Skip to main content

CORRECTION article

Front. Immunol., 06 August 2021
Sec. Comparative Immunology

Corrigendum: Foot-and-Mouth Disease Virus Capsid Protein VP1 Antagonizes TPL2-Mediated Activation of the IRF3/IFN-β Signaling Pathway to Facilitate the Virus Replication

Junhong HaoJunhong HaoChaochao ShenChaochao ShenNannan WeiNannan WeiMinghao YanMinghao YanXuegang ZhangXuegang ZhangGuowei XuGuowei XuDajun ZhangDajun ZhangJing HouJing HouWeijun CaoWeijun CaoYe JinYe JinKeshan Zhang*Keshan Zhang*Haixue Zheng*Haixue Zheng*Xiangtao LiuXiangtao Liu
  • State Key Laboratory of Veterinary Etiological Biology, National Foot-and-Mouth Disease Reference Laboratory, Lanzhou Veterinary Research Institute of Chinese Academy of Agriculture Science, Lanzhou, China

A Corrigendum on
Foot-and-Mouth Disease Virus Capsid Protein VP1 Antagonizes TPL2-Mediated Activation of the IRF3/IFN-β Signaling Pathway to Facilitate the Virus Replication

By Hao J, Shen C, Wei N, Yan M, Zhang X, Xu G, Zhang D, Hou J, Cao W, Jin Y, Zhang K, Zheng H and Liu X (2021). Front. Immunol. 11:580334. doi:

In the original article, there was a mistake in the legend for Figure 6 as published. The legend of Figure 6A was not detailed enough and could easily be misunderstood. There was no description of the vector plasmid control. The correct legend appears below. The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

In the original article, there was a mistake in Figure 6 as published. We forgot to mark the two lanes of control plasmids (pCMV-3N-Myc) in the left Myc-TPL2 image in Figure 6A during the preparation and proof stage of our paper. This mistake led to the result that the actin control line included 12 lanes, while the Myc-TPL2 line contained 10 lanes in Figure 6A. There was no description of the transfected empty plasmids in the legend of Figure 6A. The corrected Figure 6A appears below.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

FIGURE 6
www.frontiersin.org

Figure 6 (A) HEK293T cells were transfected with TPL2 functional site mutant plasmids. Vector plasmids (pCMV-3N-Myc) were used as vector control (VC). Except for plasmids of T290A and D270A, the others occupied two lanes.

Publisher’s Note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: foot-and-mouth disease virus, viral protein 1, tumor progression locus 2, interferon regulatory factor 3/interferon-β, immune escape

Citation: Hao J, Shen C, Wei N, Yan M, Zhang X, Xu G, Zhang D, Hou J, Cao W, Jin Y, Zhang K, Zheng H and Liu X (2021) Corrigendum: Foot-and-Mouth Disease Virus Capsid Protein VP1 Antagonizes TPL2-Mediated Activation of the IRF3/IFN-β Signaling Pathway to Facilitate the Virus Replication. Front. Immunol. 12:686494. doi: 10.3389/fimmu.2021.686494

Received: 27 March 2021; Accepted: 15 June 2021;
Published: 06 August 2021.

Edited and reviewed by:

Leon Grayfer, George Washington University, United States

Copyright © 2021 Hao, Shen, Wei, Yan, Zhang, Xu, Zhang, Hou, Cao, Jin, Zhang, Zheng and Liu. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Keshan Zhang, vetzks009@163.com; Haixue Zheng, zhenghaixue@caas.cn

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.