This article is a correction to:
MFAP4 Deficiency Attenuates Angiotensin II-Induced Abdominal Aortic Aneurysm Formation Through Regulation of Macrophage Infiltration and Activity
Silke Griepke1,†Emilie Grupe1,†Jes Sanddal Lindholt2,3Elizabeth Hvitfeldt Fuglsang1Lasse Bach Steffensen1Hans Christian Beck2,4Mia Dupont Larsen1Sissel Karoline Bang-Møller1Martin Overgaard2,4Lars Melholt Rasmussen2,4Kate Lykke Lambertsen5,6,7
Jane Stubbe1,2*
- 1Department of Cardiovascular and Renal Research, Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark
- 2Elite Research Centre for Individualized Medicine in Arterial Diseases (CIMA), Odense University Hospital, Odense, Denmark
- 3Department of Cardiothoracic and Vascular Surgery, Odense University Hospital, Odense, Denmark
- 4Department of Clinical Biochemistry and Pharmacology, Odense University Hospital, Odense, Denmark
- 5Department of Neurobiology, Institute of Molecular Medicine, University of Southern Denmark, Odense, Denmark
- 6Department of Neurology, Odense University Hospital, Odense, Denmark
- 7BRIDGE—Brain Research—Inter-Disciplinary Guided Excellence, Department of Clinical Research, University of Southern Denmark, Odense, Denmark
A Corrigendum on
By Griepke S, Grupe E, Lindholt JS, Fuglsang EH, Steffensen LB, Beck HC, Larsen MD, Bang-Møller SK, Overgaard M, Rasmussen LM, Lambertsen KL and Stubbe J (2022). Front. Cardiovasc. Med. 9:942342. doi: 10.3389/fcvm.2022.942342
Error in Figure/Table
In the published article, there was an error in Figure 3B as published. The same micrograph in Figure 3B as overview micrograph for both the vehicle and XPro1595 treated group stained with CD206. It is only in the representative presentation for each group it appears and does not affect the semi-quantification of CD206 between groups as they were performed on all sections from each individual mouse in the groups and does therefore not affect the overall results or conclusions. The corrected Figure 3 and its caption appear below.
Figure 3
Figure 3. Effect of TNF inhibition on infiltrating immune cells 14 days after PPE-induced AAA formation. Representative micrographs after TNF inhibition showing the distribution of infiltrating immune cells (left) and semi-quantification (right, number of positive cells/mm^2) of CD45-positive leukocytes (A), CD206-positive M2-like macrophages (B), CD3-positive T-cells (C), and Ly6G-positive neutrophils (D) in the aneurysm wall after TNF inhibition (n = 3–5). Black arrowheads indicate positive stained cells. Data are shown as mean ± SEM. None of the semi-quantifications reached significance on one-way ANOVA using Bonferroni test for multiple comparisons. M, media; Ad, adventitia.
The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.
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Keywords: cardiovascular disease, abdominal aortic aneurysm, tumor necrosis factor inhibitor, translational research, vascular inflammation
Citation: Griepke S, Grupe E, Lindholt JS, Fuglsang EH, Steffensen LB, Beck HC, Larsen MD, Bang-Møller SK, Overgaard M, Rasmussen LM, Lambertsen KL and Stubbe J (2024) Corrigendum: Selective inhibition of soluble tumor necrosis factor signaling reduces abdominal aortic aneurysm progression. Front. Cardiovasc. Med. 11:1466614. doi: 10.3389/fcvm.2024.1466614
Received: 18 July 2024; Accepted: 7 August 2024;
Published: 19 August 2024.
Edited and Reviewed by: Morgan Salmon, University of Michigan, United States
Copyright: © 2024 Griepke, Grupe, Lindholt, Fuglsang, Steffensen, Beck, Larsen, Bang-Møller, Overgaard, Rasmussen, Lambertsen and Stubbe. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.
*Correspondence: Jane Stubbe, anN0dWJiZUBoZWFsdGguc2R1LmRr
†These authors have contributed equally to this work