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CORRECTION article

Front. Mol. Neurosci., 07 December 2023
Sec. Neuroplasticity and Development
Volume 16 - 2023 | https://doi.org/10.3389/fnmol.2023.1344096

Corrigendum: Role of α2δ-3 in regulating calcium channel localization at presynaptic active zones during homeostatic plasticity

This article is a correction to:

  1. Role of α2δ-3 in regulating calcium channel localization at presynaptic active zones during homeostatic plasticity

    1. Read original article
\r\nYanfeng Zhang,&#x;Yanfeng Zhang1,2†Ting Wang&#x;Ting Wang2†Yimei CaiYimei Cai2Tao CuiTao Cui2Michelle KuahMichelle Kuah2Stefano Vicini,Stefano Vicini2,3Tingting Wang, Tingting Wang2,3*
  • 1Department of Pediatric Neurology, First Hospital of Jilin University, Changchun, Jilin, China
  • 2Department of Pharmacology and Physiology, Georgetown University Medical Center, Washington, DC, United States
  • 3Interdisciplinary Program in Neuroscience, Georgetown University Medical Center, Washington, DC, United States

A corrigendum on
Role of α2δ-3 in regulating calcium channel localization at presynaptic active zones during homeostatic plasticity

by Zhang, Y., Wang, T., Cai, Y., Cui, T., Kuah, M., Vicini, S., and Wang, T. (2023). Front. Mol. Neurosci. 16:1253669. doi: 10.3389/fnmol.2023.1253669

In the published article, there was an error in the affiliations. Instead of “1 Department of Pharmacology and Physiology, Georgetown University Medical Center, Washington, DC, United States; 2 Interdisciplinary Program in Neuroscience, Georgetown University Medical Center, Washington, DC, United States; PRESENT ADDRESS Yanfeng Zhang, Department of Pediatric Neurology, First Hospital of Jilin University, Changchun, Jilin, China” it should be “1 Department of Pediatric Neurology, First Hospital of Jilin University, Changchun, Jilin, China; 2 Department of Pharmacology and Physiology, Georgetown University Medical Center, Washington, DC, United States; 3 Interdisciplinary Program in Neuroscience, Georgetown University Medical Center, Washington, DC, United States”.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher's note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: α2δ-3, voltage gated calcium channel, presynaptic homeostatic plasticity, neurotransmitter release, trafficking, autism, epilepsy, gabapentin

Citation: Zhang Y, Wang T, Cai Y, Cui T, Kuah M, Vicini S and Wang T (2023) Corrigendum: Role of α2δ-3 in regulating calcium channel localization at presynaptic active zones during homeostatic plasticity. Front. Mol. Neurosci. 16:1344096. doi: 10.3389/fnmol.2023.1344096

Received: 25 November 2023; Accepted: 27 November 2023;
Published: 07 December 2023.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2023 Zhang, Wang, Cai, Cui, Kuah, Vicini and Wang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Tingting Wang, dHc2NTImI3gwMDA0MDtnZW9yZ2V0b3duLmVkdQ==

†These authors have contributed equally to this work

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.