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CORRECTION article

Front. Mol. Biosci., 03 November 2022
Sec. Molecular Diagnostics and Therapeutics

Corrigendum: Transcription factors BARX1 and DLX4 contribute to progression of clear cell renal cell carcinoma via promoting proliferation and epithelial–mesenchymal transition

Guoliang Sun,,&#x;Guoliang Sun1,2,3Yue Ge,&#x;Yue Ge1,2Yangjun Zhang,Yangjun Zhang1,2Libin YanLibin Yan3Xiaoliang Wu,Xiaoliang Wu1,2Wei Ouyang,Wei Ouyang1,2Zhize WangZhize Wang3Beichen DingBeichen Ding4Yucong ZhangYucong Zhang5Gongwei Long,Gongwei Long1,2Man Liu,Man Liu1,2Runlin ShiRunlin Shi1Hui Zhou,
Hui Zhou1,2*Zhiqiang Chen,Zhiqiang Chen1,2Zhangqun Ye,Zhangqun Ye1,2
  • 1Department of Urology, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
  • 2Hubei Institute of Urology, Wuhan, China
  • 3Department of Urology, The First Affiliated Hospital, College of Medicine, Zhejiang University, Hangzhou, China
  • 4Department of Urology, First Affiliated Hospital of Harbin Medical University, Harbin, China
  • 5Department of Geriatric, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China

A Corrigendum on
Transcription factors BARX1 and DLX4 contribute to progression of clear cell renal cell carcinoma via promoting proliferation and epithelial–mesenchymal transition

by Sun G, Ge Y, Zhang Y, Yan L, Wu X, Ouyang W, Wang Z, Ding B, Zhang Y, Long G, Liu M, Shi R, Zhou H, Chen Z and Ye Z (2021). Front. Mol. Biosci. 8:626328. doi: 10.3389/fmolb.2021.626328

In the published article, there was an error in Figure 6 as published. The images for “BARX1” in Figure 6C and “sh-BARX1-1” in Figure 6E were misplaced during the upload process. The corrected Figure 6 appears below.

FIGURE 6
www.frontiersin.org

FIGURE 6. BARX1 promotes cell proliferation and migration of ccRCC. (A) The efficiency of RCC cell lines stably overexpressing or silencing BARX1 was validated by RT-PCR. (B–D) Colony formation assays, 5-ethynyl-20-deoxyuridine (EdU) assays, and CCK-8 assays were performed in ccRCC cell lines. (E) Transwell migration assay was applied in ccRCC cell lines. (F) The knockdown of BARX1 downregulates the expression of proliferation and EMT-related proteins.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: transcription factor, BARX1, DLX4, biomarker, clear cell renal cell carcinoma

Citation: Sun G, Ge Y, Zhang Y, Yan L, Wu X, Ouyang W, Wang Z, Ding B, Zhang Y, Long G, Liu M, Shi R, Zhou H, Chen Z and Ye Z (2022) Corrigendum: Transcription factors BARX1 and DLX4 contribute to progression of clear cell renal cell carcinoma via promoting proliferation and epithelial–mesenchymal transition. Front. Mol. Biosci. 9:1026319. doi: 10.3389/fmolb.2022.1026319

Received: 23 August 2022; Accepted: 18 October 2022;
Published: 03 November 2022.

Edited and reviewed by:

Sergio Akira Uyemura, University of São Paulo, Brazil

Copyright © 2022 Sun, Ge, Zhang, Yan, Wu, Ouyang, Wang, Ding, Zhang, Long, Liu, Shi, Zhou, Chen and Ye. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Hui Zhou, emhibXh4ZkAxNjMuY29t

These authors have contributed equally to this work

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.