycle arrest and apoptosis via the ERK1/2/AKT/STAT3 signaling pathways in glioblastoma cells. Front. Oncol. 14:1494738. doi: 10.3389/fonc.2024.1494738

Received: 11 September 2024; Accepted: 16 September 2024;
Published: 30 September 2024.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2024 Zhang, Zhao, Xiao, Wang, Li, Zhu, Zhang, Zhang, Zhu and Dong. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Yucui Dong, dongyucui521@yeah.net; Xiao Zhu, xzhu@ytu.edu.cn

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.