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CORRECTION article

Front. Immunol.
Sec. Primary Immunodeficiencies
Volume 15 - 2024 | doi: 10.3389/fimmu.2024.1467807

Corrigendum: Abnormalities of thymic stroma may contribute to immune dysregulation in murine models of leaky severe combined immunodeficiency

Provisionally accepted
Francesca Rucci Francesca Rucci 1Pietro L. Poliani Pietro L. Poliani 2Stefano Caraffi Stefano Caraffi 1Tiziana Paganini Tiziana Paganini 3Elena Fontana Elena Fontana 2Silvia C. Giliani Silvia C. Giliani 3Frederick Alt Frederick Alt 4Luigi D. Notarangelo Luigi D. Notarangelo 5*
  • 1 Division of Immunology and The Manton Center for Orphan Disease Research, Children's Hospital Boston, Boston, United States
  • 2 Department of Pathology, University of Brescia, Brescia, Italy
  • 3 "Angelo Nocivelli" Institute for Molecular Medicine and Department of Pediatrics, University of Brescia, Brescia, Italy
  • 4 Howard Hughes Medical Institute, Children's Hospital, Immune Disease Institute and Harvard Medical School, Boston, United States
  • 5 Division of Immunology, Children's Hospital Boston, Laboratory of Clinical Immunology and Microbiology, National Institute of Allergy and Infectious Diseases (NIH), Bethesda, MA, United States

This article is a correction to:

  1. Abnormalities of Thymic Stroma may Contribute to Immune Dysregulation in Murine Models of Leaky Severe Combined Immunodeficiency

    1. Read original article

The final, formatted version of the article will be published soon.

    Keywords: Severe Combined Immunodeficiency, recombinationactivating gene 1, DNA Ligase 4, thymic epithelial cells, Thymus, Dendritic Cells, AIRE, regulatory T cells

    Received: 20 Jul 2024; Accepted: 09 Aug 2024.

    Copyright: © 2024 Rucci, Poliani, Caraffi, Paganini, Fontana, Giliani, Alt and Notarangelo. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) or licensor are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

    * Correspondence: Luigi D. Notarangelo, Division of Immunology, Children's Hospital Boston, Laboratory of Clinical Immunology and Microbiology, National Institute of Allergy and Infectious Diseases (NIH), Bethesda, 02115, MA, United States

    Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.