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CORRECTION article

Front. Mol. Biosci., 17 October 2023
Sec. Protein Biochemistry for Basic and Applied Sciences
Volume 10 - 2023 | https://doi.org/10.3389/fmolb.2023.1308875

Corrigendum: 6-Shogaol inhibits oxidative stress-induced rat vascular smooth muscle cell apoptosis by regulating OXR1-p53 axis

This article is a correction to:

  1. 6-Shogaol Inhibits Oxidative Stress-Induced Rat Vascular Smooth Muscle Cell Apoptosis by Regulating OXR1-p53 Axis

    1. Read original article
Jing Liu,, &#x;Jing Liu1,2,3*†Bin Li,&#x;Bin Li1,3†Wenlian LiWenlian Li1Taowen Pan,Taowen Pan1,3Yunpeng Diao,Yunpeng Diao1,3Fangjun Wang Fangjun Wang2*
  • 1College of Pharmacy, College of Integrative Medicine, Dalian Medical University, Dalian, China
  • 2Key Laboratory of Separation Sciences for Analytical Chemistry, Dalian Institute of Chemical Physics, Chinese Academy of Sciences, Dalian, China
  • 3Dalian Anti-Infective Traditional Chinese Medicine Development Engineering Technology Research Center, Dalian, China

A Corrigendum on
6-Shogaol inhibits oxidative stress-induced rat vascular smooth muscle cell apoptosis by regulating OXR1-p53 axis

by Liu J, Li B, Li W, Pan T, Diao Y and Wang F (2022). Front. Mol. Biosci. 9:808162. doi: 10.3389/fmolb.2022.808162

In the published article, there was an error in Figure 6 as published. The protein bands of reference GAPDH was used incorrectly in Figure 6A. The corrected Figure 6 and its caption “FIGURE 6. The regulation mechanism of p53 by OXR1. (A) The OXR1 can regulate the protein expression of p53. (B) A volcano plot showed that 275 proteins changed significantly (FDR < 0.05, S0 of 0.5), among which SKP1 was observed to change most notably with a 12.9-fold upregulation. (C) The Gene Ontology- (GO-) based pathways enrichment of the altered proteins after OXR1 depletion” appear below.

FIGURE 6
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FIGURE 6. The regulation mechanism of p53 by OXR1. (A) The OXR1 can regulate the protein expression of p53. (B) A volcano plot showed that 275 proteins changed significantly (FDR < 0.05, S0 of 0.5), among which SKP1 was observed to change most notably with a 12.9-fold upregulation. (C) The Gene Ontology- (GO-) based pathways enrichment of the altered proteins after OXR1 depletion.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: vascular smooth muscle cell, OXR1, p53, oxidative stress, apoptosis, 6-shogaol

Citation: Liu J, Li B, Li W, Pan T, Diao Y and Wang F (2023) Corrigendum: 6-Shogaol inhibits oxidative stress-induced rat vascular smooth muscle cell apoptosis by regulating OXR1-p53 axis. Front. Mol. Biosci. 10:1308875. doi: 10.3389/fmolb.2023.1308875

Received: 07 October 2023; Accepted: 10 October 2023;
Published: 17 October 2023.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2023 Liu, Li, Li, Pan, Diao and Wang. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Jing Liu, liujing8166@163.com; Fangjun Wang, wangfj@dicp.ac.cn

†These authors have contributed equally to this work

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.