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CORRECTION article

Front. Aging Neurosci., 01 March 2016
Sec. Cellular and Molecular Mechanisms of Brain-aging
Volume 8 - 2016 | https://doi.org/10.3389/fnagi.2016.00037

Corrigendum: PGC-1α silencing compounds the perturbation of mitochondrial function caused by mutant SOD1 in skeletal muscle of ALS mouse model

This article is a correction to:

  1. PGC-1 silencing compounds the perturbation of mitochondrial function caused by mutant SOD1 in skeletal muscle of ALS mouse model

    1. Read original article
\r\nYan QiYan Qi1Xiang YinXiang Yin1Shuyu WangShuyu Wang1Hongquan JiangHongquan Jiang1Xudong WangXudong Wang1Ming RenMing Ren2Honglin Feng*Honglin Feng1*
  • 1Department of Neurology, The First Clinical College of Harbin Medical University, Harbin, China
  • 2Department of Neurology, The Affiliated Hospital of Weifang Medical University, Weifang, China

A corrigendum on
PGC-1α silencing compounds the perturbation of mitochondrial function caused by mutant SOD1 in skeletal muscle of ALS mouse model

by Qi, Y., Yin, X., Wang, S., Jiang, H., Wang, X., Ren, M., et al. (2015). Front. Aging Neurosci. 7:204. doi: 10.3389/fnagi.2015.00204

Due to the author's misunderstanding of the Frontiers platform and the criteria for author contributions, Xiang-ping Su and Shi Lei were mistakenly included as authors on the Original Research article “PGC-1α silencing compounds the perturbation of mitochondrial function caused by mutant SOD1 in skeletal muscle of ALS mouse model.”

We, the authors, received technical assistance from Xiang-ping Su and Shi Lei during the process of submission, review and production, and the general use of the Frontiers system, but Xiang-ping Su and Shi Lei had no contribution to the research submitted. As such they should not be considered as authors on this article. Both Xiang-ping Su and Shi Lei agree to the removal.

The authors apologize for this error and misunderstanding.

In addition, due to the author's oversight, some minor typographical errors were included in the Materials and Methods section:

In sub-section SOD1-G93A and C57BL/6 Mice, the body weight of 4-week-old SOD1(G93A) mice was reported as “about 75 g.” The correct body weight was about 15 g.

In sub-section Quantitative Real-Time Polymerase Chain Reaction, the reverse transcription was reported as performed “using 1 g total RNA.” The correct unit was 1 μg total RNA.

In sub-section Immunoprecipitation, the protein incubation was reported as “750 μg.” The correct total protein incubated was 75 μg.

The authors apologize for these errors and any confusion caused.

These errors do not change the scientific conclusions of the article in any way.

Author Contributions

YQ: experiments design and participation, materials purchase, data collection and analysis, paper writing; YX: experiments design and participation, data collection and analysis, paper writing; SW: experiments design and participation, animal raising, specimens collection; HJ: experiments design and participation, materials purchase, technical support; XW: experiments design and participation, data collection and analysis, paper writing; MR: experimental instruction and participation, paper writing; FF: experiments design, instruction and participation, paper writing. All authors meet all four of the below criteria: (1) Substantial contributions to the conception or design of the work; or the acquisition, analysis, or interpretation of data for the work; (2) Drafting the work or revising it critically for important intellectual content; (3) Final approval of the version to be published; (4) Agreement to be accountable for all aspects of the work in ensuring that questions related to the accuracy or integrity of any part of the work are appropriately investigated and resolved.

Funding

This research was supported in part by the grants from the Natural Science Foundation of China (No. 81171186).

Conflict of Interest Statement

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

Keywords: ALS, SOD1(G93A), PGC-1α, energy metabolism, inflammation, fibrosis

Citation: Qi Y, Yin X, Wang S, Jiang H, Wang X, Ren M and Feng H (2016) Corrigendum: PGC-1α silencing compounds the perturbation of mitochondrial function caused by mutant SOD1 in skeletal muscle of ALS mouse model. Front. Aging Neurosci. 8:37. doi: 10.3389/fnagi.2016.00037

Received: 29 January 2016; Accepted: 12 February 2016;
Published: 01 March 2016.

Edited and reviewed by: George E. Barreto, Pontificia Universidad Javeriana, Colombia

Copyright © 2016 Qi, Yin, Wang, Jiang, Wang, Ren and Feng. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Honglin Feng, fenghonglin321@sina.com

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