Combined Scutellarin and C18H17NO6 Imperils the Survival of Glioma: Partly Associated With the Repression of PSEN1/PI3K-AKT Signaling Axis
- 1Institute of Neurological Disease, Department of Anesthesiology, Translational Neuroscience Center, West China Hospital, Sichuan University, Chengdu, China
- 2Institute of Neuroscience, Laboratory Zoology Department, Kunming Medical University, Kunming, China
- 3School of Public Health, Kunming Medical University, Kunming, China
by He X-Y, Xu Y, Xia Q-J, Zhao X-M, Li S, He X-Q, Wang R-R, and Wang T-H (2021) Front. Oncol. 11:663262. doi: 10.3389/fonc.2021.663262
The shared equal contribution made by authors Xiu-Ying He, and Yang Xu was not depicted in the article. These authors have contributed equally to this work.
The authors apologize for these errors and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: C18H17NO6, PSEN1, glioma, PI3K-Akt signaling, scutellarin
Citation: He X-Y, Xu Y, Xia Q-J, Zhao X-M, Li S, He X-Q, Wang R-R and Wang T-H (2022) Corrigendum: Combined Scutellarin and C18H17NO6 Imperils the Survival of Glioma: Partly Associated With the Repression of PSEN1/PI3K-AKT Signaling Axis. Front. Oncol. 12:872110. doi: 10.3389/fonc.2022.872110
Received: 09 February 2022; Accepted: 10 February 2022;
Published: 07 March 2022.
Approved by:
Frontiers Editorial Office, Frontiers Media SA, SwitzerlandCopyright © 2022 He, Xu, Xia, Zhao, Li, He, Wang and Wang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Ting-Hua Wang, tinghua_neuron@263.net; Ru-Rong Wang, wangrurong@scu.edu.cn; Xiao-Qiong He, hexqcn@aliyun.com
†These authors have contributed equally to this work