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Jia Yang1†
Yu-Hong Jiang2†
Xin Zhou3,4
Jia-Qi Yao2
Yang-Yang Wang1Jian-Qin Liu3,4
Peng-Cheng Zhang2
Wen-Fu Tang2*Zhi Li3,4*- 1School of Integrated Traditional Chinese and Western Medicine, Southwest Medical University, Luzhou, Sichuan, China
- 2Department of Integrated Traditional Chinese and Western Medicine, National Clinical Research Center for Geriatrics, West China Hospital, Sichuan University, Chengdu, China
- 3Department of Spleen and Stomach Diseases, Chinese Medicine Hospital Affiliated to Southwest Medical University, Luzhou, Sichuan, China
- 4The Key Laboratory of Integrated Traditional Chinese and Western Medicine for Prevention and Treatment of Digestive System Diseases of Luzhou city, Affiliated Traditional Medicine Hospital of Southwest Medical University, Luzhou, China
by Yang J, Jiang Y-H, Zhou X, Yao J-Q, Wang Y-Y, Liu J-Q, Zhang P-C, Tang W-F and Li Z (2024). Front. Immunol. 15:1353695. doi: 10.3389/fimmu.2024.1353695
Error in Figure/Table Legend
In the published article, there was an error in the legend for Figure 8E as published. The correct graph should display BCL-2/b-actin data, but due to an oversight, it currently shows the same data as Figure 8D (BAX/b-actin). We apologize for any confusion this may have caused and are committed to rectifying this issue promptly. The corrected figure appears below.
Figure 8
Figure 8 Administration of CH increases the apoptosis of pancreatic acinar cell in rats with AP. (A) Images from the TUNEL assay of pancreatic tissue, 100 μm scale bar. (n = 6). (B) Statistical results on the proportion of pancreatic acinar cells undergoing apoptosis in each group. Mean ± SD (n = 6) data were reported for each group, and statistical significance was observed. *P < 0.05, **P < 0.01, and ***P < 0.001 in comparison to the AP group. (C) Expression levels of BAX, BCL–2, and β–actin in various animal model groups. (n = 3). (D) Corresponding ratios of BAX/β–actin. Mean ± SD data were reported for each group, and statistical significance was observed (n = 3). **P < 0.01 and ****P < 0.0001 in comparison to the AP group. (E) Corresponding ratios of BCL–2/β–actin. Mean ± SD data were reported for each group, and statistical significance was observed (n = 3). *P < 0.05, **P < 0.01, ***P < 0.001, and ****P < 0.0001 in comparison to the AP group.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: acute pancreatitis, Chaihuang Qingyi Huoxue Granule, network pharmacology, molecular docking, pancreatic acinar cells, Traditional Chinese
Citation: Yang J, Jiang Y-H, Zhou X, Yao J-Q, Wang Y-Y, Liu J-Q, Zhang P-C, Tang W-F and Li Z (2024) Corrigendum: Material basis and molecular mechanisms of Chaihuang Qingyi Huoxue Granule in the treatment of acute pancreatitis based on network pharmacology and molecular docking-based strategy. Front. Immunol. 15:1429862. doi: 10.3389/fimmu.2024.1429862
Received: 08 May 2024; Accepted: 12 June 2024;
Published: 24 June 2024.
Edited by:
Ravi Kumar Sharma, Chandigarh University, IndiaReviewed by:
Vikas Kumar, Chandigarh University, IndiaCopyright © 2024 Yang, Jiang, Zhou, Yao, Wang, Liu, Zhang, Tang and Li. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Zhi Li, bGl6aGktc3dtdUAxMjYuY29t; Wen-Fu Tang, dGFuZ3dmQHNjdS5lZHUuY24=
†These authors have contributed equally to this work and share first authorship