Effects of a 5-HT1B Receptor Agonist on Locomotion and Reinstatement of Cocaine-Conditioned Place Preference after Abstinence from Repeated Injections in Mice
by Der-Ghazarian, T. S., Call, T., Scott, S. N., Dai, K., Brunwasser, S. J., Noudali, S. N., et al. (2017). Front. Syst. Neurosci. 11:73. doi: 10.3389/fnsys.2017.00073
Discrepancy in Animal Protocol
The authors have identified a discrepancy between the 15 mg/kg, IP cocaine priming dose used in this study, and the IACUC approved protocol dose of 10 mg/kg, IP. The dose of 15 mg/kg, IP cocaine used for the repeated drug injections in this study is correct and approved in the same IACUC protocol and caused no pain or discomfort to mice.
Conflict of Interest Statement
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
The handling Editor declared a shared affiliation, though no other collaboration, with the authors and states that the process nevertheless met the standards of a fair and objective review.
Keywords: serotonin, CP94253, sensitization, withdrawal, addiction, place conditioning
Citation: Der-Ghazarian TS, Call T, Scott SN, Dai K, Brunwasser SJ, Noudali SN, Pentkowski NS and Neisewander JL (2018) Addendum: Effects of a 5-HT1B Receptor Agonist on Locomotion and Reinstatement of Cocaine-Conditioned Place Preference after Abstinence from Repeated Injections in Mice. Front. Syst. Neurosci. 12:48. doi: 10.3389/fnsys.2018.00048
Received: 21 August 2018; Accepted: 24 September 2018;
Published: 07 November 2018.
Edited and reviewed by: Irina T. Sinakevitch, Arizona State University, United States
Copyright © 2018 Der-Ghazarian, Call, Scott, Dai, Brunwasser, Noudali, Pentkowski and Neisewander. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Janet L. Neisewander, amFuZXQubmVpc2V3YW5kZXJAYXN1LmVkdQ==
†Present Address: Kael Dai, Allen Institute for Brain Science Seattle, WA, United States
Samuel J. Brunwasser, Medical School, Washington University in St. Louis, St. Louis, MO, United States
Nathan S. Pentkowski, Department of Psychology, University of New Mexico, Albuquerque, NM, United States