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CORRECTION article

Front. Cell Dev. Biol., 24 August 2022
Sec. Cell Death and Survival

Corrigendum: Butyric acid protects against renal ischemia–reperfusion injury by adjusting the Treg/Th17 balance via HO-1/p-STAT3 signaling

Zhen Chen&#x;Zhen Chen1Miaomiao Wang&#x;Miaomiao Wang1Shikun Yang&#x;Shikun Yang2Jian ShiJian Shi1Tianhao JiTianhao Ji2Wei Ding,Wei Ding1,3Lianghua JiangLianghua Jiang4Zhiwen Fan
Zhiwen Fan5*Jing Chen
Jing Chen1*Yunjie Lu
Yunjie Lu1*
  • 1The Third Affiliated Hospital of Soochow University, Changzhou, China
  • 2Key Laboratory of Liver Transplantation, Hepatobiliary/Liver Transplantation Center, The First Affiliated Hospital of Nanjing Medical University, Chinese Academy of Medical Sciences, Nanjing, China
  • 3Wujin Hospital Affiliated With Jiangsu University, Changzhou, China
  • 4The First People’s Hospital of Kunshan, Kunshan, China
  • 5Department of Pathology, Affiliated Nanjing Drum Tower Hospital of Nanjing University School of Medicine, Nanjing, China

A Corrigendum on
Butyric acid protects against renal ischemia–reperfusion injury by adjusting the Treg/Th17 balance via HO-1/p-STAT3 signaling

by Chen Z, Wang M, Yang S, Shi J, Ji T, Ding W, Jiang L, Fan Z, Chen J and Lu Y (2021). Front. Cell Dev. Biol. 9:733308. doi: 10.3389/fcell.2021.733308

In the published article, there was an error in Figure 4H as published. We applied the incorrect picture to Figure 4H. The corrected Figure 4H and its caption “IRI + BA + BRL52537” appear below.

FIGURE 4
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FIGURE 4. BA protects against renal IRI via p-STAT3/SOCS3 signaling. (A,B) BA treatments attenuated the expression of p-STAT3 but not p-JAK2. (C,D) After BRL52537 treatment, the expression of p-STAT3 but not p-JAK2 was increased. (E–G) BRL52537 significantly attenuated the anti-inflammatory effects of BA on renal IRI. (H) HE staining indicated that BRL52537 significantly attenuated the protective effects of BA on renal IRI. Sample size = 3 in each group. Data were presented as mean ± standard deviation. N.S. p > 0.05, *p < 0.05, **p < 0.01.

The authors apologize for this error and state that this does not change the scientific conclusions of the article in any way. The original article has been updated.

Publisher’s note

All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article, or claim that may be made by its manufacturer, is not guaranteed or endorsed by the publisher.

Keywords: butyric acid, renal ischemia–reperfusion injury, Treg, Th17, HO-1, STAT3

Citation: Chen Z, Wang M, Yang S, Shi J, Ji T, Ding W, Jiang L, Fan Z, Chen J and Lu Y (2022) Corrigendum: Butyric acid protects against renal ischemia–reperfusion injury by adjusting the Treg/Th17 balance via HO-1/p-STAT3 signaling. Front. Cell Dev. Biol. 10:999965. doi: 10.3389/fcell.2022.999965

Received: 22 July 2022; Accepted: 22 July 2022;
Published: 24 August 2022.

Approved by:

Frontiers Editorial Office, Frontiers Media SA, Switzerland

Copyright © 2022 Chen, Wang, Yang, Shi, Ji, Ding, Jiang, Fan, Chen and Lu. This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

*Correspondence: Yunjie Lu, yjresearch@qq.com; Jing Chen, czyy_cj2021@163.com; Zhiwen Fan, zhiwenfannjdrum@gmail.com

These authors have contributed equally to this work and share first authorship

Disclaimer: All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers. Any product that may be evaluated in this article or claim that may be made by its manufacturer is not guaranteed or endorsed by the publisher.