Duerna Tie
Xia Da1
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- 1Department of Dermatology, Shimane University Faculty of Medicine, Izumo, Japan
- 2Department of Dermatology, Hokkaido University Graduate School of Medicine, Sapporo, Japan
- 3Division of Dermatology, Department of Medicine of Sensory and Motor Organs, Faculty of Medicine, Tottori University, Yonago, Japan
A Corrigendum on
Bullous pemphigoid IgG induces cell dysfunction and enhances the motility of epidermal keratinocytes via Rac1/proteasome activation
By Tie D, Da X, Natsuga K, Yamada N, Yamamoto O and Morita E (2019) Front. Immunol. 10:200. doi: 10.3389/fimmu.2019.00200
In the published article, there was an error in Figure 4 as published. The [Untreated] and [normal IgG] groups had the same high-magnification pictures (Row 3, columns 3 and 4). There was a mistake with the [Untreated] group. The corrected Figure 4 and its caption appear below.
FIGURE 4
Figure 4 SEM images of NHEKs stimulated with BP IgG. SEM images of NHEKs treated with BP IgGs (BP-1, BP-2, and BP-3) or normal IgG for 2 h. The lower magnification pictures show the connections of cells and filopodia. The higher magnification pictures show the cell surface and microvilli of individual cells. The alterations in the cell membrane structure are indicated with arrows.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: bullous pemphigoid, keratinocyte, IgG, cell adhesion, cell migration
Citation: Tie D, Da X, Natsuga K, Yamada N, Yamamoto O and Morita E (2023) Corrigendum: Bullous pemphigoid IgG induces cell dysfunction and enhances the motility of epidermal keratinocytes via Rac1/proteasome activation. Front. Immunol. 13:1109597. doi: 10.3389/fimmu.2022.1109597
Received: 28 November 2022; Accepted: 05 December 2022;
Published: 24 January 2023.
Edited and Reviewed by:
Takashi Hashimoto, Osaka City University, JapanCopyright © 2023 Tie, Da, Natsuga, Yamada, Yamamoto and Morita. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Eishin Morita, ZW1vcml0YUBtZWQuc2hpbWFuZS11LmFjLmpw