CORRECTION article

Front. Oncol., 14 October 2022

Sec. Molecular and Cellular Oncology

Volume 12 - 2022 | https://doi.org/10.3389/fonc.2022.1041732

Corrigendum: LncRNA FOXP4-AS1 promotes the progression of esophageal squamous cell carcinoma by interacting with MLL2/H3K4me3 to upregulate FOXP4

  • 1. Laboratory of Pathology, Hebei Cancer Institute, The Fourth Hospital of Hebei Medical University, Shijiazhuang, China

  • 2. Experimental Center, Hebei University of Chinese Medicine, Shijiazhuang, China

In the original article, there was a mistake in Figure 1 as published. In Figure 1B, “GSE151633” should be “GSE161533”. The corrected Figure 1 appears below.

Figure 1

In addition, there was a mistake in Figure 2 as published. In Figure 2F, the original image of the clone formation experiment has been reworked. The corrected Figure 2 appears below.

Figure 2

In addition, there was a mistake in Figure 3 as published. In Figure 3F, “Relative expression of FOXP4-AS1” has been changed to “Relative Expression of FOXP4-AS1”. #In Figure 3A, the title of the Y axis has changed from "Expression of FOXP4 (2-△△CT)" to "Relative Expression of FOXP4". The corrected Figure 3 appears below.

Figure 3

In addition, there was a mistake in Figure 4 as published. In Figure 4D, “Relative expression of FOXP4-AS1” has been changed to “Relative Expression of FOXP4-AS1”. In Figure 4C, the title of the Y axis has changed from “Expression of MLL2 (2-△△CT)" to “Relative Expression of MLL2”. The corrected Figure 4 appears below.

Figure 4

In addition, there was a mistake in Figure 5 as published. In Figure 5I, the original image of the clone formation experiment has been reworked. The corrected Figure 5 appears below.

Figure 5

In addition, there was a mistake in Figure 6 as published. In Figure 6I, the luciferase reporter gene experiment was reworked. The corrected Figure 6 appears below.

Figure 6

In addition, there was a mistake in Figure 7 as published. In Figure 7, the overall experiment was divided into four groups. The overexpression of FOXP4-AS1 promoted metastasis and invasion of esophageal squamous carcinoma cells, however transfection of pcDNA3.1-FOXP4-AS1 and sh-FOXP4 attenuated the ability of transfection of pcDNA3.1-FOXP4-AS1 alone to metastasize and invade esophageal squamous carcinoma cells. The corrected Figure 7 appears below.

Figure 7

In addition, there was a mistake in Figure S1 as published. In Figure S1I, “groups” should be “We divided the group into four groups”. The updated file may be viewed via the original article.

The authors apologize for these errors and state that they do not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Summary

Keywords

ESCC, FOXP4-AS1, long noncoding RNA, FOXP4, MLL2

Citation

Niu Y, Wang G, Li Y, Guo W, Guo Y and Dong Z (2022) Corrigendum: LncRNA FOXP4-AS1 promotes the progression of esophageal squamous cell carcinoma by interacting with MLL2/H3K4me3 to upregulate FOXP4. Front. Oncol. 12:1041732. doi: 10.3389/fonc.2022.1041732

Received

11 September 2022

Accepted

13 September 2022

Published

14 October 2022

Volume

12 - 2022

Edited and reviewed by

Emanuela Grassilli, University of Milano Bicocca, Italy

Updates

Copyright

*Correspondence: Zhiming Dong,

This article was submitted to Molecular and Cellular Oncology, a section of the journal Frontiers in Oncology

Disclaimer

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.

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