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CORRECTION article

Front. Immunol., 21 September 2022
Sec. NK and Innate Lymphoid Cell Biology
Volume 13 - 2022 | https://doi.org/10.3389/fimmu.2022.1032423

Corrigendum: Innate lymphoid cells are required to induce airway hyperreactivity in a murine neutrophilic asthma model

This article is a correction to:

  1. Innate Lymphoid Cells Are Required to Induce Airway Hyperreactivity in a Murine Neutrophilic Asthma Model

    1. Read original article
Anne-Charlotte JonckheereAnne-Charlotte Jonckheere1Sven F. SeysSven F. Seys1Brecht SteelantBrecht Steelant1Tatjana DecaestekerTatjana Decaesteker2Kaat DekosterKaat Dekoster3Jonathan CremerJonathan Cremer1Ellen DilissenEllen Dilissen1Dominique ScholsDominique Schols4Yoichiro IwakuraYoichiro Iwakura5Greetje Vande VeldeGreetje Vande Velde3Christine BreynaertChristine Breynaert1Rik SchrijversRik Schrijvers1Jeroen VanoirbeekJeroen Vanoirbeek6Jan L. CeuppensJan L. Ceuppens1Lieven J. Dupont,Lieven J. Dupont2,7Dominique M. A. Bullens,*Dominique M. A. Bullens1,8*
  • 1Department of Microbiology, Immunology and Transplantation, Allergy and Clinical Immunology Research Group, KU Leuven, Leuven, Belgium
  • 2Department of Chronic Diseases, Metabolism and Ageing, Laboratory of Respiratory Diseases and Thoracic Surgery, KU Leuven, Leuven, Belgium
  • 3Department of Imaging and Pathology, Biomedical MRI Unit/Molecular Small Animal Imaging Center (MoSAIC), KU Leuven, Leuven, Belgium
  • 4Department of Microbiology, Immunology and Transplantation, Laboratory of Virology and Chemotherapy, KU Leuven, Leuven, Belgium
  • 5Centre for Animal Disease Models, Research Institute for Biomedical Sciences, Tokyo University of Science, Chiba, Japan
  • 6Department of Public Health and Primary Care, Centre for Environment and Health, KU Leuven, Leuven, Belgium
  • 7Clinical Division of Respiratory Medicine, UZ Leuven, Leuven, Belgium
  • 8Clinical Division of Paediatrics, UZ Leuven, Leuven, Belgium

A corrigendum on
Innate lymphoid cells are required to induce airway hyperreactivity in a murine neutrophilic asthma model

by Jonckheere A-C, Seys SF, Steelant B, Decaesteker T, Dekoster K, Cremer J, Dilissen E, Schols D, Iwakura Y, Vande Velde G, Breynaert C, Schrijver R, Vanoirbeek J, Ceuppens JL, Dupont LJ, Bullens DMA (2022) . 13:849155. doi: .10.3389/fimmu.2022.849155

In the published article, there was an error made in the provided concentration lipopolysaccharide (LPS) applied endonasally in the murine model for neutrophilic asthma.

A correction has been made to Material and Methods section, “Neutrophilic asthma model”. The sentence previously stated:

“Wild-type, SCID, Rag2-/- γC-/-, and IL-17A-/- mice were endonasally challenged with 2 μg LPS (in a solution of 50 μl; 40 mg/ml) or 50 μl of saline (0.9% NaCl, B. Braun) on four consecutive days (Supplementary Figure 1A).”

The corrected sentence appears below:

“Wild-type, SCID, Rag2-/- γC-/-, and IL-17A-/- mice were endonasally challenged with 2 μg LPS (in a solution of 50 μl; 40 μg/ml) or 50 μl of saline (0.9% NaCl, B. Braun) on four consecutive days (Supplementary Figure 1A).”

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: innate lymphoid cells (ILCs), non-allergic asthma, murine model, neutrophilic inflammation, airway hyperreactivity

Citation: Jonckheere A-C, Seys SF, Steelant B, Decaesteker T, Dekoster K, Cremer J, Dilissen E, Schols D, Iwakura Y, Vande Velde G, Breynaert C, Schrijvers R, Vanoirbeek J, Ceuppens JL, Dupont LJ and Bullens DMA (2022) Corrigendum: Innate lymphoid cells are required to induce airway hyperreactivity in a murine neutrophilic asthma model. Front. Immunol. 13:1032423. doi: 10.3389/fimmu.2022.1032423

Received: 30 August 2022; Accepted: 01 September 2022;
Published: 21 September 2022.

Edited and Reviewed by:

Malcolm Ronald Starkey, Department of Immunology and Pathology, Monash University, Australia

Copyright © 2022 Jonckheere, Seys, Steelant, Decaesteker, Dekoster, Cremer, Dilissen, Schols, Iwakura, Vande Velde, Breynaert, Schrijvers, Vanoirbeek, Ceuppens, Dupont and Bullens. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Dominique M. A. Bullens, ZG9taW5pcXVlLmJ1bGxlbnNAa3VsZXV2ZW4uYmU=

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.